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6篇 您的检索式:作者名="J.E.TROSKO"
    题名 作者 年代 出处 被引量
1胃癌细胞与正常细胞的间隙连接通讯和促癌剂效应的研究显示文摘林仲翔 赵雅丽 韩亚玲 张汝黻 J.E.Trosko 1989实验生物学报1989,,02:1
2胃癌细胞与正常细胞的间隙连接通讯和促癌剂效应的研究显示文摘林仲翔 赵雅丽 韩亚玲 张汝黻 J.E.Trosko 1989实验生物学报1989,,02:1
3胃癌细胞与正常细胞的间隙连接通讯和促癌剂效应的研究显示文摘林仲翔 赵雅丽 韩亚玲 张汝黻 J.E.Trosko 1989实验生物学报1989,,02:1
4胃癌细胞与正常细胞的间隙连接通讯和促癌剂效应的研究显示文摘林仲翔 赵雅丽 韩亚玲 张汝黻 J.E.Trosko 1989实验生物学报1989,,02:1
5胃癌细胞与正常细胞的间隙连接通讯和促癌剂效应的研究显示文摘林仲翔 赵雅丽 韩亚玲 张汝黻 J.E.Trosko 1989实验生物学报1989,,02:1
6GAP JUNCTIONAL INTERCELLULAR COMMUNICATION (GJIC) IN RAT GLIOMA CELLS — CHARACTERIZATIONS TO DETECT INHIBITORS OF METABOLIC COOPERATION显示文摘Gap junctional intercellular communication (GJIC) as measured by metabolic cooperation was examined in a rat glioma cell line P98F47. X-ray induced mutants of P98F47 cells were grown in 6-thioguanine selective medium (6TG medium) to separate 6TG-resistant HGPRT- mutant cells (6TGr). By co-culturing 200 6TGr cells with varied high densities of the wild type 6TG-sensitive cells (6TGs), it was found that the recovery of 6TGr cells depended on the density of 6TGs cells. Higher densities of 6TGs cells reduced the recovery of 6TGr cells. These results demonstrate the ability of P98F47 cells to perform metabolic cooperation which is indicative of GJIC. When metabolic cooperation was inhibited, increased recovery of 6TGr cells was observed. Presented results also demonstrate metabolic cooperation between P98F47 glioma cells and normal rat glial cells. Effect of tumor promoting chemicals on metabolic cooperation of P98F47 cells was studied.3H-uridine nucleot de echnique林仲翔 C.C.CHANG J.E.TROSKO 1990Science China Chemistry1990,33,4:0
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