5篇
您的检索式：作者名="Junxun YIN"
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| 1 | The experiment study of spatial mask effect显示文摘masker 与 maskee 的理论分析和实验是空间的在方位角的分离证明当 masker 和 maskee 空间地被分开时,阀值趋于到落下和空间揭露的 producethe 现象。结果也证明空间揭露与频率有关。频率越高,空间揭露是越多 stronger。最大的揭露是 15 dB。主要暗示是 masker 和 maskee 什么时候采购原料,空间地在方位角被分开,因为头的影响,单耳的 Signal-to-Mask 比率(SMR ) 将增加相关转移功能(HRTF ) 。同时,由更高听觉的系统的听觉的信息的有两耳的处理是空间揭露的另一个原因。 | XIE Zhiwen YIN Junxun RAO Dan | 2006 | Chinese Journal of Acoustics2006，25，1： | 2 |
| 2 | Fingertip Detection Approach fur Finger- writing Chinese Character Recognition System显示文摘 | Yang Duanduan Jin Lianwen Yin Junxun | 2007 | Journal of South China University of Technology ( Natural Science Edition)2007，35，1： | 1 |
| 3 | Using redundant parallel architecture to improve speaker recognition performance显示文摘In this paper, we propose two kinds of modifications in speaker recognition. First, the correlations between frequency channels are of prime importance for speaker recognition. Some of these correlations are lost when the frequency domain is divided into sub-bands. Consequently we propose a particularly redundant parallel architecture for which most of the correlations are kept. Second, generally a log transformation used to modify the power spectrum is done after the filter-bank in the classical spectrum calculation. We will see that performing this transformation before the filter bank is more interesting in our case. In the processing of recognition, the Gaussian mixture model (GMM) recognition arithmetic is adopted. Experiments on speech corrupted by noise show a better adaptability of this approach in noisy environments, compared with a conventional device, especially when pruning of some recognizers is performed. | Zhengquan QIU Junxun YIN Caiyun FAN | 2008 | 控制理论与应用(英文版)2008，6，2： | 1 |
| 4 | De-noising with Novel DWT-MFCC for Speaker Recognition显示文摘 | Zhengquan Qiu Junxun Yin | 2006 | 通讯和计算机(中英文版)2006，3，7： | 0 |
| 5 | Protein phosphatase 6 (Pp6) is crucial for regulatory T cell function and stability in autoimmunity显示文摘Regulatory T(T_(reg))cells constitute a dynamic population that is critical in autoimmunity.T_(reg) cell therapies for autoimmune diseases are mainly focused on enhancing their suppressive activities.However,recent studies demonstrated that certain inflammatory conditions induce T_(reg) cell instability with diminished FoxP3 expression and convert them into pathogenic effector cells.Therefore,the identification of novel targets crucial to both T_(reg) cell function and plasticity is of vital importance to the development of therapeutic approaches in autoimmunity.In this study,we found that conditional Pp6 knockout(cKO)in T_(reg) cells led to spontaneous autoinflammation,immune cell activation,and diminished levels of FoxP3 in CD4^(+)T cells in mice.Loss of Pp6 in T_(reg) cells exacerbated two classical mouse models of T_(reg)-related autoinflammation.Mechanistically,Pp6 deficiency increased CpG motif methylation of the FoxP3 locus by dephosphorylating Dnmt1 and enhancing Akt phosphorylation at Ser473/Thr308,leading to impaired FoxP3 expression in T_(reg) cells.In summary,our study proposes Pp6 as a critical positive regulator of FoxP3 that acts by decreasing DNA methylation of the FoxP3 gene enhancer and inhibiting Akt signaling,thus maintaining T_(reg) cell stability and preventing autoimmune diseases. | Wei Cai Junxun Zhang Hong Zhou Xiangxiao Li Fangzhou Lou Yang Sun Zhenyao Xu Jing Bai Qianqian Yin Zhikai Wang Libo Sun Xiaojie Cai Sibei Tang Yue Wu Li Fan Hong Wang Honglin Wang Qun Li | 2022 | Genes & Diseases2022，9，2： | 0 |
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