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| 1 | 花曲柳窄吉丁的寄主植物范围、危害和防治对策显示文摘花曲柳窄吉丁(emeraldashborer,EAB)AgrilusplanipennisFairmaire是木犀科(Oleaceae)属Fraxinus植物树木的毁灭性蛀干害虫,目前发现其主要危害欧亚属Subgen.Fraxinus欧组(SectionFraxinus)和象蜡树组(SectionMeliodes)的一些树木种类,如美国白蜡Fraxinusamericana、洋白蜡F.pennsylvanicavarsubintegerrima、绒毛白蜡F.velutina、黑白蜡F.nigra和水曲柳F.mandshurica等。与早期文献上的记载不同,很少发现花曲柳窄吉丁对苦枥木亚属Subgen.Ornus白蜡树组(SectionOrnaster)的白蜡树F.chinensisRoxb和花曲柳F.rhynchophylla造成危害。不同寄主白蜡树对花曲柳窄吉丁的敏感性不同,洋白蜡、绒毛白蜡等北美白蜡树种受其危害重于亚洲的水曲柳。这可能是由于长期的适应进化,分布在亚洲地区的属寄主树木发展了对花曲柳窄吉丁的抗虫性。中国部分地区发生的花曲柳窄吉丁灾害应该归属于引进的北美白蜡树种对本地害虫敏感所致。在天津、黑龙江、吉林、辽宁、河北、山东、四川、台湾、内蒙古和新疆等省市和自治区,都曾发现花曲柳窄吉丁的危害或分布。白蜡属树木广泛分布于我国除了青藏地区和海南省之外的大部分地区,其中大面积引种北美白蜡树种的北方和西北地区,以及近年来种植发展水曲柳的东北地区都是花曲柳窄吉丁这种毁灭性蛀干害虫的潜在发生地。花曲柳窄吉丁在我国部分地区为检疫性林业害虫,做好检疫是杜绝其扩散蔓延的首要方法。我国局部地区出现花曲柳窄吉丁灾害说明,北美白蜡树种在我国的适应性还需要进行更进一步科学的全面的风险评估,在我国生态建设中应重视利用我国本地的白蜡树种。在花曲柳窄吉丁灾害的治理中,可采用营造混交林以降低蔓延的速度和发挥自然控制能力,加强肥水管理以增强树木的抗虫能力,清除被害木消灭虫源,以及幼虫期树干注射、成虫期喷洒化学农药等防治措施。 | 赵同海 高瑞桐 Houping LIU Leah S.BAUER 孙龙强 | 2005 | 昆虫学报2005，48，4： | 28 |
| 2 | Nutrition management in acute pancreatitis: Clinical practice consideration显示文摘Acute pancreatitis(AP)is a common gastrointestinal disease and the leading cause of hospital admission and healthcare burden among gastrointestinal disorders in many countries.Patients can present with varying degrees of inflammation and disease severity,ranging from self-limiting mild AP to devastating and fatal severe AP.Many factors contribute to malnutrition in AP,especially abnormal metabolism and catabolism related to inflammation.The concept of“pancreatic rest”is not evidence-based.There is however,emerging evidence that supports the use of oral or enteral nutrition to improve nutrition status and to reduce local and systemic inflammation,complications,and death.In mild disease,patients are generally able to initiate solid oral diet and do not require specialized nutrition care such as enteral or parenteral nutrition.In contrast,nutrition interventions are imperative in moderately severe and severe AP.The current article aims to review the latest evidence and suggest practical nutrition interventions in patients with AP,including nutrition requirements,routes of nutrition treatment,types of formula,and the role of nutritional supplements,such as glutamine,probiotics,omega-3 fatty acids,and antioxidants. | Narisorn Lakananurak Leah Gramlich | 2020 | World Journal of Clinical Cases2020，8，9： | 25 |
| 3 | 5-Methylcytosine RNA Methylation in Arabidopsis Thaliana显示文摘5-Methylcytosine (m 5 C ) 是描绘得好的 DNA 修正,并且主要也在初核质和优核质在丰富的非编码的 RNA 被报导。然而,分发和 m 5 在植物 mRNAs 的 C 仍然保持大部分未知。这里,我们报导 RNA m 5 在由使用 m 5 C RNA immunoprecipitation 由一条深定序的途径列在后面(m 5 C-RIP-seq ) 。LC-MS/MS 和点污点分析揭示 m 5 在各种各样的纸巾并且在不同发展阶段的 C mRNA 修正。m 5 C-RIP-seq 分析识别了 6045 m 5 在 4465 的 C 山峰在年轻幼苗表示了基因。我们发现那 m 5 C 在编码序列,二座山峰在开始 codons 以后并且在站 codons 前立即定位了被充实,并且随低翻译与 mRNAs 被联系活动。我们进一步表明了那 RNA (cytosine-5 )-methyltransferase,tRNA 特定的 methyltransferase 4B (TRM4B ) ,展览 m 5 C RNA methyltransferase 活动。在 TRM4B 的变化在根开发和减少的 m 5 C 山峰。TRM4B 影响涉及根开发的基因的抄本层次,它断然与他们的 mRNA 稳定性和 m 5 C 层次。我们的结果建议那 m 5 在 mRNA 的 C 是新 epitranscriptome 标记 inArabidopsis,并且这修正的那条规定是基因的不可分的部分规章的网络位于 \O 下面植物开发。 | Xuean Cui Zhe Liang Lisha Shen Qian Zhang Shengjie Bao Yuke Geng Bin Zhang Vonny Leo Leah A. Vardy Tiegang Lu Xiaofeng Gu Hao Yu | 2017 | Molecular Plant2017，10，11： | 19 |
| 4 | Epstein-Barr virus and Burkitt lymphoma显示文摘In 1964,a new herpesvirus,Epstein-Barr virus(EBV),was discovered in cultured tumor cells derived from a Burkitt lymphoma(BL)biopsy taken from an African patient.This was a momentous event that reinvigorated research into viruses as a possible cause of human cancers.Subsequent studies demonstrated that EBV was a potent growth-transforming agent for primary B cells,and that all cases of BL carried characteristic chromosomal translocations resulting in constitutive activation of the c-MYC oncogene.These results hinted at simple oncogenic mechanisms that would make Burkitt lymphoma paradigmatic for cancers with viral etiology.In reality,the pathogenesis of this tumor is rather complicated with regard to both the contribution of the virus and the involvement of cellular oncogenes.Here,we review the current understanding of the roles of EBV and c-MYC in the pathogenesis of BL and the implications for new therapeutic strategies to treat this lymphoma. | Martin Rowe Leah Fitzsimmons Andrew I Bell | 2014 | Chinese Journal of Cancer2014，33，12： | 14 |
| 5 | 重要林业蛀干害虫花曲柳窄吉丁研究现状显示文摘花曲柳窄吉丁(Agrilus planipennis Fairmaire)是我国重要的林业危险性害虫,目前发现其主要危害木犀科(Oleaceae)白蜡属(Fraxinus)的一些重要林木.它主要分布于东北亚一些国家,近年来在北美的一些地区也发现其对当地的白蜡树造成了严重危害.花曲柳窄吉丁幼虫在树皮下形成层区域蛀食危害,扁圆形坑道横向弯曲切断输导组织,使树木衰弱致死.本文对此害虫的生物学和危害特点、在我国不同地区的发育生活史、天敌昆虫和病原微生物以及控制技术等方面的研究现状进行了综述. | 赵同海 高瑞桐 Houping Liu Leah S. Bauer | 2004 | 中国造纸学报2004，19，z1： | 10 |
| 6 | 花曲柳窄吉丁在中国的分布与危害的调查研究显示文摘花曲柳窄吉丁是木犀科梣属树木毁灭性的蛀干害虫,2003年已被列入我国林业危险性有害生物名单中.本研究对中国黑龙江、吉林、辽宁、河北和山东省及天津和北京市16个梣属树木分布区进行了花曲柳窄吉丁的调查.结果显示:除在北京市和山东省滨州市以及辽宁省的一个地区未发现花曲柳窄吉丁危害外,在其它省市的13个地区都发现有该害虫,而且在部分地区其危害已较为严重,有虫株率最高达90%以上,虫口密度最高达230头/株,并已出现部分白蜡树死亡.另外,在花曲柳窄吉丁自然种群中发现有幼虫被真菌感染致死和茧蜂科天敌寄生现象,并对害虫的种群发展显示了较好的控制作用. | 高瑞桐 赵同海 Houping Liu Leah S. Bauer Toby R. Petrice | 2004 | 中国造纸学报2004，19，z1： | 7 |
| 7 | Methyl-CpG binding proteins in the nervous system显示文摘Classical methyl-CpG binding proteins contain the conserved DNA binding motif methyl-cytosine binding domain(MBD), which preferentially binds to methylated CpG dinucleotides. These proteins serve as transcriptional repressors,mediating gene silencing via DNA cytosine methylation. Mutations in methyl-CpG binding protein 2 (MeCP2) have beenlinked to the human mental retardation disorder Rett syndrome, suggesting an important role for methyl-CpG bindingproteins in brain development and function. This mini-review summarizes the recent advances in studying the diversefunctions of MeCP2 as a prototype for other methyl-CpG binding proteins in the development and function of thevertebrate nervous system. | Leah HUTNICK | 2005 | Cell Research2005，15，4： | 7 |
| 8 | Tau-induced neurodegeneration: mechanisms and targets显示文摘The accumulation of hyperphosphorylated tau is a common feature of several dementias. Tau is one of the brain microtubule-associated proteins. Here we discuss tau's functions in microtubule assembly and stabilization and with regard to its interactions with other proteins. We describe and analyze important post-translational modifications: hyperphosphorylation, ubiquitination, glycation, glycosylation, nitration, polyamination, proteolysis, acetylation, and methylation. We discuss how these post-translational modifi cations can alter tau's biological function. We analyze the role of mitochondrial health in neurodegeneration. We propose that microtubules could be a therapeutic target and review different approaches. Finally, we consider whether tau accumulation or its conformational change is related to tau-induced neurodegeneration, and propose a mechanism of neurodegeneration. | Cindy Beharry Leah S. Cohen Jing Di Kawsar Ibrahim Susan Briffa-Mirabella Alejandra del C. Alonso | 2014 | Neuroscience Bulletin2014，30，2： | 6 |
| 9 | The effect of the CHAMP intervention on fundamental motor skills and outdoor physical activity in preschoolers显示文摘Purpose: Physical activity(PA) and fundamental motor skills are important components of current and future trajectories of health in young children. This study examined the effects of a 5-week motor skill intervention on preschoolers' motor skill competence and their PA behaviors while participating in the motor skill intervention or outdoor free play(recess).Methods: A total of 102 preschoolers served as participants and were part of a motor skill intervention group(n = 64) or a control/outdoor free play group(n = 38). Children's motor skills were assessed before and after the intervention using the Test of Gross Motor Development-3 rd edition. PA during the motor skill intervention and outdoor free play was assessed using accelerometers both immediately before the start(baseline, Week 0 or Week 1) and end(late, Week 5 or Week 6) of the intervention.Results: All children significantly improved their motor skills from baseline to late assessment(p < 0.05). Children in the motor skill intervention demonstrated greater rates of change(p < 0.001) and scored higher on all motor skills at the late assessment compared with the control group(p < 0.001). There was no effect of group(control vs. intervention), but there was a significant effect of sex on children's PA during outdoor free play at baseline(p < 0.05). Similarly, there was no effect of group on PA during days with the movement program(intervention vs. outdoor free play) at either time point, but boys were more active than girls at the late assessment(p < 0.05). Last, children in the intervention engaged in more PA while participating in the intervention toward the end of the intervention than at the beginning.Conclusion: The 5-week motor skill intervention was effective at improving preschoolers' motor skills and rates of change in motor skills were higher for children who completed the intervention compared with children in the control group. Preschoolers in the intervention did demonstrate PA changes while participating in the intervention, but these changes did not translate outside of the intervention setting. | Kara K.Palmer Katherine M.Chinn Leah E.Robinson | 2019 | Journal of Sport and Health Science2019，8，2： | 6 |
| 10 | Hepatic angiosarcoma with clinical and histological features of Kasabach-Merritt syndrome显示文摘Hepatic angiosarcoma is a mesenchymal tumor originating from liver sinusoidal endothelial cells. It is an extremely rare malignant neoplasm accounting for less than 1% of primary malignant liver tumors. The deregulated coagulopathy that can be seen in hepatic angiosarcoma fulfills the clinical diagnostic criteria of disseminated intravascular coagulation. However, the mechanism that governs this coagulopathy has been poorly understood. This case report provides histological evidence of the consumption of coagulation factors along with trapped platelets occurring within the tumor, which is the foundation for the concept of Kasabach-Merritt syndrome(KMS). KMS is characterized by thrombocytopenia and hyperconsumption of coagulation factors within a vascular tumor. However, KMS associated with angiosarcoma has not been well recognized. This case report describes, for the first time, the histological evidence of KMS that occurred in an extremely rare mesenchymal malignant tumor of the liver. | Sanya Wadhwa Tae Hun Kim Leah Lin Gary Kanel Takeshi Saito | 2017 | World Journal of Gastroenterology2017，23，13： | 5 |
| 11 | Improvement in Perioperative Outcome After Hepatic Resection: Analysis of 1,803 Consecutive Cases Over the Past Decade显示文摘 | William R. Jarnagin Mithat Gonen Yuman Fong Ronald P. DeMatteo Leah Ben-Porat Sarah Little Carlos Corvera Sharon Weber Leslie H. Blumgart | 2002 | Annals of Surgery2002，，4： | 5 |
| 12 | Pre-osteoblastic MC3T3-E1 cells promote breast cancer growth in bone in a murine xenograft model显示文摘The bones are the most common sites of breast cancer metastasis. Upon arrival within the bone microenvironment, breast cancer cells coordinate the activities of stromal cells, resulting in an increase in osteoclast activity and bone matrix degradation. In late stages of bone metastasis, breast cancer cells induce apoptosis in osteoblasts, which further exacerbates bone loss. However, in early stages, breast cancer cells induce osteoblasts to secrete inflammatory cytokines purported to drive tumor progression. To more thoroughly evaluate the role of osteoblasts in early stages of breast cancer metastasis to the bones, we used green fluorescent protein-labeled human breast cancer cell lines MDA-MB-231 and MDA-MB-435, which both induce osteolysis after intra-femoral injection in athymic mice, and the murine pre-osteoblastic cell line MC3T3-E1 to modulate osteoblast populations at the sites of breast cancer metastasis. Breast cancer cells were injected directly into the femur with or without equal numbers of MC3T3-E1 cells. Tumors grew significantly larger when co-injected with breast cancer cells and MC3T3-E1 cells than injected with breast cancer cells alone. Osteolysis was induced in both groups, indicating that MC3T3-E1 cells did not block the ability of breast cancer cells to cause bone destruction. MC3T3-E1 cells promoted tumor growth out of the bone into the extraosseous stroma. These data suggest that breast cancer cells and osteoblasts communicate during early stages of bone metastasis and promote tumor growth. | Thomas M. Bodenstine Benjamin H. Beck Xuemei Cao Leah M. Cook Aimen Ismail J. Kent Powers Andrea M. Mastro Danny R. Welch | 2011 | Chinese Journal of Cancer2011，30，3： | 5 |
| 13 | Influence of Transfusions on Perioperative and Long-Term Outcome in Patients Following Hepatic Resection for Colorectal Metastases显示文摘 | David A. Kooby Jennifer Stockman Leah Ben-Porat Mithat Gonen William R. Jarnagin Ronald P. Dematteo Scott Tuorto David Wuest Leslie H. Blumgart Yuman Fong | 2003 | Annals of Surgery2003，，6： | 4 |
| 14 | Discovering small molecules as Wnt inhibitors that promote heart regeneration and injury repair显示文摘There are intense interests in discovering pro regenerative medicine leads that can promote cardiac differentiation and regeneration,as well as repair damaged heart tissues.We have combined zebrafish embryo-based screens with cardiomyogenesis assays to discover selective small molecules that modulate heart development and regeneration with minimal adverse effects.Two related compounds with novel structures,named as Cardiomogen 1 and 2(CDMG1 and CDMG2),were identified for their capacity to promote myocardial hyperplasia through expansion of the cardiac progenitor cell population.We find that Cardiomogen acts as a Wnt inhibitor by targeting p-catenin and reducing Tcf/Lef-mediated transcription in cultured cells.CDMG treatment of amputated zebrafish hearts reduces nuclear p-catenin in injured heart tissue,increases cardiomyocyte(CM)proliferation,and expedites wound healing,thus accelerating cardiac muscle regeneration.Importantly,Cardiomogen can alleviate the functional deterioration of mammalian hearts after myocardial infarction.Injured hearts exposed to CDMG1 display increased newly formed CMs and reduced fibrotic scar tissue,which are in part attributable to the^-catenin reduction.Our findings indicate Cardiomogen as a Wnt inhibitor in enhancing injury-induced CM proliferation and heart regeneration,highlighting the values of embryo-based small molecule screens in discovery of effective and safe medicine leads. | Shuying Xie Wenbin Fu Guangju Yu Xueli Hu Kaa Seng Lai Xiangwen Peng Yating Zhou Xuejiao Zhu Ptamen Christov Leah Sawyer Terri T.Ni Gary A.Sulikowski Zhongzhou Yang Ethan Lee Chunyu Zeng Wei E.Wang Tao P.Zhong | 2020 | Journal of Molecular Cell Biology2020，12，1： | 4 |
| 15 | 轻度缺乏磷的日粮中随着钙和有效磷比值增加对保育猪生长性能和骨骼发育的不利影响显示文摘文章旨在评估在含植酸酶日粮中(250 FTU/kg)增加钙(Ca)和有效磷(aP)比例对保育猪生长性能和骨骼矿物质含量(骨密度,BMC)的影响。试验将480头保育猪(体重BW=5.7±0.6 kg)随机分配为7个处理,Ca∶aP递增分别为:1.25∶1、1.50∶1、1.75∶1、2.00∶1、2.25∶1、2.50∶1、2.75∶1,每个处理7个重复(其中6个重复饲喂2.75∶1日粮),每个重复10头猪。从试验正式开始前7 d至第0天,所有仔猪饲喂相同日粮。试验期分为两个阶段(阶段1和阶段2),分别为1—14 d和15—28 d,试验猪只在试验期饲喂处理日粮。阶段1和阶段2日粮配方中有效磷比例分别为0.33%和0.27%(大约为需要量的90%)。测定体重(BW)、平均日增重(ADG)、平均日采食量(ADFI)和体重与饲料比(G∶F)。试验第28天使用双X射线骨密度仪测定每头猪肱骨的BMC。数据采用PROC MIXED(SAS 9.3)线性混合模型分析。利用正交多项式对比计算Ca∶aP递增的线性和二次效应。整个28 d试验期,Ca∶aP递增导致ADG(353、338、328、304、317、291和280 g/d;P<0.01)、ADFI(539、528、528、500、533、512和489 g/d;P<0.05)和G∶F(0.68、0.66、0.64、0.62、0.61、0.59和0.58;P<0.01)呈线性递减。Ca∶aP递增也会导致第14天和第28天BW降低(P<0.01)。肱骨的BMC随着Ca∶aP递增而减少(6.2、6.3、5.7、5.9、5.5、5.6和5.3 g;P<0.01)。下面是Ca∶aP对各指标的回归分析影响,其中X是Ca∶aP:ADG[g/d]=339-36x,r^2=0.81;G:F=0.61-0.03x,r^2=0.72;BMC[g]=6.4-0.27x,r^2=0.43。所有结果表明,任何高于这次试验中所使用的最低Ca∶aP的钙水平日粮均会损害动物生长性能和骨骼发育。有必要进一步研究日粮中更低水平钙含量。 | 何颖(编译) Spenser L.Becker Stacie A.Gould Amy L.Petry Leah M.Kellesvig John.F.Patience | 2020 | 猪业科学2020，37，12： | 3 |
| 16 | Retinal regeneration requires dynamic Notch signaling显示文摘Retinal damage in the adult zebrafish induces Müller glia reprogramming to produce neuronal progenitor cells that proliferate and differentiate into retinal neurons.Notch signaling,which is a fundamental mechanism known to drive cell-cell communication,is required to maintain Müller glia in a quiescent state in the undamaged retina,and repression of Notch signaling is necessary for Müller glia to reenter the cell cycle.The dynamic regulation of Notch signaling following retinal damage also directs proliferation and neurogenesis of the Müller glia-derived progenitor cells in a robust regeneration response.In contrast,mammalian Müller glia respond to retinal damage by entering a prolonged gliotic state that leads to additional neuronal death and permanent vision loss.Understanding the dynamic regulation of Notch signaling in the zebrafish retina may aid efforts to stimulate Müller glia reprogramming for regeneration of the diseased human retina.Recent findings identified DeltaB and Notch3 as the ligand-receptor pair that serves as the principal regulators of zebrafish Müller glia quiescence.In addition,multi-omics datasets and functional studies indicate that additional Notch receptors,ligands,and target genes regulate cell proliferation and neurogenesis during the regeneration time course.Still,our understanding of Notch signaling during retinal regeneration is limited.To fully appreciate the complex regulation of Notch signaling that is required for successful retinal regeneration,investigation of additional aspects of the pathway,such as post-translational modification of the receptors,ligand endocytosis,and interactions with other fundamental pathways is needed.Here we review various modes of Notch signaling regulation in the context of the vertebrate retina to put recent research in perspective and to identify open areas of inquiry. | Leah J.Campbell Jaclyn L.Levendusky Shannon A.Steines David R.Hyde | 2022 | Neural Regeneration Research2022，17，6： | 3 |
| 17 | RACK1 regulates neural development显示文摘Receptor for activated C kinase 1(RACK1)is an evolutionarily conserved scaffolding protein within the tryptophan-aspartate(WD)repeat family of proteins.RACK1 can bind multiple signaling molecules concurrently,as well as stabilize and anchor proteins.RACK1 also plays an important role at focal adhesions,where it acts to regulate cell migration.In addition,RACK1 is a ribosomal binding protein and thus,regulates translation.Despite these numerous functions,little is known about how RACK1 regulates nervous system development.Here,we review three studies that examine the role of RACK1 in neural development.In brief,these papers demonstrate that(1)RACK-1,the C.elegans homolog of mammalian RACK1,is required for axon guidance;(2)RACK1 is required for neurite extension of neuronally differentiated rat PC12cells;and(3)RACK1 is required for axon outgrowth of primary mouse cortical neurons.Thus,it is evident that RACK1 is critical for appropriate neural development in a wide range of species,and future discoveries could reveal whether RACK1 and its signaling partners are potential targets for treatment of neurodevelopmental disorders or a therapeutic approach for axonal regeneration. | Leah Kershner Kristy Welshhans | 2017 | Neural Regeneration Research2017，12，7： | 3 |
| 18 | Efficacy of selective laser trabeculoplasty following incisional glaucoma surgery显示文摘AIM: To evaluate the efficacy of selective laser trabeculoplasty(SLT) in glaucomatous eyes with previous incisional glaucoma surgery. METHODS: A retrospective cohort of eyes that underwent SLT at a single institution from 2013-2015 were followed for 1 y. Reduction in intraocular pressure(IOP) following SLT was evaluated in eyes with prior trabeculectomy with Ex Press mini shunt(Alcon, Ft Worth, TX, USA), Ahmed valve(New World Medical, Cucamonga, CA, USA), or combined phacoemulsification-trabeculectomy. A control group was included with eyes without prior surgery that underwent SLT. Success was defined as >20% drop in IOP from pre-SLT baseline. RESULTS: One-hundred and six eyes were included with 53 in both the prior glaucoma surgery(PGS) and no prior glaucoma surgery(NPGS) groups. Mean pre-SLT IOP was 19.2±4.3 and 20.6±6.0 mm Hg for PGS and NPGS groups, respectively(P=0.17). Both groups produced statistically significant IOP reductions at 1 and 6 mo(P<0.04). At 6 mo, mean IOP reduction reached 7.3% and 10.8% for the PGS and NPGS groups, respectively(P=0.42). Overall, 27.9% and 31.7% of eyes in PGS and NPGS groups met success criteria at 1 y(P=0.70). In the PGS group, eyes with baseline IOP ≥21 mm Hg had IOP reductions of 18.1%(P<0.001), 16.7%(P<0.01), and 8.4%(P=0.31) compared to eyes with baseline IOP <21 mm Hg who had IOP reductions of 2.3%(P=0.39), 3.4%(P=0.19), and 1.1%(P=0.72) at 1, 6 mo, and 1 y, respectively. CONCLUSION: SLT is efficacious in eyes with prior incisional glaucoma surgery and results in similar IOP reductions compared to eyes without PGS. A larger IOP reduction is observed following SLT in eyes with higher pre-SLT IOP. | Robert Allan Sharpe Leah L Kammerdiener David B.Williams Sudeep K.Das Matthew J.Nutaitis | 2018 | International Journal of Ophthalmology(English edition)2018，11，1： | 3 |
| 19 | Improvement in Perioperative Outcome After Hepatic Resection: Analysis of 1,803 Consecutive Cases Over the Past Decade显示文摘 | William R. Jarnagin Mithat Gonen Yuman Fong Ronald P. DeMatteo Leah Ben-Porat Sarah Little Carlos Corvera Sharon Weber Leslie H. Blumgart | 2002 | Annals of Surgery2002，，4： | 3 |
| 20 | Evidence for alterations in central noradrenergic signaling in irritable bowel syndrome显示文摘 | Steven Berman Brandall Suyenobu Bruce D. Naliboff Joshua Bueller Jean Stains Heng Wong Mark Mandelkern Leah Fitzgerald Gordon Ohning Arpana Gupta Jennifer S. Labus Kirsten Tillisch Emeran A. Mayer | 2012 | Neuroimage2012，，4： | 3 |
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