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8篇 您的检索式:作者名="Liuwang"
    题名 作者 年代 出处 被引量
1Mice generated from tetraploid complementation competent iPS cells show similar developmental features as those from ES cells but are prone to tumorigenesis显示文摘ManTong ZhuoLv LeiLiu HuiZhu Qin-YuanZheng Xiao-YangZhao WeiLi Yi-BoWu Hai-JiangZhang Hua-JunWu Zhi-KunLi FanyiZeng LiuWang Xiu-JieWang Jia-HaoSha QiZhou 2011Cell Research2011,21,11:11
2The complete mitochondrial genome of the Keeled box turtle Pyxidea mouhotii and phylogenetic analysis of major turtle groups显示文摘The complete mitochondrial genome (16,837 bp) from the Keeled box turtle (Pyxidea mouhotii) was determined. The genome content, gene order, and base composition conformed to the consensus vertebrate type mtDNA. However, a remarkable feature was found in this molecule: a large number of (ATTATATC) n direct tandem repeats followed by (TA) n microsatellite at the 3′ end of the control region (D-loop), which might be useful as molecular markers for studying population genetics and helpful for species identification and conser- vation. Besides, to review phylogenetic relationships among major turtle lineages, maximum-likelihood (ML) and Bayesian (BI) analyses were conducted based on concatenated sequences of 13 protein-coding genes from 16 taxa. The resultant ML and BI analyses showed homological topologies, which only differed on the exact placement of Platysternon. Nevertheless, the results strongly supported that 1) Pyxidea mouhotii and Cuora aurocapitata formed a monophyletic clade, whereas Cyclemys atripons was not closer to the Pyxidea-Cuora than to Chinemys reevesii, suggesting that Cyclemys and the Cuora group (containing Pyxidea) may have originated from two ancestors; 2) the Geoemydidae with Testudinidae was a sister group rather than with the Emydidae.Li Zhang Liuwang Nie Chenghe Cao Ying Zhang 2008Journal of Genetics and Genomics2008,35,1:3
3Changes in secretory pathway Ca^(2+)-ATPase 2 following focal cerebral ischemia/reperfusion injury显示文摘This study aimed to investigate changes in secretory pathway Ca2+-ATPase 2 expression following cerebral ischemia/reperfusion injury, and to define the role of Ca2+-ATPases in oxidative stress. A rat model of cerebral ischemia/reperfusion injury was established using the unilateral middle cerebral artery occlusion method. Immunohistochemistry and reverse transcription-PCR assay results showed that compared with the control group, the expression of secretory pathway Ca2+-ATPase 2 protein and mRNA in the cerebral cortex and hippocampus of male rats did not significantly change during the ischemic period. However, secretory pathway Ca2+-ATPase 2 protein and mRNA expression reduced gradually at 1, 3, and 24 hours during the reperfusion period. Our experimental findings indicate that levels of secretory pathway Ca2+-ATPase 2 protein and mRNA expression in brain tissue change in response to cerebral ischemia/reperfusion injury.Tonglin Lu Zhiping Hu Liuwang Zeng Zheng Jiang 2013Neural Regeneration Research2013,8,1:2
4A new neural network model for the state-of-charge estimation in the battery degradation process 显示文摘LiuWang Kang Xuan Zhao Jian Ma 2014Applied Energy2014,121,:1
5Transient cerebral ischemia leads to TGF - β2 expression in golgi apparatus organdies 显示文摘Zhiping H Jie F Liuwang Z 2008Cur Neurovascul Res2008,5,3:1
6The role of the Golgi apparatus in oxidative stress: is this organelle less significant than mitochondria?显示文摘Zheng Jiang Zhiping Hu Liuwang Zeng Wei Lu Hainan Zhang Ting Li Han Xiao 2011Free Radical Biology and Medicine2011,,8:1
7Morphological Alteration of Golgi Apparatus and Subcellular Compartmentalization of TGF-β1 in Golgi Apparatus in Gerbils Following Transient Forebrain Ischemia显示文摘Zhiping Hu Liuwang Zeng Lesi Xie Wei Lu Jie Zhang Ting Li Xiang Wang 2007Neurochemical Research2007,,11:1
8Identification of potential nuclear reprogramming and differentiation factors by a novel selection method for cloning chromatin-binding proteins显示文摘Nuclear reprogramming is critical for animal cloning and stem cell creation through nuclear transfer, which requires extensive remodeling of chromosomal architecture involving dramatic changes in chromatin-binding proteins. To understand the mechanism of nuclear reprogramming, it is critical to identify chromatin-binding factors specify the reprogramming process. In this report, we have developed a high-throughput selection method, based on T7 phage display and chromatin immunoprecipitation, to isolate chromatin-binding factors expressed in mouse embryonic stem cells using primary mouse embryonic fibroblast chromatin. Seven chromatin-binding proteins have been isolated by this method. We have also isolated several chromatin-binding proteins involved in hepatocyte differentiation. Our method provides a powerful tool to rapidly and selectively identify chromatin-binding proteins. The method can be used to study epigenetic modification of chromatin during nuclear reprogramming, cell differentiation, and transdifferentiation.LiuWang AihuaZheng LingYi ChongrenXu MingxiaoDing HongkuiDeng 2005中国生物学文摘2005,19,4:0
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