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18篇 您的检索式：作者名="MILNES JT"

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		题名	作者	年代	出处	被引量
	1	Clemastine, a conventional antihistamine, is a high potency inhibitor of the HERG K + channel显示文摘	Ridley JM Milnes JT Hancox JC	2006	J Mol Cell Cardiol2006，40，：	1
	2	High affinity HERG K + channel blockade by the antiarrhythmic agent dronedaronc: resistance to mutations of the S6 residues Y652 and F656显示文摘	Ridley JM Milnes JT Witchel HJ	2004	Biochem Biophys Res Commun2004，325，3：	1
	3	High affin- ity HERG k + channel blockade by the antiarrhythmic agent dronedarone: resistance to mutations of the $6 residues Y652 and F656 显示文摘	Ridley JM Milnes JT Witchel HJ	2004	Biochem Biophys Res Commun2004，325，3：	1
	4	hERG K^+ channel blockade by the antipsychotic drug thioridazine: an obligatory role for the S6 helix residue F656 显示文摘	Milnes JT Witchel HJ Leaner JL	2006	Biochem Biophys Res Commun2006，351，：	1
	5	Blockade of HERG potassium currents by fluvoxamine: incompleteattenuation by S6 mutations at F656 or Y652显示文摘	MILNES JT CROCIANI O ARCANGELI A	2003	Br J Pharmacol2003，139，5：	1
	6	Preferential closed channel blockade of HERG potassium currentsby chemically synthesised BeKm-1 scorpion toxin显示文摘	MILNES JT DEMPSEY CE RIDLEY JM	2003	FEBS Lett2003，547，13：	1
	7	High affinityHERG K ( + ) channel blockade by the antiarrhythmie agent dronedarone : resistance to mutations of the S6 residues Y652 and F656 显示文摘	RIDLEY JM MILNES JT WITCHEL H J	2004	Biochem Biophys Res Commun2004，325，3：	1
	8	Reduced ryanodine receptor to dihydropyridine receptor ratio may underlie sliwed contraction in a rabbit model of left ventricular cardiac hypertaophy显示文摘	Milnes JT MacLeod KT	2001	J Mol Cell Cardiol2001，33，：	1
	9	High affinity HERG K ( + ) channel blockade by the antiarrhythmic agent dronedarone : resistance to mutations of the $6 residues Y652 and F656 显示文摘	RIDLEY JM MILNES JT WITCHEL HJ	2004	Biochem Biophys Res Commun2004，325，3：	1
	10	Reduced ryanodine receptor to dihydropyridine receptor ratio may underlie slowed contraction in a rabbit model of left ventricular cardiac hypertrophy 显示文摘	Milnes JT Macleod KT	2001	J Mol Cell Cardiol2001，33，：	1
	11	New drugs targeting the ca rdiac ultra -rapid delayed- rectifier current (I Kur): Rationale, pharmacology and evidence for potential therapeutic value 显示文摘	Ford JW Milnes JT	2008	J Cardiovasc Pharmaeol2008，52，2：	1
	12	New pharmacological approaches to atrial fibrillation显示文摘	Milnes JT Madge DJ Ford JW		0，，：	1
	13	Reduced ryanodine receptor to dihydropyridine recepor ratio may under lines lowed contraction in arabit model of left ventricular cardiac hypertrophy显示文摘	Milnes JT Mac Leod KT	2001	J Mol Cell Cardiol2001，33，3：	1
	14	New drugs targeting the cardiac ultra-rapid delayed-rectifier current (I Kur): Rationale,pharmacology and evidence for potential therapeutic value显示文摘	Ford JW Milnes JT	2008	J Cardiovasc Pharmacol2008，52，2：	1
	15	Troubleshooting problems with in vitro screening of drugs for QT interval prolonga- tion using HERG K + channels expressed in mammalian cell lines and Xenopus oocytes 显示文摘	Witchel HJ Milnes JT Mitcheson JS	2002	J Pharmacol Toxicol Methods2002，48，2：	1
	16	Reduced ryanodine receptor to dihydropyridine receptor ratio may underlie sliwed contraction in a rabbit model of left ventricular cardiac hypertaophy显示文摘	Milnes JT MacLeod KT	2001	J Mol Cell Cardiol2001，33，3：	1
	17	New drugs targeting the cardiacultra-rapid delayed-rectifier current (I Kur): rationale, pharmacology and evidence for potential therapeutic value显示文摘	Ford JW Milnes JT	2008	J Cardiovasc Pharmacol2008，52，2：	1
	18	New drugs targeting the cardiac ultra-rapid delayed-rectifi- er current(J Kur):rationale,pharmacology and evidence for potential thera- peutic value显示文摘	Ford JW Milnes JT	2008	J Cardiovasc Pharmacol2008，52，2：	1

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