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29篇 您的检索式:作者名="Matthess"
    题名 作者 年代 出处 被引量
1Persistence and transport of bacteria and viruses in groundwater-a conceptual evaluation 显示文摘Matthess G Pekdeger A Schroeter J 1955Journal of Contaminant Hydrology1955,2,2:1
2Thoughts on the current assessment of Polo-like kinase inhibitor drug discovery显示文摘Strebhardt K Becket S Matthess Y 2014Expert Opin Drug Discov2014,10,1:1
3Cdk1/Cyclin B1ControlsFas-Mediated Apoptosis by Regulating Caspase-8Activity显示文摘Matthess Y Raab M Sanhaji M 2010Mol Cell Biol2010,30,24:1
4Cancer inhibition in nude mice after systemic application of U6 promoter-driven short hairpin RNAs against PLKI显示文摘Spankuch B Matthess Y Knecht R 0,,11:1
5Stable gene silencing of cyclin B1 in tumor cells increases suscep tibility to taxol and leads to growth arrest in vivo显示文摘Yuan J Krmer A Matthess Y 2006Oncogene2006,25,12:1
6Free-living physical activity in COPD: Assessment with accelerometer and activity checklist显示文摘Moy Marilyn L Matthess Kirby Stolzmann Kelly Reilly John Garshick Eric 2009Journal of Rehabilitation Research and Development2009,,2:1
7Stable gene silencing of cyclin B1 in tumor cells increases susceptibility to taxol and leads to growth arrest in vivo显示文摘YUAN J KRAMER A MATTHESS Y 2006Oncogene2006,25,12:1
8Cancer inhibition in nude mice after systemic application of U6 promoter-driven short hairpin显示文摘Spankuch B Matthess Y Knecht R 2004J Natl Cancer Inst2004,96,11:1
9Stable gene silencing of cyclin B1 in tumour cells increases susceptibility to taxol and leads to growth arrest in vivo 显示文摘Yuan J Kramer A Matthess Y 2006Oncogene2006,25,12:1
10Silencing of mammalian genes by tetracycline-inducible shRNA expression 显示文摘Kappel S Matthess Y Kaufmann M 2007Nat Protoc2007,2,12:1
11Cancer inhibition in nude mice after systemic application of U6promoter-driven short hairpin RNAs against PLK1 显示文摘Spankuch B Matthess Y Knecht R 2004J Natl Cancer Inst2004,96,11:1
12pERK 1/2 inhibitcaspase - 8 induced apoptosis in cancer cells by phosphoryla-ting it in a cell cycle specific manner显示文摘Mandal R Raab M Matthess Y 2014Mol Oncol2014,8,2:1
13Stable gene silencing of cyclin B1 in tumor ceils increases susceptibility to taxol and leads to growth arrest in vivo 显示文摘Yuan J Krmer A Matthess Y 2006Oncogene2006,25,:1
14Silencing of mammalian genes by tetracycline-inducible shRNA expression 显示文摘Kappel S Matthess Y Kaufmann M 2007Nat Protoc2007,2,12:1
15Stable gene silencing of eyclin B1 in tumor cells increases susceptibility to taxol and leads to growth arrest in vivo显示文摘Yuan J Krmer A Matthess Y 2006Oncogene2006,25,12:1
16Conditional inhibition of cancer cell proliferation by tetracycline-responsive, H1 promoter- driven silencing of PLK1 显示文摘Matthess Y Kappel S Sptinkuch B 2005Oncogene2005,24,:1
17Tumor inhibition by genomicaUy integrated inducible RNAi-cassettes 显示文摘Kappel S Matthess Y Zimmer B 2006Nucleic Acids Res2006,34,:1
18Isotope signatures associated with early meteoric diagenesis 显示文摘ALLEN J R MATTHESS R K 1982Sedimentology1982,29,:1
19Ligand stimulation of CD95 induces activation of PIk3 followed by phosphorylation of caspase-8显示文摘在有它的 ligand 的 CD95 受体的相互作用之上,适配器分子 FADD (MORT1 ) 的顺序的协会, caspases-8/10,和 caspase-8/10 管理者 c 扭动支持形式导致导致死亡的发信号建筑群的形成。这里,我们识别像马球的 kinase (Plk ) 3 死亡受体 CD95 作为一个新相互作用合伙。Plk3 的酶的活动与它的 ligand 增加 CD95 受体的后面的相互作用。大美人(击倒) 或 caspase-8, CD95 或 FADD 击倒在 CD95 刺激之上阻止 Plk3 的激活,为 Plk3 激活建议一个功能的磁盘的一个要求。而且,我们为 Plk3 作为新底层识别 caspase-8。Phosphorylation 在 T273 上发生并且导致 caspase-8 proapoptotic 功能的刺激。在在一个营救实验或在 CRISPR/Cas9 产生的 Plk3 击倒房间表示 non-phosphorylatable caspase-8-T273A 异种的房间的 CD95 的刺激减少显著地处理 caspase-8。低 T273 phosphorylation 在 95 个肛门肿瘤病人的一个队与低 Plk3 表示显著地相关。我们的数据在 Plk 家庭以内建议 kinase 激活的新奇机制并且与高 Plk3 在肿瘤为外来的死亡小径的刺激建议一个新模型表示。Christina Helmke Monika Raab Franz Rodel Yves Matthess Thomas Oellerich Ranadip Mandal Mourad Sanhaji Henning Urlaub Claus Rodel Sven Becker Klaus Strebhardt 2016Cell Research2016,26,8:1
20Tumor inhibition by genomically integrated inducible RNAi-cassettes 显示文摘Kappel S Matthess Y Zimmer B 2006Nucleic Acids Res2006,34,16:1
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