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7篇 您的检索式:作者名="Neal Olson"
    题名 作者 年代 出处 被引量
1A multi-center prospective cohort study of benign breast diseaseand risk of subsequent breast cancer显示文摘Geoffrey CKabat Joan GJones Neal Olson 2010Cancer Causes Control2010,21,23:1
2Self - esteem and Counterfactual Thinking显示文摘Neal J Rome James M Olson 1993Journal of Personality and Social Psychology1993,65,1:1
3Identification of metabolites of 2,3,7,8- tetrachlorodibenzo-p-dioxin (TCDD) formed on incubation with isolated rat hepatocytes 显示文摘Sawahata T Olson J R Neal R A 1982Biochem Biophys Res Commun1982,105,1:1
4A multi-center prospective cohort study of benign breast disease and risk of subsequent breast cancer显示文摘Geoffrey C Kabat Joan G Jones Neal Olson 2010Cancer Causes Control2010,21,14:1
5Energy level alignment in PCDTBT:PC 70 BM solar cells: Solution processed NiO x for improved hole collection and efficiency显示文摘Erin L. Ratcliff Jens Meyer K. Xerxes Steirer Neal R. Armstrong Dana Olson Antoine Kahn 2012Organic Electronics2012,,5:1
6Factors associated with success of telaprevir-and boceprevir-based triple therapy for hepatitis C virus infection显示文摘AIM To evaluate new therapies for hepatitis C virus(HCV), data about real-world outcomes are needed.METHODS Outcomes of 223 patients with genotype 1 HCV who started telaprevir-or boceprevir-based triple therapy(May 2011-March 2012) at the Mount Sinai Medical Center were analyzed. Human immunodeficiency viruspositive patients and patients who received a liver transplant were excluded. Factors associated with sustained virological response(SVR24) and relapse were analyzed by univariable and multivariable logistic regression as well as classification and regression trees. Fast virological response(FVR) was defined as undetectable HCV RNA at week-4(telaprevir) or week-8(boceprevir). RESULTS The median age was 57 years, 18% were black, 44% had advanced fibrosis/cirrhosis(FIB-4 ≥ 3.25). Only 42%(94/223) of patients achieved SVR24 on an intention-totreat basis. In a model that included platelets, SVR24 was associated with white race [odds ratio(OR) = 5.92, 95% confidence interval(CI): 2.34-14.96], HCV sub-genotype 1b(OR = 2.81, 95%CI: 1.45-5.44), platelet count(OR = 1.10, per x 104 cells/μL, 95%CI: 1.05-1.16), and IL28 B CC genotype(OR = 3.54, 95%CI: 1.19-10.53). Platelet counts > 135 x 103/μL were the strongest predictor of SVR by classification and regression tree. Relapse occurred in 25%(27/104) of patients with an end-oftreatment response and was associated with non-FVR(OR = 4.77, 95%CI: 1.68-13.56), HCV sub-genotype 1a(OR = 5.20; 95%CI: 1.40-18.97), and FIB-4 ≥ 3.25(OR = 2.77; 95%CI: 1.07-7.22). CONCLUSION The SVR rate was 42% with telaprevir-or boceprevirbased triple therapy in real-world practice. Low platelets and advanced fibrosis were associated with treatment failure and relapse.Kian Bichoupan Neeta Tandon Valerie Martel-Laferriere Neal M Patel David Sachs Michel Ng Emily A Schonfeld Alexis Pappas James Crismale Alicia Stivala Viktoriya Khaitova Donald Gardenier Michael Linderman William Olson Ponni V Perumalswami Thomas D Schiano Joseph A Odin Lawrence U Liu Douglas T Dieterich Andrea D Branch 2017World Journal of Hepatology2017,9,11:0
7Social-epigeneticmediators for racial disparities in pulmonary impairment among childhood cancer survivors显示文摘Dear Editor Previous research found that childhood cancer survivors of African ancestry have significantly higher morbidity and mortality than those of European ancestry[1].However,after adjusting for socio-economic factors,the magnitudes of racial health disparities are either substantially decreased or become statistically non-significant[1],suggesting that social and economic determinants may contribute to racial health disparities.Recently,we conducted epigenome-wide association studies(EWAS)for three key social determinants of health(SDOHs),namely,personal educational attainment,personal income,and neighborhood deprivation among survivors of childhood cancer,where 130 epigenome-wide significant SDOH-CpG associationswere identified among European ancestry survivors,and 25 of which were also validated in African ancestry survivors[2].Notably,many SDOH-associated CpG sites are also associated with tobacco use.Nan Song Qian Dong Cheng Chen Qian Li Heather Mulder Emily Plyler John Easton Emily Walker Scott Olson Geoffrey Neale Kevin RKrull Deo Kumar Srivastava Kirsten K.Ness Jinghui Zhang Melissa M.Hudson Leslie L.Robison I-Chan Huang Zhaoming Wang 2023Cancer Communications2023,43,2:0
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