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| 1 | Alterations of calcium metab- olism and of parathyroid function in primary aldosteronism, and their reversal by spironolactone or by surgical removal of aldoste- rone-producing adenomas显示文摘 | Rossi E Sani C Perazzoli F | 1995 | Am J Hypertens1995，8，9： | 1 |
| 2 | Heart rate variability in patients with Sj6gren' s syndrome显示文摘 | Tumiati B Perazzoli F Negro A | 2000 | Clin Rheumatol2000，19，6： | 1 |
| 3 | Glutathione content and glutathione peroxidase expression in in vivo and in vitro matured equine oocytes显示文摘 | Luciano A M Goudet G Perazzoli F | 2006 | Mol Reprod Dev2006，73，5： | 1 |
| 4 | In vitro reproductive toxicity of polychlorinated biphenyls: effects on cccyte maturation and developmental competence in cattle 显示文摘 | Pocar P Perazzoli F Luciano AM | 2001 | Mol Reprod Dev2001，58，4： | 1 |
| 5 | Necrotizing lymphadenitis in systemic lupuserythematosus Kiikuchi′s disease or Kkikuchis-like disease显示文摘 | Tumiati B Casoli P Perazzoli F | 1999 | J ClinRheumatol1999，5，： | 1 |
| 6 | In vitro reproductive toxicity of polychlorinated biphenyls:effects on oocyte maturation and developmental ompetence in cattle显示文摘 | Perazzoli F Luciano AM | 2001 | Mol Reprod Dev2001，58，4： | 1 |
| 7 | - 344C/T polymorphism of (TYP11B2 gene in Italian patients with idiopathic low renin hypertension 显示文摘 | Rossi E Regolisti G Perazzoli F | 2001 | Am J Hypertens2001，14，91： | 1 |
| 8 | Glutathione content and glutathione peroxidase expression in in vivo and in vitro ma- tured equineoocytes显示文摘 | Luciano A M Goudet G Perazzoli F | 2006 | MolReprod Dev2006，73，5： | 1 |
| 9 | In vitro reproductive toxicity of polychlorinated biphenyls : effects on oocyte maturation and developmental competence in cattle 显示文摘 | Pocar P Perazzoli F Luciano AM | 2001 | Mol Reprod Dev2001，58，4： | 1 |
| 10 | In vitro reproductive tox-icity of polych-lorinated biphenyls:effects on oocyte maturation anddevelopmental competence in cattle显示文摘 | Pocar P Perazzoli F Luciano AM | 2001 | Mol Reprod Dev2001，58，4： | 1 |
| 11 | Alterations of calci- um metabolism and of parathyroid function in primary al- dosteronism, and their reversal by spironolactone or by surgical removal of aldosterone-producing adenomas 显示文摘 | Rossi E Sani C Perazzoli F | 1995 | Am J Hypertens1995，8，： | 1 |
| 12 | 344C/T polymorphism of CYP11B2 gene in Italian patients with idiopathic low rennin hypertension显示文摘 | Rossi E Regolisti G Perazzoli F | 2001 | Am J Hypertens2001，14，91： | 1 |
| 13 | Paclitaxel-loaded hollow-poly(4-vinylpyridine) nano- particles enhance drug chemotherapeutic efficacy in lung and breast cancer cell lines显示文摘Paclitaxel (PTX ) ,为胸和肺癌症的治疗的最有效的细胞毒素之一,被它的严重副作用和低肿瘤选择限制。在这个工作,( 4-vinylpyridine )( hollow-p4VP ) hollow-poly , nanoparticles ( NP )被用于第一次产生 PTX@p4VP NP ,采用在 NP 的中心的一个金核心是进一步的一种新奇在技术氧化了生产 PTX 分子能被合并到的空结构。当在血(没有溶血) 和房间文化(没有 cytotoxicity ) 上测试了时, hollow-p4VP NP 展出好物理化学的性质和显示的优秀 biocompatibility。有趣地, PTX@p4VP NP 显著地在人的肺(A-549 ) 和胸(MCF-7 ) 增加了 PTX cytotoxicity 有 PTX IC 50 的重要减小的癌症房间(从在 A-549 的 5.9 ~ 3.6 nM 并且从在 MCF-7 的 13.75 ~ 4.71 nM ) 。另外, PTX@p4VP 在 A-549 和 MCF-7 多细胞的肿瘤球状体(山) 的体积引起了减少,一在里面在 vivo 肿瘤模仿的 vitro 系统,与免费 PTX 相比。这项增加的 antitumoral 活动被有效房间成为主观伴随并且增加了 apoptosis,特别在我们的结果为 hollow-p4VP NP 能改进 PTX 的 antitumoral 活动的第一条证据提供的肺癌症山。这个系统能被用作新 nanoplatform 克服当前的胸和肺癌症治疗的限制。 | Rafael Contreras-Caceres Maria C. Leiva Raul Ortiz Amelia Diaz Gloria Perazzoli Miguel A.Casado-Rodriguez Consolacion Melguizo Jose M. Baeyens Juan M. Lopez-Romero Jose Prados | 2017 | Nano Research2017，10，3： | 1 |
| 14 | Necrotizing lymphadenitis in systemic lupus erythematosus kikuchi's disease or kikuchilike disease显示文摘 | Tumiati B Casoli P Perazzoli F | 1999 | J Clin Rheumatol1999，5，： | 1 |
| 15 | High prevalence of primary aldosteronism using postcaptopril plasma aldosterone to renin ratio as a screening test among Italian hypertensives显示文摘 | Ermanno Rossi Giuseppe Regolisti Aurelio Negro Carlo Sani Simona Davoli Franco Perazzoli | 2002 | American Journal of Hypertension2002，，10： | 1 |
| 16 | -344C/T polymorphism of CYP11B2 gene in Italian patients with idiopathic low renin hypertension显示文摘 | Rossi E Regolisti G Perazzoli F | 2001 | Am J Hypertens2001，14，91： | 1 |
| 17 | Neerotizing Lymphadeni- tis in Systemic Lupus Erythematosus Kikuchi' s Disease or a Ki- kuchi' s-Like Disease? 显示文摘 | Tumiati B Casoli P Perazzoli F | 1999 | J Clin Rheumatol1999，5，3： | 1 |
| 18 | The challenge of drug resistance in pancreatic ductal adenocarcinoma:a current overview显示文摘Pancreatic ductal adenocarcinoma(PDAC)has one of the highest mortality rates among all cancer types.Its delayed diagnosis precludes curative resection,thus most of the current therapies against PDAC are based on chemo-and radiotherapy.Unfortunately,these strategies are insufficient to improve its poor prognosis.Despite the advances made in chemotherapy(e.g.nab-paclitaxel and gemcitabine),many patients with PDAC are unable to benefit from them due to the rapid development of drug resistance.Currently,more than 165 genes have been found to be implicated in drug resistance of pancreatic tumors,including different integrins,mucins,NF-κB,RAS and CXCR4.Moreover,drug resistance in PDAC is thought to be mediated by the modulation of miRNAs(e.g.miRNA-21,miRNA-145 and miRNA-155),which regulate genes that participate in cell proliferation,invasion and metastasis.Finally,cancer stem cells are intimately related to drug resistance in PDAC due to their ability to overexpress ABC genes-involved in drug transport-,and enzymes such as aldehyde dehydrogenases-implicated in cellular drug metabolism-and poly(ADP-ribose)polymerases-involved in drug-induced DNA damage repair.Understanding the mechanisms involved in drug resistance will contribute to the development of efficient therapeutic strategies and to improve the prognosis of patients with PDAC. | Francisco Quinonero Cristina Mesas Kevin Doello Laura Cabeza Gloria Perazzoli Cristina Jimenez-Luna Ana Rosa Rama Consolación Melguizo Jose Prados | 2019 | Cancer Biology & Medicine2019，16，4： | 1 |
| 19 | Temozolomide resis- tance in glioblastoma cell lines: implication of MGMT, MMR, P-glycoprotein and CD133 expression 显示文摘 | Perazzoli G Prados J Ortiz R | | PLoS One0，10，01： | 1 |
| 20 | 一个新发现的患利德尔综合征家庭中,患者呈现似原发性高血压的临床表型显示文摘利德尔综合征(Liddle’s syndrome,LS)是一个拟似盐皮质激素过多的,由单基因异常造成的,表现为血压升高的综合征。目前在年轻低钾性高血压患者的鉴别诊断中考虑该疾病的存在。该研究的目的是:①评估一个新近发现的来自意大利西西里地区携带上皮钠通道P617L功能增强性13亚基的家族的临床表型。该研究组曾经报道在一个与新发现无关的西西里患者中存在这种基因突变并表现包括低血钾的典型LS症状。②确定新发现的家庭和先前发现家庭是否存在一种尚未明确的生物关系。 | Ermanno Rossi Enrico Farnetti Davide Nicoli Marco Sazzini Franco Perazzoli Giuseppe Regolisti Chiara Grasselli Rosaria Santi Aurelio Negro Vincenzo Mazzeo Franco Mantero Donata Luiselli Bruno Casali 罗冬梅 叶鹏 | 2012 | 中华高血压杂志2012，20，2： | 0 |
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