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371篇 您的检索式:作者名="Rumi"
    题名 作者 年代 出处 被引量
1Functions of Xyloglucan in Plant Cells显示文摘当 xyloglucan 的数字的增加在植物在纤维素 microfibrils 之间拴住时,房间围增加墙刚硬,这些的降级拴住原因松开的墙。降级也能由于 xyloglucanase 的行动由于 xyloglucan endotransglucosylase 或通过直接水解作用的行动通过 xyloglucan oligosaccharides 的集成发生。这是 xyloglucan 和它的碎片 oligosaccharides 的增加引起植物织物紧张那么戏剧性地增加并且减少的原因。包含 xyloglucanase 和绗维素酶的 overexpression 的实验在墙中揭示了 xyloglucans 的角色。在由 xyloglucanase 的转基因的表示的白杨的墙 xyloglucan 的降级,例如,不是仅仅在主要的墙中加速了茎延伸,而且在第二等的墙中堵住了正直茎的 gravitropism。当纤维素 microfibrils 在他们的非结晶的区域被整修,绗维素酶的 Overexpression 也在墙中减少了 xyloglucan 内容,导致在 Arabidopsis 叶子并且在有扰乱的叶运动的 sengon 的增加的房间体积。hemicellulose xyloglucan,在它是的功能一 tether,在松开并且紧缩纤维素 microfibrils 起一个关键作用:它使细胞能在生长和区别地区改变它的形状并且在细胞成熟以后保留它的最后的形状。Takahisa Hayashi Rumi Kaida 2011Molecular Plant2011,4,1:14
2Xyloglucan for Generating Tensile Stress to Bend Tree Stem显示文摘响应环境变化,被子植物树由形成紧张木头把他们的茎弄弯,它由充满纤维素的 G 组成(凝胶状) 在纤维房间的墙中的层并且在第二等的木部产生反常张力的应力。我们生产了转基因的白杨植物 overexpressing 在房间墙中减少每特定的多糖,,的几 endoglycanases 第二等的木部由主要、第二等的墙层组成。当水平地放了时,转基因的白杨 overexpressing xyloglucanase 的茎的基础区域不能独自在木部的紧张方面由于低紧张向上弯曲。在野类型的植物, xyloglucan 在多重 layering 期间在 G 层的内部表面被发现。在 situ xyloglucan endotransglucosylase ( XET ),活动证明整个 xyloglucan 的加入,潜在地因为墙紧缩,在内部表面层 S1 和 S2 开始了并且在整个G层开发被保留,当为墙松开的 xyloglucan heptasaccharide ( XXXG )的加入发生在膨胀地区的主要的墙中时。我们建议 xyloglucan 网络被 XET 增强形成在墙界限之间的一个进一步的连接并且分泌了 xyloglucans 以便承受在纤维素 G 层 microfibrils 以内创造的张力的压力。Kei'ichi Baba Yong Woo Park Tomomi Kaku Rumi Kaida Miyuki Takeuchi Masato Yoshida Yoshihiro Hosoo Yasuhisa Ojio Takashi Okuyama Toru Taniguchi Yasunori Ohmiya Teiji Kondo Ziv Shani Oded Shoseyov Tatsuya Awano Satoshi Serada Naoko Norioka Shigemi Norioka Takahisa Hayashi 2009Molecular Plant2009,2,5:4
3Breast cancer as photodynamic therapy target: Enhanced therapeutic efficiency by overview of tumor complexity显示文摘Photodynamic therapy is a minimally invasive and clinically approved procedure for eliminating selected malignant cells with specific light activation of a photosensitizer agent. Whereas interstitial and intra-operative approaches have been investigated for the ablation of a broad range of superficial or bulky solid tumors such as breast cancer, the majority of approved photodynamic therapy protocols are for the treatment of superficial lesions of skin and luminal organs. This review article will discuss recent progress in research focused mainly on assessing the efficacies of various photosensitizers used in photodynamic therapy, as well as the combinatory strategies of various therapeutic modalities for improving treatments of parenchymal and/or stromal tissues of breast cancer solid tumors. Cytotoxic agents are used in cancer treatments for their effect on rapidly proliferating cancer cells. However, such therapeutics often lack specificity, which can lead to toxicity and undesirable side effects. Many approaches are designed to targettumors. Selective therapies can be established by focusing on distinctive intracellular(receptors, apoptotic pathways, multidrug resistance system, nitric oxidemediated stress) and environmental(glucose, pH) differences between tumor and healthy tissue. A rational design of effective combination regimens for breast cancer treatment involves a better understanding of the mechanisms and molecular interactions of cytotoxic agents that underlie drug resistance and sensitivity.María Julia Lamberti Natalia Belén Rumie Vittar Viviana Alicia Rivarola 2014World Journal of Clinical Oncology2014,5,5:3
4Etiology-related determinants of liver stiffness values in chronic viral hepatitis B or C显示文摘Mirella Fraquelli Cristina Rigamonti Giovanni Casazza Maria Francesca Donato Guido Ronchi Dario Conte Mariagrazia Rumi Pietro Lampertico Massimo Colombo 2010Journal of Hepatology2010,,4:3
5A 28-Year Study of the Course of Hepatitis Δ Infection: A Risk Factor for Cirrhosis and Hepatocellular Carcinoma显示文摘Raffaella Romeo Ersilio Del Ninno Mariagrazia Rumi Antonio Russo Angelo Sangiovanni Roberto de Franchis Guido Ronchi Massimo Colombo 2009Gastroenterology2009,,5:2
6Role of tumor-associated macrophages in the progression of hepatocellular carcinoma显示文摘Ken Shirabe Yohei Mano Jun Muto Rumi Matono Takashi Motomura Takeo Toshima Kazuki Takeishi Hidekaki Uchiyama Tomoharu Yoshizumi Akinobu Taketomi Masaru Morita Shunichi Tsujitani Yoshihisa Sakaguchi Yoshihiko Maehara 2012Surgery Today2012,,1:2
7Acceleration of Cell Growth by Xyloglucan Oligosaccharides in Suspension-Cultured Tobacco Cells显示文摘进暂停有教养的烟草房间的墙的 xyloglucan oligosaccharide (XXXG ) 的加入加速了房间分割跟随的房间扩大,改变的房间形状从对球形的、减少的房间尺寸,和引起的房间聚集圆柱。荧光灯在强壮的加入发生在表面上的地方,加到文化媒介的 XXXG 被发现被合并到全部墙的表面不仅,而且在在房间之间的接口墙中在房间分割期间。房间扩大比在纵的方向在横向的方向总是是更大的然后立即,扩大面对 XXXG 导致了房间分割;这个过程可能导致在有教养的烟草房间看见的房间聚集的高水平。我们断定进墙的这 oligosaccharide 的集成能在有教养的房间加速不仅房间扩大,而且房间分割。Rumi Kaid Satoko Sugawara Kanako Negoro Hisae Maki Takahisa Hayashi Takako S. Kaneko 2010Molecular Plant2010,3,3:2
8Energy consumption, real income and temporal causality: results from a multi-country study based on cointegration and error-correction modelling techniques显示文摘Abul M.M. Masih Rumi Masih 1996Energy Economics1996,,3:2
9Involvement of serum retinoids and Leiden mutation in patients with esophageal, gastric, liver, pancreatic, and colorectal cancers in Hungary显示文摘AIM: To analyze the serum levels of retinoids and Leiden mutation in patients with esophageal, gastric, liver,pancreatic, and colorectal cancers.METHODS: The changes in serum levels of retinoids (vitamin A, α- and β-carotene, α- and β-cryptoxanthin,zeaxanthin, lutein) and Leiden mutation were measured by high liquid performance chromatography (HPLC)and polymerase chain reaction (PCR) in 107 patients (70 males/37 females) with esophageal (0/8), gastric (16/5), liver (8/7), pancreatic (6/4), and colorectal (30/21including 9 patients suffering from in situ colon cancer)cancer. Fifty-seven healthy subjects (in matched groups)for controls of serum retinoids and 600 healthy blood donors for Leiden mutation were used.RESULTS: The serum levels of vitamin A and zeaxanthin were decreased significantly in all groups of patients with gastrointestinal (GI) tumors except for vitamin A in patients with pancreatic cancer. No changes were obtained in the serum levels of α- and β-carotene,α- and β-cryptoxanthin, zeaxanthin, lutein in patients with GI cancer. The prevalence of Leiden mutation significantly increased in all groups of patients with GI cancer.CONCLUSION: Retinoids (as environmental factors)are decreased significantly with increased prevalence of Leiden mutation (as a genetic factor) in patients before the clinical manifestation of histologically different (planocellular and hepatocellular carcinoma, and adenocarcinoma) GI cancer.Gyula Mózsik Gy(o|¨)rgy Rumi András D(o|¨)m(o|¨)t(o|¨)r Mária Figler Beáta Gasztonyi El(?)d Papp Alajos Pár Gabriella Pár József Belágyi Zoltán Matus Béla Melegh 2005World Journal of Gastroenterology2005,11,48:2
10Loosening Xyloglucan Accelerates the Enzymatic Degradation of Cellulose in Wood显示文摘以便创造在纤维素的树,在生物资源的最丰富的部件,能是酶地 hydrolyzed 高度为 bioethanol 的生产,我们从几棵转基因的白杨,每 overexpressing 任何一个 xyloglucanase,绗维素酶, xylanase,或 galactanase 检验了木部的 saccharification。绗维素酶准备完成的纤维素降级的水平比在野类型的植物的木部在木部 overexpressing xyloglucanase 和在木部 overexpressing xylanase 和绗维素酶更大的大部分是显著地更大的。尽管降级的高度根本发生在所有木部 loci,纤维素 microfibrils 的水晶的区域高度在木部 overexpressing xyloglucanase 被降级。因为在 microfibrils 和 xyloglucans 之间的建筑群能是对纤维素降级特别地抵抗的一个区域,松开 xyloglucan 能在木头便于纤维素的酶的水解作用。Rumi Kaida Tomomi Kaku Kei'ichi Baba Masafumi Oyadomari Takashi Watanabe Koji Nishida Toshiji Kanaya Ziv Shani Oded Shoseyov Takahisa Hayashi 2009Molecular Plant2009,2,5:2
11Localization from connectivity in sensor networks 显示文摘Shang Y Rumi W Zhang Y 2004IEEE Transactions on Parallel and Distributed Systems2004,15,11:1
12The metabolism and n-6/n-3 ratio of essential fatty acids in rats:effect of dietary arachidonic acid and a mixture of sesame lignans(sesamin and episesamin ) 显示文摘 Megumi Ogawa Osamu lgarashi 1998Lipids1998,33,6:1
13Hallervorden- Spatz dis ease:clinical and MRI study of 11 cases diagnosed in life显示文摘 NARDOCCI N RUMI V 1992J Neurol1992,239,:1
14Effect of sesamin on mitochondrial and peroxisomal β-oxidation of arachidonic and eicosapentaenoic aicds liver显示文摘Rumi Umeda- Sawada Megumi Ogawa Mayu- ymi Nakamura 2001Lipids2001,36,5:1
15Clinical effect of driver mutations of JAK2, CALR, or MPL in primary myelofibrosis 显示文摘Rumi E Pietra D Pascutto C 2014Blood2014,124,7:1
16Long-term titrated recombinant interferon-alpha 2a in chronic hepatitis C: a randomized controlled trial显示文摘Rumi M G 1995Viral Hepatol1995,2,2:1
17JAK2 or CALR mutation status defines subtypes of essentialthrombocythemia with substantially different clinical course and outcomes显示文摘Rumi E Pietra D Ferretti V 2014Blood2014,123,10:1
18Deep sequencing reveals double mutations in cis of MPL exon 10 in myeloproliferative neoplasms显示文摘Pietra D Brisci A Rumi E 0,,04:1
19Aspirin in pregnant pa- tients with essential thrombocythemia: a retrospective analysis of 129 pregnancies显示文摘Passamonti F Rumi E Randi M L 2010J Thromb Haemost2010,8,2:1
20Prognostic factors for thrombosis, myelofibrosis, and leukemia in essential thrombocy- themia: a study of 605 patients显示文摘Passamonti F Rumi E Arcaini L 2008Haematologica2008,93,11:1
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