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| 1 | 球孢白僵菌对油桐尺蛾的生物效力(英文)显示文摘分别在2005年和2006年的5、6月份,在Kamalpur和Hunterpara茶园、孟加拉西部以及印度地区对一种昆虫致病真菌球孢白僵菌对油桐尺蛾Buzura(=Biston)suppressaria Guen.的生物效力进行了评估。以农场主常用的化学杀虫剂40%SP灭多虫和25%EC氯氰菊酯作为标准检测物。试验中球孢白僵菌浓度为1.50 g,2.00 g和2.5 g/lit.of water;灭多虫和氯氰菊酯浓度分别为0.75 mL,1.00 mL和1.50 mL,和0.50 mL,1.00 mL和1.50 mL/lit.of water。在喷洒2.5 g/lit.of water球孢白僵菌3天后,Kamalpur和Hunterpara茶园中油桐尺蛾种群个体数分别降低了88.00%和84.00%。死的毛虫变成黑色,垂悬在叶片上。并且发现球孢白僵菌的杀虫活性与高剂量的灭多虫和氯氰菊酯的相当。 | Sibani Sankar GHATAK Md. Wasim REZA | 2007 | 昆虫学报2007，50，9： | 9 |
| 2 | A Second Genetic Polymorphism in Methylenetetrahydrofolate Reductase (MTHFR) Associated with Decreased Enzyme Activity显示文摘 | Ilan Weisberg Pamela Tran Benedicte Christensen Sahar Sibani Rima Rozen | 1998 | Molecular Genetics and Metabolism1998，，3： | 2 |
| 3 | Faster monte carlo simulations at low temperatures: The waiting time method显示文摘 | Dall J Sibani P | 2001 | Computer Physics Communication2001，141，2： | 1 |
| 4 | Comparing extremal and thermal explorations of energy landscapes 显示文摘 | Boettcher S Sibani P | 2005 | European Physics J2005，44，3： | 1 |
| 5 | Characterization of six novel mutations in the methylenetetrahydrofolate reductase (MTHF ) gene fn patients with homocystinuria显示文摘 | Sibani S Ohristensen B O'Ferra]l E | 2012 | um Murat2012，15，： | 1 |
| 6 | Characterization of six novel mutations in the methylenetetrahydrofolate reductase (MTHFR) gene in patients with homocystinuria显示文摘 | Sibani S Christensen B O'Ferrall E | 2000 | Hum Mutat2000，15，3： | 1 |
| 7 | Effects of chelating agents EDTA,phytin and deferoxamine on the heavy metal complexes of DNA显示文摘 | Sibani S Sandhya D Medini K P | 1999 | Journal of the Indian Chemical Society1999，76，1： | 1 |
| 8 | Characterization of six novel mutations in the methylenetetrahydrofolate reductase (MTHFR) gene in patients with homocystinuria显示文摘 | Sibani S Christensen B Ferrall EO | 2000 | Hum Mutat2000，15，3： | 1 |
| 9 | Characterization of mutations in severe methylenetetrahydrofolate reductase deficiency reveals an FAD-responsive mutation显示文摘 | Sibani S Leclerc D Weisberg IS | 2003 | Hum Mutat2003，21，5： | 1 |
| 10 | A second genetic polymorphism in methylenetetrahydrofolate reductase(MTHFR) associated With decreased enzyme activity显示文摘 | Weisberg I Tran P Christensen B Sibani S Rozen R | 1998 | Mol Genet Metab1998，64，： | 1 |
| 11 | Characterization of mutations in severe methylenetetrahydrofolate reductase deficiency reveals an FAD-responsive mutation显示文摘 | Leclerc D Weisberg IS | 2003 | Hum Mutat2003，21，： | 1 |
| 12 | Characterization of six novel mutations in the methylenetet rahydrofolate reductase (MTHFR) gene in patients with homocystinuria显示文摘 | SIBANI S CHRISTENSEN B FERRALL EO | | 0，，03： | 1 |
| 13 | Relaxation in complex systems:Local minima and their exponents显示文摘 | Sibani P Hoffmann K H | 1991 | Europhysics Letters1991，16，5： | 1 |
| 14 | Impact of Dnmtl deficiency, with and without low folate diets, on tumor numbers and DNA methylation in Min mice显示文摘 | Trasler J Deng L Melnyk S Pogribny I Hiou - Tim F Sibani S Oakes C Li E James SJ Rozen R | 2003 | Carcinogenesis2003，24，： | 1 |
| 15 | Relaxation in Complex Systems:Local Minima and Their Exponents显示文摘 | Sibani P Hoffmann K H | 1991 | Europhysics Letter1991，16，5： | 1 |
| 16 | The interrelationship between cerebral ischemic stroke and glioma: a comprehensive study of recent reports显示文摘Glioma and cerebral ischemic stroke are two major events that lead to patient death worldwide.Although these conditions have different physiological incidences,~10%of ischemic stroke patients develop cerebral cancer,especially glioma,in the postischemic stages.Additionally,the high proliferation,venous thrombosis and hypercoagulability of the glioma mass increase the significant risk of thromboembolism,including ischemic stroke.Surprisingly,these events share several common pathways,viz.hypoxia,cerebral inflammation,angiogenesis,etc.,but the proper mechanism behind this co-occurrence has yet to be discovered.The hypercoagulability and presence of the D-dimer level in stroke are different in cancer patients than in the noncancerous population.Other factors such as atherosclerosis and coagulopathy involved in the pathogenesis of stroke are partially responsible for cancer,and the reverse is also partially true.Based on clinical and neurosurgical experience,the neuronal structures and functions in the brain and spine are observed to change after a progressive attack of ischemia that leads to hypoxia and atrophy.The major population of cancer cells cannot survive in an adverse ischemic environment that excludes cancer stem cells(CSCs).Cancer cells in stroke patients have already metastasized,but early-stage cancer patients also suffer stroke for multiple reasons.Therefore,stroke is an early manifestation of cancer.Stroke and cancer share many factors that result in an increased risk of stroke in cancer patients,and vice-versa.The intricate mechanisms for stroke with and without cancer are different.This review summarizes the current clinical reports,pathophysiology,probable causes of co-occurrence,prognoses,and treatment possibilities. | Mrinal K.Ghosh Dipankar Chakraborty Sibani Sarkar Arijit Bhowmik Malini Basu | 2019 | Signal Transduction and Targeted Therapy2019，4，1： | 0 |
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