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| 1 | Desflu- rane but not sevoflurane can increase lung resistance via tachykinin pathways 显示文摘 | SATOH JI YAMAKAGE M KOBAYASHI T | 2009 | Br J Anaesth2009，102，5： | 1 |
| 2 | Cytokines and neurotrophic factors fail to affect Nogo-A mRNA expression in differentiated human neurons:Implications for inflammation-related axonal regeneration in the central nervous system显示文摘 | Satoh JI Kuroda Y | 2002 | Neuropathol Appl Neurobiol2002，28，2： | 1 |
| 3 | Inhibitory effects of the al- pha-2 adrenergie agonists elonidine and dexmedetomidine on enhanced airway tone in ovalbumin-sensitized guinea pigs 显示文摘 | Yamakage M Iwasaki S Satoh JI | 2008 | Eur J Anaesthesiol2008，25，1： | 1 |
| 4 | Differential expression of heat shock protein 27 in human neurons and glial cells in culture显示文摘 | Satoh JI | 1995 | J Neurosci Res1995，41，6： | 1 |
| 5 | Molecular network of microRNA targets in Alzhei- mer' s disease brains显示文摘 | Satoh JI | 2012 | Exp Neurol2012，235，2： | 1 |
| 6 | Cytokines and neurotrophic factors fail to affect Nogo-A mRNA expression in differentiated human neurons: Implications for inflammation-related axonal regeneration in the central nervous system 显示文摘 | Satoh JI Kuroda Y | 2002 | Neuropathol Appl Neurobiol2002，28，2： | 1 |
| 7 | Nogo-A and Nogo receptor expression in demyelinating lesions of multiple sclerosis显示文摘 | Satoh JI Onoue H Arima K | 2005 | J Neuropatho Exp Neurol2005，64，2： | 1 |
| 8 | Detection of Anti- Nogo Receptor Autoantibody in the Serum of Multiple Sclero- sand Controls显示文摘 | ONOUE H SATOH JI OGAWA M | 2007 | Acta Neurol Scand(S0001-6314)2007，115，3： | 1 |
| 9 | Differential expression of heat shock protein HSP27 in human neurons and glial cells in culture显示文摘 | Satoh JI Kim SU | 1995 | J Neurosci Res1995，41，6： | 1 |
| 10 | A Ds-insertion mutant of OSH6 (Oryza sativa Homeobox 6) exhibits outgrowth of vestigial leaf-like structures, bracts, in rice显示文摘 | Sung Han Park Chul Min Kim Byoung Il Je Su Hyun Park Soon Ju Park Hai Long Piao Yuan-hu Xuan Mi Sook Choe Kouji Satoh Shoshi Kikuchi Kon Ho Lee Young Soon Cha Byung Ohg Ahn Hyeon So Ji Doh Won Yun Myung Chul Lee Seok-cheol Suh Moo Young Eun Chang-deok Han | 2007 | Planta2007，，1： | 1 |
| 11 | Nogo-A and nogo receptor expression in demyelinating lesions of multiple sclerosis显示文摘 | Satoh JI Onoue H Arima K | 2005 | J Neuropathol Exp Neurol2005，64，2： | 1 |
| 12 | Superwoman1-cleistogamy, a hopeful allele for gene containment in GM rice显示文摘 | Yoshida H Itoh JI Ohmori S Miyoshi K Horigome A Uchida E Kimizu M Matsumura Y Kusaba M Satoh H Nagato Y | 2007 | Plant Biotechnology Journal2007，5，6： | 1 |
| 13 | Association of codon 167 Ser/Asn heterozygosity in the parkin gene with sporadic Parkinson's disease显示文摘 | Satoh JI Kuroda Y | 1999 | Neuroreport1999，10，13： | 1 |
| 14 | Different inhibitory effects of sevoflurane on hyperreactive airway smooth muscle contractility in ovalbumin-sensitized and chronic cigarette-smoking guinea pig models 显示文摘 | Iwasaki S Yamakage M Satoh JI | 2006 | Anesthesiology2006，105，4： | 1 |
| 15 | Relationship between response to lusutrombopag and splenic volume显示文摘AIM To assess the correlation between the efficacy of lusutrom-bopag and clinical characteristics in patients with chronic liver disease.METHODS In this retrospective, multicenter study, which conducted at four locations in Japan, 50 thrombocytopenic patients with chronic liver disease were enrolled. All patients received oral lusutrombopag(3.0 mg/d for 7 d) for chronic liver disease. We assessed the increase in platelet count after the trial drug administration. A treatment response was defined as a platelet count ≥ 5 × 104/μL and an increased platelet count ≥ 2 × 104/μL from baseline after drug administration. We evaluated the response to lusutrombopag compared to baseline clinical characteristics in patients with chronic liver disease.RESULTS The numbers of responders and non-responders were 40(80.0%) and 10(20.0%), respectively. The patients were divided into a responder and non-responder group, and we added factors that may correspond to successful treatment with lusutrombopag. Splenic volume and body weight were lower in the responder group than in the nonresponder group. White blood cell count and hemoglobin level were higher in responders compared with nonresponders. Using a logistic regression model to assess the relationship between response to lusutrombopag and clinical characteristics, multivariate analysis confirmed that splenic volume was an independent factor that predicted the response of platelet counts(P = 0.025; odds ratio = 11.2; 95% confidence interval: 1.354-103.0). Splenic volume negatively correlated to changes in platelet count(r =-0.524, P = 0.001).CONCLUSION Splenic volume influences the change in platelet counts after administration of lusutrombopag in patients with chronic liver disease. | Haruki Uojima Yoshitaka Arase Norio Itokawa Masanori Atsukawa Takashi Satoh Koji Miyazaki Hisashi Hidaka Ji Hyun Sung Makoto Kako Kota Tsuruya Tatehiro Kagawa Katsuhiko Iwakiri Ryouichi Horie Wasaburo Koizumi | 2018 | World Journal of Gastroenterology2018，24，46： | 1 |
| 16 | Detection of anti-Nogo recep-tor autoantibody in the serum of multiple sclerosis and controls 显示文摘 | Onoue H Satoh JI Ogawa M | 2007 | Acta Neurol Scand2007，115，： | 1 |
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