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| 1 | Effects of treatment with sulfonlurea drugs or insulin on ischemia induced myocardial dysfunctionin type 2diabetes 显示文摘 | Scognamiglio Roldano Avogaro Angelo Vigili-de-Kreutzenberg Saula | 2002 | Diabetes2002，51，： | 1 |
| 2 | Endothelial dysfunction in type 2 diabetes mellitus显示文摘 | Angelo Avogaro Gian Paolo Fadini Alessandra Gallo Elisa Pagnin Saula de Kreutzenberg | 2005 | Nutrition Metabolism and Cardiovascular Diseases2005，，： | 1 |
| 3 | Pro-inflammatory monocyte-macrophage polarization imbalance in human hypercholesterolemia and atherosclerosis显示文摘 | Gian Paolo Fadini Francesco Simoni Roberta Cappellari Nicola Vitturi Silvia Galasso Saula Vigili de Kreutzenberg Lorenzo Previato Angelo Avogaro | 2014 | Atherosclerosis2014，，2： | 1 |
| 4 | Sirtuin 1 stabilization by HuR represses TNF-ct and glucose-induced E-selectin release and endothelial cell adhesiveness in vitro: relevance to human metabolic syndrome 显示文摘 | Giulio C Saula Vigili DK Arianna C | 2014 | Clinical Science2014，127，7： | 1 |
| 5 | Insulin signaling and life span 显示文摘 | Angelo Avogaro Saula Vigili de Kreutzenberg Gian Paolo Fadini | 2010 | Pflugers Arch Eur J Physiol2010，459，： | 1 |
| 6 | Gender differences in endothelial progenitor cells and cardiovascular risk profile:the role of female estrogens显示文摘 | Paolo FG Saula DK Mattia A | 2008 | Arteriosclerosis Thrombosis & Vascular Biology2008，，： | 1 |
| 7 | Characteristics and outcomes of the hyperglycemic hyperosmolar non-ketotic syndrome in a eohort of 51 consecutive cases at a single center显示文摘 | Gian PF Saula VK Mauro R | 2011 | Diabetes Research and Clini- cal Practice2011，94，2： | 1 |
| 8 | Endothelial dysfunction: Causes and con- sequences in patients with diabetes mellitus Original Research Article显示文摘 | ANGELO AVOGARO SAULA VIGILI DE KREUTZENBERG G1ANPAOLO FADINI | 2008 | Diabetes Research and Clinical Practice2008，82，2： | 1 |
| 9 | Hypertrophic pyloric stenosis in the Third World显示文摘 | Saula PW Hadley GP | 2011 | Trop Doct2011，41，4： | 1 |
| 10 | Mechanisms of endothelial dysfunc-tion in obesity 显示文摘 | ANGELO A SAULA V K | 2005 | Clin Chimica Acta2005，360，12： | 1 |
| 11 | Mechanisms of endothelial dysfunc- tion in obesity显示文摘 | Angelo A Saula VK | 2005 | Clinica Chimica Acta2005，360，12： | 1 |
| 12 | Notch signaling in female cancers: a multifaceted node to overcome drug resistance显示文摘Drug resistance is one of the main challenges in cancer therapy,including in the treatment of female-specific malignancies,which account for more than 60%of cancer cases among women.Therefore,elucidating the underlying molecular mechanisms is an urgent need in gynecological cancers to foster novel therapeutic approaches.Notably,Notch signaling,including either receptors or ligands,has emerged as a promising candidate given its multifaceted role in almost all of the hallmarks of cancer.Concerning the connection between Notch pathway and drug resistance in the afore-mentioned tumor contexts,several studies focused on the Notch-dependent regulation of the cancer stem cell(CSC)subpopulation or the induction of the epithelial-to-mesenchymal transition(EMT),both features implicated in either intrinsic or acquired resistance.Indeed,the present review provides an up-to-date overview of the published results on Notch signaling and EMT-or CSC-driven drug resistance.Moreover,other drug resistance-related mechanisms are examined such as the involvement of the Notch pathway in drug efflux and tumor microenvironment.Collectively,there is a long way to go before every facet will be fully understood;nevertheless,some small pieces are falling neatly into place.Overall,the main aim of this review is to provide strong evidence in support of Notch signaling inhibition as an effective strategy to evade or reverse resistance in female-specific cancers. | Maria V.Giuli Angelica Mancusi Eugenia Giuliani Isabella Screpanti Saula Checquolo | 2021 | Cancer Drug Resistance2021，4，4： | 0 |
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