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2篇 您的检索式:作者名="Sera Yu"
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1Water stable and matrix addressable OLED fiber textiles for wearable displays with large emission area显示文摘Organic light-emitting diode(OLED)fibers with favorable electroluminescence properties and interconnectable pixel configurations have represented the potential for wearable electronic textile displays.Nevertheless,the current technology of OLED fiber-based textile displays still leaves to be desired due to several challenges,including limited emission area and lack of encapsulation systems.Here we present a fibrous OLED textile display that can attain a large emission area and long-term stability by implementing addressable networks comprised of integrated phosphorescence OLED fibers and by designing multilayer encapsulations.The integrated fiber configuration offers decoupled functional fiber surfaces for an interconnectable 1-dimensional OLED pixel array and a data-addressing conductor.Tailored triadic metal/ultrathin oxide/polymer multilayer enables not only the oxygen/water permeation inhibition but also the controllable conductive channels of dielectric antifuses.Together with reliable bending stability,the long-term operation of OLED textiles in water manifests the feasibility of the present device concept toward water-resistant full-emitting-area fibrous textile displays.Hayoung Song Young Jin Song Jinwook Hong Ki Suk Kang Sera Yu Ha-Eun Cho Jae-Hun Kim Sung-Min Lee 2022npj Flexible Electronics2022,6,1:0
2A plasma membrane nucleotide-binding leucinerich repeat receptor mediates the recognition of the Ralstonia pseudosolanacearum effector RipY in Nicotiana benthamiana显示文摘Bacterial wilt disease caused by several Ralstonia species is one of the most destructive diseases in Solanaceae crops.Only a few functional resistance genes against bacterial wilt have been cloned to date.Here,we showthat the broadly conserved typeⅢsecreted effector RipY is recognized by the Nicotiana benthamiana immune system,leading to cell death induction,induction of defense-related gene expression,and restriction of bacterial pathogen growth.Using a multiplexed virus-induced gene-silencing-based N.benthamiana nucleotide-binding and leucine-rich repeat receptor(NbNLR)library,we identified a coiled-coil(CC)nucleotide-binding and leucine-rich repeat receptor(CNL)required for recognition of RipY,which we named RESISTANCE TO RALSTONIA SOLANACEARUM RIPY(RRS-Y).Genetic complementation assays in RRS-Y-silenced plants and stable rrs-y knockout mutants demonstrated that RRS-Y is sufficient to activate RipY-induced cell death andRipY-induced immunity to Ralstonia pseudosolanacearum.RRS-Y function is dependent on the phosphate-binding loop motif of the nucleotide-binding domain but independent of the characterized signaling components ENHANCED DISEASE SUSCEPTIBILITY 1,ACTIVATED DISEASE RESISTANCE 1,and N REQUIREMENT GENE 1 and the NLR helpers NB-LRR REQUIRED FOR HR-ASSOCIATED CELL DEATH-2,-3,and-4 in N.benthamiana.We further show that RRS-Y localization at the plasma membrane is mediated by two cysteine residues in the CC domain and is required for RipY recognition.RRSY also broadly recognizes RipY homologs across Ralstonia species.Lastly,we show that the C-terminal region of RipY is indispensable for RRS-Y activation.Together,our findings provide an additional effector/receptor pair system to deepen our understanding of CNL activation in plants.Boyoung Kim Wenjia Yu Haseong Kim Qian Dong Sera Choi Maxim Prokchorchick Alberto PMacho Kee Hoon Sohn Cécile Segonzac 2023Plant Communications2023,4,6:0
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