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| 1 | ‘Cancer associated fibroblasts’ – more than meets the eye显示文摘 | Shalom Madar Ido Goldstein Varda Rotter | 2013 | Trends in Molecular Medicine2013，，： | 2 |
| 2 | Mutant p53-a potential player in shaping the tumor-stroma crosstalk显示文摘A plethora of studies suggest that the non-transformed cellularand non-ceHular comp on ents of the tumor,collectively known as the tumor microenvironment,have a significa nt impact on the tumorigenic process.It was suggested that the microenvironment,which initially restricts tumor development,is recruited by the tumor and main tains a crosstalk that further promotes cancer progression. | Yan Stein Ronit Aloni-Grinstein Varda Rotter | 2019 | Journal of Molecular Cell Biology2019，11，7： | 2 |
| 3 | Mutant p53 Prolongs NF-κB Activation and Promotes Chronic Inflammation and Inflammation-Associated Colorectal Cancer显示文摘 | Tomer Cooks Ioannis S. Pateras Ohad Tarcic Hilla Solomon Aaron J. Schetter Sylvia Wilder Guillermina Lozano Eli Pikarsky Tim Forshew Nitzan Rozenfeld Noam Harpaz Steven Itzkowitz Curtis C. Harris Varda Rotter Vassilis G. Gorgoulis Moshe Oren | 2013 | Cancer Cell2013，，5： | 1 |
| 4 | Modulation of the Vitamin D3 Response by Cancer-Associated Mutant p53显示文摘 | Perry Stambolsky Yuval Tabach Giulia Fontemaggi Lilach Weisz Revital Maor-Aloni Zahava Sigfried Idit Shiff Ira Kogan Moshe Shay Eyal Kalo Giovanni Blandino Itamar Simon Moshe Oren Varda Rotter | 2010 | Cancer Cell2010，，3： | 1 |
| 5 | Corrigendum to‘p53 balances between tissue hierarchy and anarchy’显示文摘In this article (p.559, third paragraph), the reference in ‘The identified ESC gene signature-derived genes correlated with poor patient survival and human tumors harboring p53 hotspot mutations (Lonetto et al., 2018)’ was incorrectly cited. It should be changed to ‘(Koifman et al., 2018)’. The conclusions of the review are not affected and the authors apologize for this error. | Gabriela Koifman Ronit Aloni-Grinstein Varda Rotter | 2019 | Journal of Molecular Cell Biology2019，11，12： | 0 |
| 6 | p53 balances between tissue hierarchy and anarchy显示文摘Normal tissues are organized in a hierarchical model,whereas at the apex of these hierarchies reside stem cells (SCs) capable of self-renewal and of producing differentiated cellular progenies,leading to normal development and homeostasis.Alike,tumors are organized in a hierarchical manner,with cancer SCs residing at the apex,contributing to the development and nourishment of tumors .p53,the well-known 'guardian ofthe genome',possesses various roles in embryonic development as well as in adult SC life and serves as the ‘guardian of tissue hierarchy’.Moreover,p53 serves as a barrier for dedifferentiation and reprogramming by constraining the cells to a somatic state and preventing their conversion to SCs.On the contrary,the mutant forms of p53 that lost theirtumor suppressor activity and gain oncogenic functions serve as 'inducers oftissue anarchy' and promote cancer development.In this review,we discuss these two sides ofthe p53 token that sentence a tissue either to an ordered hierarchy and life or to anarchy and death.A better understanding of these processes may open new horizons for the development of new cancer therapies. | Gabriela Koifman Ronit Aloni-Grinstein Varda Rotter | 2019 | Journal of Molecular Cell Biology2019，11，7： | 0 |
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