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| 1 | Abdominal Visceral and Subcutaneous Adipose Tissue Compartments: Association With Metabolic Risk Factors in the Framingham Heart Study显示文摘 | Caroline S. Fox Joseph M. Massaro Udo Hoffmann Karla M. Pou Pal Maurovich-Horvat Chun-Yu Liu Ramachandran S. Vasan Joanne M. Murabito James B. Meigs L Adrienne Cupples Ralph B. D’Agostino Christopher J. O’Donnell | 2007 | Circulation2007，，1： | 2 |
| 2 | Total serum bilirubin and risk of cardiovascular disease in the Framingham of spring study 显示文摘 | Levy D Adrienne L | 2001 | The American Journal of Cardiology2001，87，10： | 1 |
| 3 | Effect of granulocyte-monocyte colony-stimulating factor therapy on leukocyte function and clearance of serious infection in nonneutropenic patients显示文摘 | Alan J R Peter K L Adrienne D | 2005 | Chest2005，127，： | 1 |
| 4 | Kinetic modeling of phase selection during non-equilibrium solidification of a tungsten-carbon system显示文摘 | Marjos D D Nasr M G Adrienne S L | 2002 | Acta Materialia2002，50，6： | 1 |
| 5 | JH and ^13C NMR Assignments of Some Green Tea Polyphenols 显示文摘 | Adrienne L D Cai Ya Alan P D | 1996 | Magn Reson Chem1996，34，11： | 1 |
| 6 | Total serum bilirubin and risk of cardiovascular disease in the Framingham offspring study显示文摘 | Diousse L Levy D Adrienne L | 2001 | The American Journal of Cardiology2001，87，10： | 1 |
| 7 | IH and 13C-NMR assign- ments of some green tea polyphenols 显示文摘 | Adrienne L D Ya C Alan P D | 1996 | Magn Reson Chem1996，34，： | 1 |
| 8 | Oil red-O stains non-adi-pogenic cell:a precautionary note显示文摘 | ADRIENNE D K MELINDA E F DERI L | 2004 | Cytotechnology2004，46，14： | 1 |
| 9 | Abdominal Visceral and Subcutaneous Adipose Tissue Compartments: Association With Metabolic Risk Factors in the Framingham Heart Study显示文摘 | Caroline S. Fox Joseph M. Massaro Udo Hoffmann Karla M. Pou Pal Maurovich-Horvat Chun-Yu Liu Ramachandran S. Vasan Joanne M. Murabito James B. Meigs L Adrienne Cupples Ralph B. D’Agostino Christopher J. O’Donnell | 2007 | Circulation2007，，1： | 1 |
| 10 | Vie total maximum daily loads as a process, not a singular value : adaptive watershed management 显示文摘 | Paul L Freedman P E A M ASCE Adrienne D Nemura | 2004 | Journal of Environment Engineering2004，，6： | 1 |
| 11 | Association of cholesterol ester transfer protein-Taq I B polymorphism with variations in lipoprotein subclasses and coronary heart disease risk显示文摘 | Ordovas JM Adrienne Cupples L Corella D | 2000 | Arterioscler Thromb Vasc Biol2000，20，： | 1 |
| 12 | Spinal cord injury reprograms muscle fibroadipogenic progenitors to form heterotopic bones within muscles显示文摘The cells of origin of neurogenic heterotopic ossifications(NHOs), which develop frequently in the periarticular muscles following spinal cord injuries(SCIs) and traumatic brain injuries, remain unclear because skeletal muscle harbors two progenitor cell populations: satellite cells(SCs), which are myogenic, and fibroadipogenic progenitors(FAPs), which are mesenchymal. Lineage-tracing experiments using the Cre recombinase/Lox P system were performed in two mouse strains with the fluorescent protein Zs Green specifically expressed in either SCs or FAPs in skeletal muscles under the control of the Pax7 or Prrx1 gene promoter, respectively. These experiments demonstrate that following muscle injury, SCI causes the upregulation of PDGFRα expression on FAPs but not SCs and the failure of SCs to regenerate myofibers in the injured muscle, with reduced apoptosis and continued proliferation of muscle resident FAPs enabling their osteogenic differentiation into NHOs. No cells expressing Zs Green under the Prrx1 promoter were detected in the blood after injury, suggesting that the cells of origin of NHOs are locally derived from the injured muscle. We validated these findings using human NHO biopsies. PDGFRα+mesenchymal cells isolated from the muscle surrounding NHO biopsies could develop ectopic human bones when transplanted into immunocompromised mice, whereas CD56+myogenic cells had a much lower potential. Therefore, NHO is a pathology of the injured muscle in which SCI reprograms FAPs to undergo uncontrolled proliferation and differentiation into osteoblasts. | Hsu-Wen Tseng Dorothée Girard Kylie A.Alexander Susan M.Millard Frédéric Torossian Adrienne Anginot Whitney Fleming Jules Gueguen Marie-Emmanuelle Goriot Denis Clay Beulah Jose Bianca Nowlan Allison R.Pettit Marjorie Salga François Genêt Marie-Caroline Le Bousse-Kerdilès Sébastien Banzet Jean-Pierre Lévesque | 2022 | Bone Research2022，10，2： | 0 |
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