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| 1 | A woman with red eye and hypokalemia:a case of acquired Gitelman' s syndrome显示文摘 | Schwarz C Barisani T Bauer E | | 0，，7： | 1 |
| 2 | Cholelithiasis incirrhosis:analysis of 500 cases显示文摘 | Conte D Barisani D Mandellic C | 1991 | Am J Gast roenterology1991，86，11： | 1 |
| 3 | Cholelithiasisin cirrhosis: analysis of 500 cases显示文摘 | CONTE D BARISANI D MANDELLI C | 1991 | Am J Gastroenterol1991，86，11： | 1 |
| 4 | Celiac Disease: From Pathogenesis to Novel Therapies显示文摘 | Detlef Schuppan Yvonne Junker Donatella Barisani | 2009 | Gastroenterology2009，，6： | 1 |
| 5 | Solitary erythematous, ten tier plaque of the heel in a young infant显示文摘 | Vaccari S Ismaili A Barisani A | 2013 | Dermatology Online Journal2013，19，9： | 1 |
| 6 | Evolutionary and Functional Analysis of Celiac Risk Loci Reveals SH2B3 as a Protective Factor against Bacterial Infection显示文摘 | Alexandra Zhernakova Clara C. Elbers Bart Ferwerda Jihane Romanos Gosia Trynka Patrick C. Dubois Carolien G.F. de Kovel Lude Franke Marije Oosting Donatella Barisani Maria Teresa Bardella Leo A.B. Joosten Paivi Saavalainen David A. van Heel Carlo Catassi | 2010 | The American Journal of Human Genetics2010，，6： | 1 |
| 7 | Food additives can act as triggering factors in celiac disease:Current knowledge based on a critical review of the literature显示文摘Celiac disease(CeD) is an autoimmune disorder, mainly affecting the small intestine, triggered by the ingestion of gluten with the diet in subjects with a specific genetic status. The passage of gluten peptides through the intestinal barrier, the uptake by antigen presenting cells and their presentation to T cells represent essential steps in the pathogenesis of the disease. CeD prevalence varies in different populations, but a tendency to increase has been observed in various studies in recent years. A higher amount of gluten in modern grains could explain this increased frequency, but also food processing could play a role in this phenomenon. In particular, the common use of preservatives such as nanoparticles could intervene in the pathogenesis of CeD, due to their possible effect on the integrity of the intestinal barrier, immune response or microbiota. In fact, these alterations have been reported after exposure to metal nanoparticles,which are commonly used as preservatives or to improve food texture,consistency and color. This review will focus on the interactions between several food additives and the intestine, taking into account data obtained in vitro and in vivo, and analyzing their effect in respect to the development of CeD in genetically predisposed individuals. | Clara Mancuso Donatella Barisani | 2019 | World Journal of Clinical Cases2019，7，8： | 1 |
| 8 | Evolutionary and Functional Analysis of Celiac Risk Loci Reveals SH2B3 as a Protective Factor against Bacterial Infection显示文摘 | Alexandra Zhernakova Clara C. Elbers Bart Ferwerda Jihane Romanos Gosia Trynka Patrick C. Dubois Carolien G.F. de Kovel Lude Franke Marije Oosting Donatella Barisani Maria Teresa Bardella Leo A.B. Joosten Paivi Saavalainen David A. van Heel Carlo Catassi | 2010 | The American Journal of Human Genetics2010，，6： | 1 |
| 9 | Six new coeliac disease loci replicatad in an Italian population confirm association with coeliac disease 显示文摘 | Romanos J Barisani D Trynka G | 2009 | J Med Genet2009，46，1： | 1 |
| 10 | A new human cell line,PDSS-26,from poorly diff-erentiated synovial sarcoma,with unique chromosomal anomalies显示文摘 | Berardi A C Parafioriti A Barisani D | 2003 | Cancer Genet Cytogenet2003，146，2： | 1 |
| 11 | Cholelithiasis in cirrhosis:analysis of 500 cases显示文摘 | Conte D Barisani D Mandellic C | 1991 | Am J Gastroenterology1991，86，11： | 1 |
| 12 | Hepcidin and iron-related gene expression in subjects with Dysmetabolic Hep-atic Iron Overload显示文摘 | Barisani D Pelucchi S Mariani R | | 0，，1： | 1 |
| 13 | Cholelithiasls in cirrhosis:analysis of 500 cases显示文摘 | CONTE D BARISANI D MANDELLI C | 2003 | Am J Gastroenterol2003，86，11： | 1 |
| 14 | Celiac disease : frompathogenesis to novel therapies 显示文摘 | Schuppan D Junker Y Barisani D | 2009 | Gastroenterology2009，137，6： | 1 |
| 15 | Evoluzione della tecnica di Janowitz per la risoluzione della dislocazione abomasale sinistra secondo Barisani显示文摘 | Barisani C | 2004 | Summa2004，，5： | 1 |
| 16 | Analysis of HLA and Non-HLA Alleles Can Identify Individuals at High Risk for Celiac Disease显示文摘 | Jihane Romanos Cleo C. van Diemen Ilja M. Nolte Gosia Trynka Alexandra Zhernakova Jingyuan Fu Maria Teresa Bardella Donatella Barisani Ross McManus David A. van Heel Cisca Wijmenga | 2009 | Gastroenterology2009，，3： | 1 |
| 17 | Cost-effective HLA typing with tagging SNPs predicts celiac disease risk haplotypes in the Finnish, Hungarian, and Italian populations显示文摘 | Lotta Koskinen Jihane Romanos Katri Kaukinen Kirsi Mustalahti Ilma Korponay-Szabo Donatella Barisani Maria Teresa Bardella Fabiana Ziberna Serena Vatta Gy?rgy Széles Zsuzsa Pocsai Kati Karell Katri Haimila Róza ádány Tarcisio Not Alessandro Ventura Markku | 2009 | Immunogenetics2009，，4： | 1 |
| 18 | Cholelithiasis in cirrhosis analysis of 500cases显示文摘 | Barisani D Mandelli C | 1991 | Am J Gastroenteral1991，86，11： | 1 |
| 19 | Celiac disease:From genetics to epigenetics显示文摘Celiac disease(CeD)is a multifactorial autoimmune disorder spread worldwide.The exposure to gluten,a protein found in cereals like wheat,barley and rye,is the main environmental factor involved in its pathogenesis.Even if the genetic predisposition represented by HLA-DQ2 or HLA-DQ8 haplotypes is widely recognised as mandatory for CeD development,it is not enough to explain the total predisposition for the disease.Furthermore,the onset of CeD comprehend a wide spectrum of symptoms,that often leads to a delay in CeD diagnosis.To overcome this deficiency and help detecting people with increased risk for CeD,also clarifying CeD traits linked to disease familiarity,different studies have tried to make light on other predisposing elements.These were in many cases genetic variants shared with other autoimmune diseases.Since inherited traits can be regulated by epigenetic modifications,also induced by environmental factors,the most recent studies focused on the potential involvement of epigenetics in CeD.Epigenetic factors can in fact modulate gene expression with many mechanisms,generating more or less stable changes in gene expression without affecting the DNA sequence.Here we analyze the different epigenetic modifications in CeD,in particular DNA methylation,histone modifications,non-coding RNAs and RNA methylation.Special attention is dedicated to the additional predispositions to CeD,the involvement of epigenetics in developing CeD complications,the pathogenic pathways modulated by epigenetic factors such as microRNAs and the potential use of epigenetic profiling as biomarker to discriminate different classes of patients. | Elisa Gnodi Raffaella Meneveri Donatella Barisani | 2022 | World Journal of Gastroenterology2022，28，4： | 0 |
| 20 | Impact of COVID-19 on inflammatory bowel disease practice and perspectives for the future显示文摘Coronavirus disease 2019(COVID-19)is an infectious disease caused by the novel coronavirus severe acute respiratory syndrome coronavirus 2(SARS-CoV-2);since its first description in December 2019,it has rapidly spread to a global pandemic.Specific concerns have been raised concerning patients with inflammatory bowel diseases(IBD),which are chronic autoimmune inflammatory disorders of the gut that frequently require immunosuppressive and biological therapies to control their activity.Accumulating evidence has so far demonstrated that patients with IBD are not at increased risk of contracting severe acute respiratory syndrome coronavirus 2 infection.As for the general population,the identified risk factors for severe COVID-19 course among IBD patients have been established to be advanced age and the presence of comorbidities.Treatment with high-dose corticosteroids has also been associated with an increased risk of death in IBD patients with COVID-19.Information on COVID-19 is constantly evolving,with data growing at a rapid pace.This will guarantee better knowledge and stronger evidence to help physicians in the choice of the best therapeutic approach for each patient,concurrently controlling for the risk of IBD disease under treatment and the risk of COVID-19 adverse outcomes and balancing the two.Moreover,the impact of the enormous number of severe respiratory patients on healthcare systems and facilities has led to an unprecedented redeployment of healthcare resources,significantly impacting the care of patients with chronic diseases.In this newly changed environment,the primary aim is to avoid harm whilst still providing adequate management.Telemedicine has been applied and is strongly encouraged for patients without the necessity of infusion therapy and whose conditions are stable.The severe acute respiratory syndrome coronavirus 2 pandemic has already revolutionized the management of patients with chronic immune-mediated diseases such as IBD.Direct and indirect effects of the COVID-19 pandemic will be present for some time.This is the reason why continuous research,rapid solutions and constantly updated guidelines are of utmost importance.The aim of the present review is,therefore,to point out what has been learned so far as well as to pinpoint the unanswered questions and perspectives for the future. | Chiara Viganò Giacomo Mulinacci Andrea Palermo Donatella Barisani Lorena Pirola Maria Fichera Pietro Invernizzi Sara Massironi | 2021 | World Journal of Gastroenterology2021，27，33： | 0 |
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