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8篇 您的检索式:作者名="Kaiser FJ"
    题名 作者 年代 出处 被引量
1DMP1 mutations in autosomal recessive hypophosphatemia implicate a bone matrix protein in the regulation of phosphate homeostasis显示文摘Lorenz-Depiereux B Bastepe M Benet-Pages A Amyere M Wagenstaller J Muller-Barth U Badenhoop K Kaiser SM Rittmaster RS Shlossberg AH Olivares JL Loris C Ramos FJ Glorieux F Vikkula M Juppner H Strom TM 0,,11:1
2Trpsl, a regu- lator of chondrocyte proliferation and differentiation, inter- acts with the activator form of Gli3 显示文摘Wuelling M Kaiser FJ Buelens LA 2009Dev Biol2009,328,1:1
3The dystonia gene DYT1 is repressed by the transcription factor THAP1 (DYT6) 显示文摘Kaiser FJ Osmanoric A Rakovic A 2010Ann Neurol2010,68,4:1
4Mutations in the cyclin family member FAM58A cause an X-linked dominant disorder characterized by syndactyly, telecanthus and anogenital and renal malformations显示文摘Unger S Bohm D Kaiser FJ 2008Nat Genet2008,40,3:1
5The dysto- nia gene DYT1 IS repressed by the transcription fac- tor THAP1 (DYT6) 显示文摘Kaiser FJ Osmanoric A Rakovic A 2010Ann Neurol2010,68,4:1
6Amphiphilic secondary structure:design of peptide hormones显示文摘 Kezdy FJ 1984Science1984,223,:1
7Whole genome survey of copy number variation in the spontaneously hypertensive tah telationship to quantitative trait loci, gene expression, and blood pressure 显示文摘Charchar FJ Kaiser M Bingham AJ 2010Hypertension2010,55,6:1
8Trps1, a regulator of chondrocyte proliferation and differentiation, interacts with the activator form of Gli3 显示文摘Wuelllng M Kaiser FJ Buelens LA 2009Dev Biol2009,328,1:1
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