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| 1 | The Role of 3-Canal Biomechanics in Angular Motion Transduction by the Human Vestibular Labyrinth显示文摘 | Marytheresa A. Ifediba Suhrud M. Rajguru Timothy E. Hullar Richard D. Rabbitt | 2007 | Annals of Biomedical Engineering2007，，7： | 2 |
| 2 | Three-Dimensional Biomechanical Model of Benign Paroxysmal Positional Vertigo显示文摘 | Suhrud MR Marytheresa AI Richard DR | 2004 | Annals of Biomedical Engineering2004，32，6： | 1 |
| 3 | The Role of 3-Canal Biomechanics in Angular Motion Transduction by the Human Vestibular Labyrinth显示文摘 | Marytheresa A. Ifediba Suhrud M. Rajguru Timothy E. Hullar Richard D. Rabbitt | 2007 | Annals of Biomedical Engineering2007，，7： | 1 |
| 4 | Three-Dimensional Biomechanical Model of Benign Paroxysmal Positional Vertigo显示文摘 | Suhrud M. Rajguru Marytheresa A. Ifediba Richard D. Rabbitt | 2004 | Annals of Biomedical Engineering2004，，6： | 1 |
| 5 | Acute damage threshold for infrared neural stimulation of the cochlea: functional and histological evaluation 显示文摘 | Vinay Goyal Suhrud Rajguru Agnella I Matic | 2012 | The Anatomical Record2012，295，11： | 1 |
| 6 | Optical cochlear implants : evaluation of surgical approach and laser parameters in cats 显示文摘 | Suhrud MR Agnella IM Alan MR | 2010 | Hearing Research2010，54，6： | 1 |
| 7 | Infrared neural stimulation:Beam path in the guinea pig cochlea显示文摘 | Laura E M Suhrud M R Agnella D I | 2011 | Hearing Research2011，6，6： | 1 |
| 8 | The Role of 3-Canal Biomechanics in Angular Motion Transduction by the Human Vestibular Labyrinth显示文摘 | Marytheresa A. Ifediba Suhrud M. Rajguru Timothy E. Hullar Richard D. Rabbitt | 2007 | Annals of Biomedical Engineering2007，，7： | 1 |
| 9 | Three-Dimensional Biomechanical Model of Benign Paroxysmal Positional Vertigo显示文摘 | Suhrud M. Rajguru Marytheresa A. Ifediba Richard D. Rabbitt | 2004 | Annals of Biomedical Engineering2004，，6： | 1 |
| 10 | Oxidative stress and COVID-19-associated neuronal dysfunction: mechanisms and therapeutic implications显示文摘Severe acute respiratory syndrome(SARS)-CoV-2 virus causes novel coronavirus disease 2019(COVID-19),and there is a possible role for oxidative stress in the pathophysiology of neurological diseases associated with COVID-19.Excessive oxidative stress could be responsible for the thrombosis and other neuronal dysfunctions observed in COVID-19.This review discusses the role of oxidative stress associated with SARS-CoV-2 and the mechanisms involved.Furthermore,the various therapeutics implicated in treating COVID-19 and the oxidative stress that contributes to the etiology and pathogenesis of COVID-19-induced neuronal dysfunction are discussed.Further mechanistic and clinical research to combat COVID-19 is warranted to understand the exact mechanisms,and its true clinical effects need to be investigated to minimize neurological complications from COVID-19. | Dylan R.Bowen Suhrud Pathak Rishi M.Nadar Rachel D.Parise Sindhu Ramesh Manoj Govindarajulu Austin Moore Jun Ren Timothy Moore Muralikrishnan Dhanasekaran | 2023 | Acta Biochimica et Biophysica Sinica2023，55，8： | 0 |
| 11 | A scientific perspective of how and why Omicron is less severe than SARS-CoV-2显示文摘Omicron is currently the dominant variant of severe acute respiratory syndrome coronavirus 2(SARS-CoV-2),the coronavirus responsible for the coronavirus disease 2019(COVID-19)pandemic.Omicron is associated with mild symptoms,although it can cause harmful effects in high-risk patient populations.Omicron and COVID-19 affect multiple organ systems,including the respiratory system,gastrointestinal tract,cardiovascular system,central nervous system,ophthalmic system,genitourinary tract,and musculoskeletal system.COVID-19 infects additional organ systems,including the hematological system,hepatobiliary system,renal system,and dermatologic system.The viral-induced complications were compared to discuss the effects of Omicron versus the authentic SARS-CoV-2 virus,revealing less detrimental outcomes for Omicron.Moreover,COVID-19 is more likely to infect older adults,males,and obesity with mild to severe symptoms.Omicron causes mild symptoms in younger populations and overweight females.Data were acquired using PubMed,Centers for Disease Prevention and Control,and the World Health Organization.COVID-19 and Omicron mechanisms causing organ system-related complications are likely because of the natural immune response to the active infection,the uncontrollable release of cytokines causing cytokine release syndrome,and direct viral damage through angiotensin-converting enzyme 2/transmembrane serine protease 2 receptor binding and entrance to the host cell for infection. | Rachel Parise Sindhu Ramesh Jun Ren Manoj Y.Govindarajulu Rishi M.Nadar Suhrud Pathak Timothy Moore Muralikrishnana Dhanasekaran | 2023 | Emergency and Critical Care Medicine2023，3，3： | 0 |
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