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| 1 | Accruals Quality and In- ternal Control over Financial Reporting 显示文摘 | JEFFREY D WEILI G SARAH M | 2007 | Journal of Ac- counting Review2007，，1： | 1 |
| 2 | Preparation and characterization o{ a novel microporous PE membrane supporting composite gel polymer electrolyte显示文摘 | Yang Mujie Li Weili Wang G G | 2005 | Solid State Ionies:2005，176，3738： | 1 |
| 3 | The Disclosure of Material Weaknesses in Inter- nal Control After The Sarbanes-Oxley Act显示文摘 | Weili G Sarah M V | 2005 | Accounting Horizons2005，19，3： | 1 |
| 4 | Accruals quality and internal control over financial reporting 显示文摘 | Doyle J Weili G Sarah M | 2007 | Journal of Accounting Re- view2007，72，7： | 1 |
| 5 | A novel absorb/transmit FSS for secure indoor wireless networks with reduced mul- tipath fading 显示文摘 | Kiani G I Weily A R Esselle K P | 2006 | Microwave and Wireless Components l~q- ters IEEE2006，16，6： | 1 |
| 6 | Identification of pancreatic cancer stem cells显示文摘 | Chen weiLi David G Heidt | 2007 | Cancer Res2007，67，3： | 1 |
| 7 | Preparation and characterization of a novel microporous PE membrane supporting composite gel polymer electrolyte 显示文摘 | Yang Mujie Li Weili Wang G G | 2005 | Solid State Ionics2005，176，3738： | 1 |
| 8 | Determinants of Weak- nesses in Internal Control over Financial Reporting 显示文摘 | Jeffrey D Weili G Sarah M | 2007 | Journal of Accounting and Economics2007，44，12： | 1 |
| 9 | Oblique incidence performance of' a novel fre-quency selective surface absorber 显示文摘 | G Kiani K Ford K Esselle A Weily C Panagamu- wa | 2007 | IEEE Trans Antennas Propag2007，55，10： | 1 |
| 10 | Accruals quality and internal control over financial re- porting 显示文摘 | Doyle J Weili G Sarah M | 2007 | Journal of Accounting Review2007，72，7： | 1 |
| 11 | Research of UL54-specific siRNA on herpes simplex virus type Ⅱ replication显示文摘 | Qing G Weili W Fanqin Z | 2011 | Int J Dermatol2011，50，3： | 1 |
| 12 | Role of mitogen- and stress-activated kinases in inflammatory arthritis显示文摘Lysophosphatidic acid(LPA) is a pleiotropic lipid mediator that promotes motility, survival, and the synthesis of chemokines/cytokines in human fibroblast-like synoviocytes(FLS) from patients with rheumatoid arthritis. LPA activates several proteins within the mitogen activated protein(MAP) kinase signaling network, including extracellular signal-regulated kinases(ERK) 1/2 and p38 MAP kinase(MAPK). Upon docking to mitogen- and stress-activated kinases(MSKs), ERK1/2 and p38 MAPK phosphorylate serine and threonine residues within its C-terminal domain and cause autophosphorylation of MSKs. Activated MSKs can then directly phosphorylate c AMP response element-binding protein(CREB) at Ser133 in FLS. Phosphorylation of CREB by MSKs is essential for the production of pro-inflammatory and anti-inflammatory cytokines. However, other downstream effectors of MSK1/2 such as nuclear factor-kappa B, histone H3, and high mobility group nucleosome binding domain 1 may also regulate gene expression in immune cells involved in disease pathogenesis. MSKs are master regulators of cell function that integrate signals induced by growth factors, pro-inflammatory cytokines, and cellular stresses, as well as those induced by LPA. | Sylvain G Bourgoin Weili Hui | 2015 | World Journal of Pharmacology2015，4，4： | 0 |
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