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| 1 | 未破裂颅内动脉瘤患者管理指南美国心脏协会/美国卒中协会针对医疗专业人员的指南显示文摘目的这份更新版指南旨在为未破裂颅内动脉瘤患者的管理提供全面的循证建议。方法写作组采用系统文献回顾(1977年1月至2014年6月),参考同期已发表的循证指南、个人文件和专家意见来总结现有的证据,指出现有知识的差距;如果合适,则根据美国心脏协会( American Heart Association, AHA)标准做出推荐。本指南经过了包括卒中委员会领导层以及AHA科学声明监督委员会在内的广泛同行评议,然后由AHA科学咨询和协调委员会审批通过。结果本指南对未破裂颅内动脉瘤患者的医疗诊治提出了循证建议,涉及临床表现、自然史、流行病学、危险因素、筛查、诊断、影像学以及手术和血管内治疗的转归。 | B. Gregory Thompson Robert D. Brown Sepideh Amin-Hanjani Joseph P. Broderick Kevin M. Cockroft E. Sander Connolly Gary R. Duckwiler Catherine C. Harris Virginia J. Howard S. Claiborne (Clay) Johnston Philip M. Meyers Andrew Molyneux Christopher S. Ogilvy Andrew J. Ringer James Torner 肖国栋 徐加平 石际俊 尤寿江 张霞 刘慧慧 曹勇军 | 2015 | 国际脑血管病杂志2015，23，8： | 127 |
| 2 | 美国临床肿瘤学会IV期非小细胞肺癌化疗的临床实践指南更新显示文摘本文旨在为IV期非小细胞肺癌患者的治疗提供更新版推荐。本文资料检索源自2002年以来公布的相关随机试验文献。此指南范围限于化疗与生物治疗。更新委员会对这些文献进行了总结并提供了推荐更新。162篇文献符合标准被纳入参考。本推荐基于可改善总生存期的治疗方法。仅改善无进展生存期的治疗方法推动了对毒性及生存质量的监测。对于体力状态评分为0分或1分患者的一线治疗,可推荐以铂类为基础的细胞毒性药物的两药联用。对铂类治疗有禁忌的患者,可采用非铂类细胞毒性两药联合。对于体力状态评分为2分的患者,单一细胞毒性药物即可。对于疾病进展或经过4个周期的治疗仍对治疗无反应的患者,应停止一线细胞毒性化疗。即使在6个周期后患者对治疗仍有反应,亦应停止两药细胞毒性化疗。对于伴有明确的表皮生长因子受体(epidermal growth factor receptor,EGFR)突变的患者,可推荐一线采用吉非替尼治疗;对于EGFR突变为阴性或不明确的患者,细胞毒性化疗更佳。除具有特定临床特征的患者外,可推荐贝伐单抗与卡铂-紫杉醇联用。对于通过免疫组化证实EGFR阳性的肿瘤患者,可推荐西妥昔单抗与顺铂-长春瑞滨联用。多西紫杉醇、厄洛替尼、吉非替尼或培美曲塞被推荐作为二线治疗。对于未曾接受过厄洛替尼或吉非替尼治疗的患者,可推荐厄洛替尼作为三线治疗。现有数据不足以推荐常规三线采用细胞毒性药物。已有的证据也不足以推荐常规应用分子标记物选择化疗。 | Christopher G. AZZOLI Sherman Baker JR Sarah TEMIN William PAO Timothy ALIFF Julie BRAHMER David H. JOHNSON Janessa L. LASKIN Gregory MASTERS Daniel MILTON Luke NORDQUIST David G. PFISTER Steven PIANTADOSI Joan H. SCHILLER Reily SMITH Thomas J. SMITH John R. STRAWN David TRENT Giuseppe GIACCONE 丁燕(翻译) 南娟(翻译) 刘谦(翻译) 周清华(校对) 陈军(校对) | 2010 | 中国肺癌杂志2010，13，3： | 43 |
| 3 | Guidelines for the Management of Aneurysmal Subarachnoid Hemorrhage: A Guideline for Healthcare Professionals From the American Heart Association/American Stroke Association显示文摘 | E. Sander Connolly Alejandro A. Rabinstein J. Ricardo Carhuapoma Colin P. Derdeyn Jacques Dion Randall T. Higashida Brian L. Hoh Catherine J. Kirkness Andrew M. Naidech Christopher S. Ogilvy Aman B. Patel B. Gregory Thompson Paul Vespa | 2012 | Stroke2012，，6： | 3 |
| 4 | Local Therapy with Continued EGFR Tyrosine Kinase Inhibitor Therapy as a Treatment Strategy in EGFR-Mutant Advanced Lung Cancers That Have Developed Acquired Resistance to EGFR Tyrosine Kinase Inhibitors显示文摘 | Helena A. Yu Camelia S. Sima James Huang Stephen B. Solomon Andreas Rimner Paul Paik M. Catherine Pietanza Christopher G. Azzoli Naiyer A. Rizvi Lee M. Krug Vincent A. Miller Mark G. Kris Gregory J. Riely | 2013 | Journal of Thoracic Oncology2013，，3： | 2 |
| 5 | Serotonin type 3 receptor subunit gene polymorphisms associated with psychosomatic symptoms in irritable bowel syndrome:A multicenter retrospective study显示文摘BACKGROUND Single-nucleotide polymorphisms(SNPs)of the serotonin type 3 receptor subunit(HTR3)genes have been associated with psychosomatic symptoms,but it is not clear whether these associations exist in irritable bowel syndrome(IBS).AIM To assess the association of HTR3 polymorphisms with depressive,anxiety,and somatization symptoms in individuals with IBS.METHODS In this retrospective study,623 participants with IBS were recruited from five specialty centers in Germany,Sweden,the United States,the United Kingdom,and Ireland.Depressive,anxiety,and somatization symptoms and sociodemographic characteristics were collected.Four functional SNPs—HTR3A c.-42C>T,HTR3B c.386A>C,HTR3C c.489C>A,and HTR3E c.*76G>A—were genotyped and analyzed using the dominant and recessive models.We also performed separate analyses for sex and IBS subtypes.SNP scores were calculated as the number of minor alleles of the SNPs above.The impact of HTR3C c.489C>A was tested by radioligand-binding and calcium influx assays.RESULTS Depressive and anxiety symptoms significantly worsened with increasing numbers of minor HTR3C c.489C>A alleles in the dominant model(F_(depressive)=7.475,P_(depressive)=0.006;F_(anxiety)=6.535,P_(anxiety)=0.011).A higher SNP score(range 0-6)was linked to a worsened depressive symptoms score(F=7.710,P-linear trend=0.006)in IBS.The potential relevance of the HTR3C SNP was corroborated,showing changes in the expression level of 5-HT3AC variant receptors.CONCLUSION We have provided the first evidence that HTR3C c.489C>A is involved in depressive and anxiety symptoms in individuals with IBS.The SNP score indicated that an increasing number of minor alleles is linked to the worsening of depressive symptoms in IBS. | Sabrina Berens Yuanjun Dong Nikola Fritz Jutta Walstab Mauro D'Amato Tenghao Zheng Verena Wahl Felix Boekstegers Justo Lorenzo Bermejo Cristina Martinez Stefanie Schmitteckert Egbert Clevers Felicitas Engel Annika Gauss Wolfgang Herzog Robin Spiller Miriam Goebel-Stengel Hubert Mönnikes Viola Andresen Frieling Thomas Jutta Keller Christian Pehl Christoph Stein-Thöringer Gerard Clarke Timothy G Dinan Eamonn M Quigley Gregory Sayuk Magnus Simrén Jonas Tesarz Gudrun Rappold Lukas van Oudenhove Rainer Schaefert Beate Niesler | 2022 | World Journal of Gastroenterology2022，28，21： | 2 |
| 6 | Per-oral endoscopic myotomy white paper summary显示文摘 | Stavros N. Stavropoulos David J. Desilets Karl-Hermann Fuchs Christopher J. Gostout Gregory Haber Haruhiro Inoue Michael L. Kochman Rani Modayil Thomas Savides Daniel J. Scott Lee L. Swanstrom Melina C. Vassiliou | 2014 | Surgical Endoscopy2014，，7： | 2 |
| 7 | 2011 ACCF/AHA Focused Update of the Guideline for the Management of Patients With Peripheral Artery Disease (Updating the 2005 Guideline)显示文摘 | Thom W. Rooke Alan T. Hirsch Sanjay Misra Anton N. Sidawy Joshua A. Beckman Laura K. Findeiss Jafar Golzarian Heather L. Gornik Jonathan L. Halperin Michael R. Jaff Gregory L. Moneta Jeffrey W. Olin James C. Stanley Christopher J. White John V. White R. E | 2011 | Journal of the American College of Cardiology2011，，： | 2 |
| 8 | The effects of temperature and nutrients on the growth and dynamics of toxic and non-toxic strains of Microcystis during cyanobacteria blooms显示文摘 | Timothy W. Davis Dianna L. Berry Gregory L. Boyer Christopher J. Gobler | 2009 | Harmful Algae2009，，5： | 2 |
| 9 | Transfusion-related acute lung injury in ICU patients admitted with gastrointestinal bleeding显示文摘 | Alexander B. Benson Gregory L. Austin Mary Berg Kim K. McFann Sila Thomas Gina Ramirez Hugo Rosen Christopher C. Silliman Marc Moss | 2010 | Intensive Care Medicine2010，，10： | 2 |
| 10 | Predicting Survival in Pulmonary Arterial Hypertension: Insights From the Registry to Evaluate Early and Long-Term Pulmonary Arterial Hypertension Disease Management (REVEAL)显示文摘 | Raymond L. Benza Dave P. Miller Mardi Gomberg-Maitland Robert P. Frantz Aimee J. Foreman Christopher S. Coffey Adaani Frost Robyn J. Barst David B. Badesch C. Gregory Elliott Theodore G. Liou Michael D. McGoon | 2010 | Circulation2010，，2： | 2 |
| 11 | Assessing risks from mining-induced ground movements near gas wells显示文摘The proliferation of unconventional gas well development in the Northern Appalachian coalfields has raised a number of mine safety concerns.Unconventional wells,which extract gas from deep shale formations,are characterized by gas volumes and pressures that are significantly higher than those observed at many conventional wells.The gas is composed largely of methane as well as other hydrocarbons.Hundreds of planned and actively producing wells penetrate protective coal pillars or barriers within active mine boundaries,including chain pillars located between longwall panels.Gas released from a well damaged by mining-induced ground movements could pose a risk to miners by flowing into the mine atmosphere.The mining-induced ground movements that may cause well damage include conventional subsidence,non-conventional subsidence(e.g.bedding plane slip),pillar failure,and floor instability.This paper describes the known risk factors for each of the four failure mechanisms.It includes a framework that can guide the risk assessment process when mining takes place near gas or oil wells. | Christopher Mark Gregory M.Rumbaugh | 2020 | International Journal of Mining Science and Technology2020，30，1： | 2 |
| 12 | Dynamic glial response and crosstalk in demyelination-remyelination and neurodegeneration processes显示文摘Multiple sclerosis is an autoimmune disease in which the immune system attacks the myelin sheath in the central nervous system.It is characterized by blood-brain barrier dysfunction throughout the course of multiple sclerosis, followed by the entry of immune cells and activation of local microglia and astrocytes.Glial cells(microglia, astrocytes, and oligodendrocyte lineage cells) are known as the important mediators of neuroinflammation, all of which play major roles in the pathogenesis of multiple sclerosis.Network communications between glial cells affect the activities of oligodendrocyte lineage cells and influence the demyelination-remyelination process.A finely balanced glial response may create a favorable lesion environment for efficient remyelination and neuroregeneration.This review focuses on glial response and neurodegeneration based on the findings from multiple sclerosis and major rodent demyelination models.In particular, glial interaction and molecular crosstalk are discussed to provide insights into the potential cell-and molecule-specific therapeutic targets to improve remyelination and neuroregeneration. | Tianci Chu Lisa B.E.Shields Wenxin Zeng Yi Ping Zhang Yuanyi Wang Gregory N.Barnes Christopher B.Shields Jun Cai | 2021 | Neural Regeneration Research2021，16，7： | 2 |
| 13 | ACCF/ACR/AHA/NASCI/SCMR 2010 Expert Consensus Document on Cardiovascular Magnetic Resonance显示文摘 | W. Gregory Hundley David A. Bluemke J. Paul Finn Scott D. Flamm Mark A. Fogel Matthias G. Friedrich Vincent B. Ho Michael Jerosch-Herold Christopher M. Kramer Warren J. Manning Manesh Patel Gerald M. Pohost Arthur E. Stillman Richard D. White Pamela K. Wo | 2010 | Journal of the American College of Cardiology2010，，23： | 1 |
| 14 | Updated Clinical Classification of Pulmonary Hypertension显示文摘 | Gérald Simonneau Ivan M. Robbins Maurice Beghetti Richard N. Channick Marion Delcroix Christopher P. Denton C. Gregory Elliott Sean P. Gaine Mark T. Gladwin Zhi-Cheng Jing Michael J. Krowka David Langleben Norifumi Nakanishi Rogério Souza | 2009 | Journal of the American College of Cardiology2009，，1： | 1 |
| 15 | Independent Validation of the Secondary Intracerebral Hemorrhage Score With Catheter Angiography and Findings of Emergent Hematoma Evacuation显示文摘 | Josser E. Delgado Almandoz Bharathi D. Jagadeesan Christopher J. Moran DeWitte T. Cross Gregory J. Zipfel Jin-Moo Lee Javier M. Romero Colin P. Derdeyn | 2012 | Neurosurgery2012，，1： | 1 |
| 16 | Partial Nephrectomy Is Associated with Improved Overall Survival Compared to Radical Nephrectomy in Patients with Unanticipated Benign Renal Tumours 显示文摘 | Christopher J. Weight Gregory Lieser Benjamin T. Larson Tianming Gao Brian R. Lane Steven C. Campbell Inderbir S. Gill Andrew C. Novick Amr F. Fergany | 2010 | European Urology2010，，2： | 1 |
| 17 | Inflammation and cancer revisited: An hypothesis on the oncogenic potential of the apoptotic tumor cell显示文摘 | Christopher D. Gregory | 2013 | Autoimmunity2013，，5： | 1 |
| 18 | A piRNA Pathway Primed by Individual Transposons Is Linked to De Novo DNA Methylation in Mice显示文摘 | Alexei A. Aravin Ravi Sachidanandam Deborah Bourc’his Christopher Schaefer Dubravka Pezic Katalin Fejes Toth Timothy Bestor Gregory J. Hannon | 2008 | Molecular Cell2008，，6： | 1 |
| 19 | The relationship between place attachment and landscape values: Toward mapping place attachment 显示文摘 | Gregory Brown Christopher Raymond | 2007 | Applied Geography2007，，27： | 1 |
| 20 | Routine use of the Ion Torrent AmpliSeq? Cancer Hotspot Panel for identification of clinically actionable somatic mutations显示文摘 | Gregory J. Tsongalis Jason D. Peterson Francine B. de Abreu Christopher D. Tunkey Torrey L. Gallagher Linda D. Strausbaugh Wendy A. Wells Christopher I. Amos | 2014 | Clinical Chemistry and Laboratory Medicine2014，，5： | 1 |
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