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15篇 您的检索式:作者名="Emma I"
    题名 作者 年代 出处 被引量
1Association of human cy- clinE with a periodic G1 -S phase protein kinase 显示文摘Vjekoslav D Emma L Steven I R 1992Sci- ence1992,257,25:1
2Spatial analysis of human granulocytic ehrlichiosis near Lyme, connectiuct显示文摘Emma K C James I M Robert H 2002Emerg Infect Dis2002,8,9:1
3Antioxidative action of the ethanolic extract and some hydroxycoumarins of Fraxinus ornus bark显示文摘EMMA M M NEDJALKA V I Y IVANKA N K 1994Food Chemistry1994,51,2:1
4Baeulovirus proteins IE-1, LEF-3, and P143 interact with DNA/n vivo: a formaldehyde cross-linking study显示文摘Emma I Kaniela S Roll K 2004Virology2004,329,2:1
5Dupilumab Treatment in Adults with Moderate-to-Severe Atopic Dermatitis显示文摘Lisa A. Beck Diamant Tha?i Jennifer D. Hamilton Neil M. Graham Thomas Bieber Ross Rocklin Jeffrey E. Ming Haobo Ren Richard Kao Eric Simpson Marius Ardeleanu Steven P. Weinstein Gianluca Pirozzi Emma Guttman-Yassky Mayte Suárez-Fari?as Melissa D. Hager Ne 2014The New England Journal of Medicine2014,,:1
6Spatial Anal ysis of Human Granulocytic Ehrlichiosis near Lyme,Connecticut 显示文摘Emma K Chaput James I Meek Robert Heimer 2002Emerging Inf Dis2002,8,:1
7Role for Bovine viral diarrhea virus Erns glycoprotein in the control of activation of beta interferon by double-stranded RNA显示文摘Munir I Emma P Stephen G 2004Journal of Virology2004,78,1:1
8Telemonitoring and self-management in the control of hypertension (TASMINH2): a randomised controlled trial显示文摘Richard J McManus Jonathan Mant Emma P Bray Roger Holder Miren I Jones Sheila Greenfield Billingsley Kaambwa Miriam Banting Stirling Bryan Paul Little Bryan Williams FD Richard Hobbs 2010The Lancet2010,,9736:1
9Spatial Analysis of Human Granulocytic Ehrlichiosis near Lyme, Connecticut显示文摘Emma K Chaput James I Meek Robert Heimer 2002Emerging Inf Dis2002,8,:1
10Endothelial cell O-glycan deficiency causes blood/lymphatic misconnections and consequent fatty liver disease in mice显示文摘Fu Jianxin Gerhardt Holger McDaniel J Michael Xia Baoyun Liu Xiaowei Ivanciu Lacramioara Ny Annelii Hermans Karlien Silasi-Mansat Robert McGee Samuel Nye Emma Ju Tongzhong Ramirez Maria I Carmeliet Peter Cummings Richard D Lupu Florea Xia 2008Journal of Clinical Investigation2008,,11:1
11Association of human cyclinE with a periodic G1-S phase protein kinase显示文摘Vjekoslav D Emma L Steven I R 1992Science1992,257,25:1
12Hypo-activity induced skeletal muscle atrophy and potential nutritional interventions: A review显示文摘Periods of hypo-activity result in profound changes in skeletal muscle morphology and strength. This review primarily addresses the differential impact of de-training, bed-rest, limb immobilisation and unilateral lower limb suspension on muscle morphology, strength and fatigability. The degree of muscle atrophy differs depending on the hypo-activity model and the muscles in question, with the leg and postural muscles being the most susceptible to atrophy. Hypo-activity also results in the dramatic loss of strength that often surpasses the loss of muscle mass, and consequently, the nervous system and contractile properties adapt to adjust for this excessive loss of strength. In addition, the degree of muscle strength loss is different depending on the hypo-activity model, with immobilisation appearing to have a greater impact on strength than unloaded models. There is a step-wise difference in the magnitude of muscle loss so that, even after accounting for differential durations of interventions immobilisation ≥ unilateral lower limb suspension ≥ bed-rest ≥ de-training. Muscle fatigability varies between hypoactivity models but the results are equivocal and thismay be due to task-specific adaptations. This review also addresses potential nutritional interventions for attenuating hypo-activity induced muscle atrophy and strength declines, in the absence of exercise. Essential amino acid supplementation stands as a strong candidate but other supplements are good contenders for attenuating hypo-activity induced atrophy and strength losses. Several potential nutritional supplements are highlighted that could be used to combat muscle atrophy but extensive research is needed to determine the most effective.Emma L Bostock Christopher I Morse Keith Winwood Islay Mc Ewan Gladys L Onambélé-Pearson 2013World Journal of Translational Medicine2013,2,3:1
13AnN-acetylglucosamine oligomer binding agglutinin (lectin) from ripe Cyphomandra betacea send fruits 显示文摘Antonio R S Maria I I Emma N Q etal 2001Plant Science2001,160,4:1
14Cultured Olfactory Ensheathing Cells Express Growth Factor, Brain-derived Neurotrophic Factor, Glia Cell Line-derived Neurotrophic Factor and Their Receptors显示文摘Emma W Adrian K W Meng I C 2001Mol Brain Res2001,,88:1
15Conventional type 1 dendritic cells protect against age-related adipose tissue dysfunction and obesity显示文摘Conventional dendritic cells(cDCs)scan and integrate environmental cues in almost every tissue,including exogenous metabolic signals.While cDCs are critical in maintaining immune balance,their role in preserving energy homeostasis is unclear.Here,we showed that Batf3-deficient mice lacking conventional type 1 DCs(cDC1s)had increased body weight and adiposity during aging.This led to impaired energy expenditure and glucose tolerance,insulin resistance,dyslipidemia,and liver steatosis.cDC1 deficiency caused adipose tissue inflammation that was preceded by a paucity of NK1.1+invariant NKT(iNKT)cells.Accordingly,among antigen-presenting cells,cDC1s exhibited notable induction of IFN-γproduction by iNKT cells,which plays a metabolically protective role in lean adipose tissue.Flt3L treatment,which expands the dendritic cell(DC)compartment,mitigated diet-induced obesity and hyperlipidemia in a Batf3-dependent manner.This effect was partially mediated by NK1.1+cells.These results reveal a new critical role for the cDC1-iNKT cell axis in the regulation of adipose tissue homeostasis.Elena Hernández-García Francisco J.Cueto Emma C.L.Cook Ana Redondo-Urzainqui Sara Charro-Zanca Iñaki Robles-Vera Ruth Conde-Garrosa Ivana Nikolić Guadalupe Sabio David Sancho Salvador Iborra 2022Cellular & Molecular Immunology2022,19,2:0
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