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6篇 您的检索式：作者名="Jan NB"

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		题名	作者	年代	出处	被引量
	1	The insulin-like growth factor (IGF) system and its relation to infliximab treatment in adult patients with Crohn’s disease显示文摘	Martin Eivindson Henning Gr?nb?k Kristin Skogstrand Poul Thorsen Jan Frystyk Allan Flyvbjerg Jens F. Dahlerup	2007	Scandinavian Journal of Gastroenterology2007，，4：	1
	2	The insulin-like growth factor (IGF)-system in active ulcerative colitis and Crohn’s disease: Relations to disease activity and corticosteroid treatment显示文摘	Martin Eivindson Henning Gr?nb?k Allan Flyvbjerg Jan Frystyk Erik Zimmermann-Nielsen Jens Frederik Dahlerup	2006	Growth Hormone & IGF Research2006，，1：	1
	3	Procalcitonin reflects bacteremia and bacterial load in urosepsis syndrome:a prospective observational study显示文摘	Cees N Tobias NB Jan WW	2010	Critical Care2010，14，6：	1
	4	Peptide Receptor Radionuclide Therapy with Y-DOTATOC and 177Lu-DOTATOC in Advanced Neuroendocrine Tumors: Results from a Danish Cohort Treated in Switzerland显示文摘	Pfeifer Andreas Klaus Gregersen Tine Gr?nb?k Henning Hansen Carsten Paln?s Müller-brand Jan Herskind Bruun Karin Krogh Klaus Kj?r Andreas Knigge Ulrich	2011	EN2011，，3：	1
	5	Combination therapy in hy- pertension显示文摘	Alan HG Jan NB Barry LC	2010	JASH2010，4，1：	1
	6	Soluble membrane attack complex in ascites in patients with liver cirrhosis without infections显示文摘AIM: To study complement activation in 46 patients with alcoholic cirrhosis and ascites but no spontaneous bacterial peritonitis (SBP) and 10 healthy controls. METHODS: Complement activation was determined by the measurement of soluble membrane attack complex (sMAC) concentrations in ascites and plasma. In patients, metabolic liver function was determined by the galactose elimination capacity and the clinical status assessed by the Model of End-Stage Liver Disease and Child-Pugh scores. RESULTS: Ascites sMAC levels were markedly higherthan in the corresponding plasma sample (median (range): 596 (170 - 1519) vs 160 (77 - 848) μg/L; P < 0.01). Ascites sMAC levels correlated positively with liver status. There was no relationship between ascites sMAC and leukocyte count. No relationship between ascites sMAC and blood C-reactive protein, albumin or neutrophile count was found. Plasma sMAC concentrations were slightly higher in patients than in controls [130 μg/L (70 - 204); P = 0.04]. Neither sMAC in ascites nor plasma was related to mortality. CONCLUSION: The increased sMAC concentration in ascites and plasma indicate an activation of the complement system in cirrhosis even in the absence of SBP. This was particularly evident in the peritoneal fluid and most marked in patients with preserved liver status. The high ascites sMAC levels may reflect transudation of membrane attack complexes from the liver. Whether this complement activation has any clinical implications remains to be clarified.	Mette Bjerre Peter Holland-Fischer Henning Grφnbk Jan Frystyk Troels K Hansen Hendrik Vilstrup Allan Flyvbjerg	2010	World Journal of Hepatology2010，2，6：	0

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