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| 1 | Epidemiological, laboratory, diagnostic and public health aspects of human brucellosis in western Iran显示文摘Objective:To determine brucellosis's epidemiologic,laboratory,diagnostic and public health features considering brucellosis is endemic in Azna County,western Iran.Methods:This descriptive cross-sectional study was investigated on 43 patients with brucellosis in Azna County. The subjects were the patients with symptoms correspondent with brucellosis and positive Wright and 2ME tests. A questionnaire about demographic,epidemiological and laboratory findings was filled in. Afterwards,patients were treated using usual antimicrobial drugs regimen. The collected data were analyzed by SPSS software version 16.Results:Forty-three subjects were found to be positive in laboratory tests. Incidence of Brucellosis was 59.31 per hundred thousand population. About 34.9% of patients were female and 65.1%male. Nearly 95.2% of human cases were living in rural and 4.8%in urban areas. Around 20.9%of patients had history of animal contact. The commonest transmission was unpasteurized dairy products(79.1%). The most contagious seasons were summer and spring(60.3%). The most common age group was 15-24(27.9%),and about 60.5%of the patients were between 15-44 years old. Disease was more common among housewives(30.2%) and farmers(20.9%). The majority of the patients had Wright test titre=1:320(54.1%) and 2ME test titre=1:160(56.1%) in serological titration.Doxycycline with Rifampin was used for treatment of the greatest of patients(60.4%).Conclusions:In order to control this zoonotic disease,close cooperation of health and veterinary organizations is necessary. | Hamid Kassiri Hamid Amani MassoudLotfi | 2013 | Asian Pacific Journal of Tropical Biomedicine2013，3，8： | 19 |
| 2 | 基质金属蛋白酶1的组织抑制剂通过介导CD63与整合素β_1的相互作用促进心肌纤维化显示文摘心肌纤维化指细胞外基质纤维胶原蛋白的过度积累。纤维化是各种心肌病的关键特征,会影响心脏收缩和舒张功能。金属蛋白酶1组织抑制剂(tissue inhibitor of metalloproteinase-1,TIMP1)水平在心肌纤维化过程中始终增加,被认为是纤维化的标记物。 | Takawale A Zhang P Patel VB Wang X Oudit G Kassiri Z 刘莉 叶鹏 | 2017 | 中华高血压杂志2017，25，4： | 13 |
| 3 | The molecular physiology of the cardiac transient outward potassium current (Ito) in normal and diseased myocardium显示文摘 | Oudit G Y Kassiri Z Sah R | 2001 | J Mol Cell Cardiol2001，33，： | 3 |
| 4 | Angiotensin-Converting Enzyme 2 Suppresses Pathological Hypertrophy, Myocardial Fibrosis, and Cardiac Dysfunction显示文摘 | JiuChang Zhong Ratnadeep Basu Danny Guo Fung L. Chow Simon Byrns Manfred Schuster Hans Loibner Xiu-hua Wang Josef M. Penninger Zamaneh Kassiri Gavin Y. Oudit | 2010 | Circulation2010，，7： | 2 |
| 5 | Reduction of Ito causes hypertrophy in neonatal rat ventricular myocytes显示文摘 | Zobel C Nguyen TT | 2002 | Circ Res2002，90，： | 1 |
| 6 | The role of action potential prolongation and altered intracellular calcium handling in the pathogenesis of heart failure 显示文摘 | Wickenden AD Kaprielian R Kassiri Z | 1998 | Cardiovasc Res1998，37，： | 1 |
| 7 | Loss of angiotensin-eonverting enzyme 2 accelerates maladaptive left ventricular remodeling in response to myocardial infarction 显示文摘 | Kassiri Z Zhong Jiuchang Guo Danny | 2009 | Circ Heart Fail2009，2，5： | 1 |
| 8 | Angiotensin Ⅱ-mediated oxi- dative stress and inflammation mediate the age-dependent cardiomy- opathy in ACE2 null mice 显示文摘 | Oudit GY Kassiri Z Patel MP | 2007 | Cardiovasc Res2007，75，1： | 1 |
| 9 | Role ofPI3 kinase gamma in excitation contraction coupling and heart disease 显示文摘 | Oudit GY Kassiri Z | 2007 | Cardiovasc Hematol Disord Drug Targets2007，7，4： | 1 |
| 10 | Role of signaling pathways in the myocardial response to biomechanical stress and in mechanotransduction in the heart显示文摘 | GUO D KASSIRI Z OUDIT GY | | 0，，02： | 1 |
| 11 | Relationship between K+ channel down-regulation and I in rat ventricular myocytes fo llowing myocardial infarction显示文摘 | Wickenden AD Kassiri Z | 1999 | J Physiol1999，517，： | 1 |
| 12 | TIMP3 defi-ciency leads to dilated cardiomyopathy显示文摘 | FEDAK PW SMOOKLER DS KASSIRI Z | 2004 | Circulation2004，110，16： | 1 |
| 13 | Cortical tubers, cognition, and epilepsy in tuberous sclerosis显示文摘 | Kassiri J Snyder TJ Bhargava R | 2011 | Pediatr Neurol2011，，44： | 1 |
| 14 | Loss of angiotensin-converting en- zyme 2 accelerates maladaptive left ventrieular remodeling in re- sponse to myocardial infarction 显示文摘 | Kassiri Z Zhong J Guo D | 2009 | Circ Heart Fail2009，2，5： | 1 |
| 15 | Angiotensin Ⅱ mediated oxidative stress and inflammation mediate the age-dependent cardiomyopathy in ACE2 null mice显示文摘 | Kassiri Z Patel MP | 2007 | Cardiovasc Res2007，75，： | 1 |
| 16 | Relationship between K+ channel downregulation and i in rat ventricular myocytes following myocardial infarction显示文摘 | Kaprielian R Wickenden AD Kassiri Z | 1999 | J Physiol1999，517，： | 1 |
| 17 | Loss of angiotensin - converting enzyme 2 accelerates maladaptive left ventricular remodeling in re- sponse to myocardial infarction 显示文摘 | Kassiri Z Zhong J Guo D | 2009 | Cire Heart Fail2009，2，5： | 1 |
| 18 | Metalloproteinase inhibitor TIMP - 1 affects hepatocyte cell cycle via HGF activation in murine liver regeneration 显示文摘 | Mohammed FF Pennington C J Kassiri Z | 2005 | Hepatology2005，41，4： | 1 |
| 19 | TIMP-3 deficiency leads to dilated cardiomyopathy显示文摘 | Fedak PW Smooklcr DS Kassiri Z | 2004 | Circulation2004，110，16： | 1 |
| 20 | Loss of angiotensin-converting enzyme 2 accelerates alaladaptive left ventricular reveling in response to myocardial infarction显示文摘 | Kassiri Z Zhong J Guo D | 2009 | Circ Heart Fail2009，2，： | 1 |
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