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7篇 您的检索式：作者名="Noboru Fukuda"

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		题名	作者	年代	出处	被引量
	1	Moderate low temperature preserves the stemness of neural stem cells and suppresses apoptosis of the cells via activation of the cold-inducible RNA binding protein显示文摘	Kosuke Saito Noboru Fukuda Taro Matsumoto Yuji Iribe Akiko Tsunemi Tomohiko Kazama Chikako Yoshida-Noro Nariyuki Hayashi	2010	Brain Research2010，，：	1
	2	Surgical result of skull base surgery for the treatment of head and neck malignancies involving skull base:multi-institutional studies on 143 cases in Japan显示文摘	Satoshi Fukuda Noboru Sakai Shin-Etsu Kamata	2001	Auris Nasus Larynx2001，28，：	1
	3	Contribution of synthetic pheontype on the enhanced angiotensin Ⅱ-generating system in vascular smooth muscle cells from spontaneously hypertensive rats 显示文摘	Fukuda Noboru Flu WY Satoh Chikara	1999	Hypertension1999，17，8：	1
	4	Growth Characteristics,angiotensin Ⅱ generation,and microarray-determinede gene expression in vascular smooth muscle cells from young spontaneously hypertensive rats显示文摘	Hu WY Fukuda Noboru Kanmatsuse Katsuo	2002	Hypertension2002，20，7：	1
	5	Angiotensin Ⅱ type 2 receptor gene transfer downregulates Angiotensin Ⅱ type 1a receptor in vascular smooth muscle cells 显示文摘	Xue-Qing Jin Noboru Fukuda Jin Zi	2002	Hypertension2002，39，10：	1
	6	Inhibition of malignant phenotypes of human osteosarcoma cells by a gene silencer, a pyrrole–imidazole polyamide, which targets an E-box motif显示文摘	Masashi Taniguchi Kyoko Fujiwara Yuji Nakai Toshinori Ozaki Nobuko Koshikawa Kojima Toshio Motoaki Kataba Asako Oguni Hiroyuki Matsuda Yukihiro Yoshida Yasuaki Tokuhashi Noboru Fukuda Takahiro Ueno Masayoshi Soma Hiroki Nagase	2014	FEBS Open Bio2014，，：	1
	7	Review of novel therapeutic medicines targeting androgen signaling in castration-resistant prostate cancer显示文摘Prostate cancer is the most common male malignant neoplasm.Androgens and the androgen receptor(AR)play a key role in the onset and progression of prostate cancer.The expression of the AR is still preserved in the majority of patients with castration-resistant prostate cancer(CRPC).CRPC is considered to be induced by the following mechanisms:(1)sustained AR activation by enhancing intracellular conversion of adrenal androgens to dehydrotestosterone via a de novo route;(2)AR hypersensitivity;(3)promiscuous activation of AR signaling;and(4)outlaw pathways.Recent advances in the treatment of CRPC include novel medicinestargeting AR signaling pathways.In addition,functional molecular studies have shown that some of the ARregulated genes and AR coregulators are prognostic markers and potential therapeutic targets for prostate cancer,particularly in the castration-resistant state.Therefore,identification of the AR signaling pathways responsible for establishment of CRPC is critical for developing new strategies for the treatment of CRPC.	Daisuke Obinata Kyoko Fujiwara Kenya Yamaguchi Ken-ichi Takayama Tomohiko Urano Hiroki Nagase Satoshi Inoue Satoru Takahashi Noboru Fukuda	2014	World Journal of Clinical Urology2014，3，3：	0

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