|
|
|
题名
|
作者
|
年代
|
出处
|
被引量
|
| 1 | Autophagy is a key feature in the pathogenesis of systemic sclerosis显示文摘 | Tracy Frech Ivana Domenico Maureen A. Murtaugh Monica P. Revelo Dean Y. Li Allen D. Sawitzke Stavros Drakos | 2014 | Rheumatology International2014,,3: | 1 |
| 2 | Family support, financial needs, and health care needs of rural elderly in China: A field study显示文摘 | Hong Li Martin B. Tracy | 1999 | Journal of Cross - Cultural Gerontology1999,,4: | 1 |
| 3 | Use of Emergency Medical Service Transport Among Patients With ST-Segment–Elevation Myocardial Infarction: Findings From the National Cardiovascular Data Registry Acute Coronary Treatment Intervention Outcomes Network Registry–Get With the Guidelines显示文摘 | Robin Mathews Eric D. Peterson Shuang Li Matthew T. Roe Seth W. Glickman Stephen D. Wiviott Jorge F. Saucedo Elliott M. Antman Alice K. Jacobs Tracy Y. Wang | 2011 | Circulation2011,,2: | 1 |
| 4 | Relationship of hemolymph phenol oxidase and mosquito age in Aedes aegypti显示文摘 | LI J TRACY J W CHRISTENSEN B M | 1992 | J Invertebrate Pathol1992,60,2: | 1 |
| 5 | Syncytin-1 modulates placental trophoblast cell proliferation by promoting G1/S transition显示文摘 | Qiang Huang Jinping Li Fengchao Wang Matthew T. Oliver Tracy Tipton Ya Gao Shi-Wen Jiang | 2013 | Cellular Signalling2013,,4: | 1 |
| 6 | Association between depression and vascular disease in systemic lupus erythematosus显示文摘 | Carol MG Tracy LI Abdus S | 2012 | J Rheumatol2012,39,2: | 1 |
| 7 | Dapagliflozin as Monotherapy in Drug-Naive Asian Patients With Type 2 Diabetes Mellitus: A Randomized, Blinded, Prospective Phase III Study显示文摘 | Linong Ji Jianhua Ma Hongmei Li Traci A. Mansfield Caroline L. T’joen Nayyar Iqbal Agata Ptaszynska James F. List | 2013 | Clinical Therapeutics2013,,: | 1 |
| 8 | Methicillin-resistant Staphylococus aureus,Hwwaii,2000~2002显示文摘 | Li Fenfang Sarah Y Park Tracy L Ayers | 2005 | Emerg lnfect Dis2005,11,8: | 1 |
| 9 | Safety and efficacy of fingolimod in patients with relapsing-remitting multiple sclerosis (FREEDOMS II): a double-blind, randomised, placebo-controlled, phase 3 trial显示文摘 | Peter A Calabresi Ernst-Wilhelm Radue Douglas Goodin Douglas Jeffery Kottil W Rammohan Anthony T Reder Timothy Vollmer Mark A Agius Ludwig Kappos Tracy Stites Bingbing Li Linda Cappiello Philipp von Rosenstiel Fred D Lublin | 2014 | Lancet Neurology2014,,6: | 1 |
| 10 | Relationship of the hemolymph phenol oxidase and mosquito age in Aedes aegypti显示文摘 | Li J Tracy JW Christensen BM | 1992 | J Invertebr Pathol1992,60,2: | 1 |
| 11 | GLP-1 receptor stimulation preserves primary cortical and dopaminergic neurons in cellular and rodent models of stroke and Parkinsonism显示文摘 | Li YZ Tracy AP Mark S | 2009 | PNAS2009,106,4: | 1 |
| 12 | Cardiovascular effects of pulmonary exposure to single-wall carbon nanotubes显示文摘 | Li Zheng Tracy Hulderman Rebecca Salmen | 2007 | Environmental Health Perspectives2007,115,3: | 1 |
| 13 | Free cholesterol-loaded macrophage are an abundant source of tumor necrosis factor-a and interleukin- 6 显示文摘 | Yankun li Robert FS Tracie DS | 2005 | J Biol Chem2005,280,21: | 1 |
| 14 | Phenol oxidase activity in hemolymph compartments of Aedes aegypti during melanotic encapsulation reactions against microfilariae显示文摘 | Li J Tracy JW Christensen BM | 1992 | Dev Comp Immunol1992,16,1: | 1 |
| 15 | Relationship of hemolymph phenol oxidase and mosquito age in Aedes aegypti显示文摘 | Li J Tracy JW Christensen BM | 1992 | J Invertebrate Pathol1992,60,2: | 1 |
| 16 | Preserving cognitive function in patients with Alzheimer's disease:The Alzheimer's disease neuroprotection research initiative(ADNRI)显示文摘The global trend toward aging populations has resulted in an increase in the occurrence of Alzheimer's disease(AD)and associated socioeconomic burdens.Abnormal metabolism of amyloid-β(Aβ)has been proposed as a significant pathomechanism in AD,supported by results of recent clinical trials using anti-Aβantibodies.Nonetheless,the cognitive benefits of the current treatments are limited.The etiology of AD is multifactorial,encompassing Aβand tau accumulation,neuroinflammation,demyelination,vascular dysfunction,and comorbidities,which collectively lead to widespread neurodegeneration in the brain and cognitive impairment.Hence,solely removing Aβfrom the brain may be insufficient to combat neurodegeneration and preserve cognition.To attain effective treatment for AD,it is necessary to(1)conduct extensive research on various mechanisms that cause neurodegeneration,including advances in neuroimaging techniques for earlier detection and a more precise characterization of molecular events at scales ranging from cellular to the full system level;(2)identify neuroprotective intervention targets against different neurodegeneration mechanisms;and(3)discover novel and optimal combinations of neuroprotective intervention strategies to maintain cognitive function in AD patients.The Alzheimer's Disease Neuroprotection Research Initiative's objective is to facilitate coordinated,multidisciplinary efforts to develop systemic neuroprotective strategies to combat AD.The aim is to achieve mitigation of the full spectrum of pathological processes underlying AD,with the goal of halting or even reversing cognitive decline. | Jie Liu Heleen van Beusekom Xian-Le Bu Gong Chen Paulo Henrique Rosado de Castro Xiaochun Chen Xiaowei Chen Andrew N.Clarkson Tracy D.Farr Yuhong Fu Jianping Jia Jukka Jolkkonen Woojin Scott Kim Paula Korhonen Shen Li Yajie Liang Guang-Hui Liu Guiyou Liu Yu-Hui Liu Tarja Malm Xiaobo Mao Joaquim Miguel Oliveira Mike M.Modo Pedro Ramos-Cabrer Karsten Ruscher Weihong Song Jun Wang Xuanyue Wang Yun Wang Haitao Wu Lize Xiong Yi Yang Keqiang Ye Jin-Tai Yu Xin-Fu Zhou Marietta Zille Colin L.Masters Piotr Walczak Boltze Johannes Xunming Ji Yan-Jiang Wang | 2023 | Neuroprotection2023,1,2: | 0 |
| 17 | Racial Differences in Left Ventricular Filling Pressure Following Acute Aerobic Exercise Between Chinese and Caucasians显示文摘Background Left ventricular filling pressure(LVFP)is an important early indicator of heart failure that is more prevalent in Caucasians than Chinese.Exercise-induced change in LVFP may provide more incremental information to assess dias-tolic function.But it was unknown whether there was difference in LVFP following acute exercise between Caucasians and Chinese.Purpose The purpose of this study was to investigate the change of LVFP following an acute 45-min aerobic exercise in healthy Caucasian and Chinese individuals.Methods Sixty participants(30 Caucasians and 30 Chinese,half was male,respectively)performed an acute bout of aero-bic exercise at 70%of heart rate reserve.Hemodynamics,Left ventricle(LV)morphology and function parameters were measured at baseline,then at 30-min and 60-min post-exercise.Results There was a similar LV ejection fraction,LV fraction shorten,lateral E/eʹand lateral eʹbetween Chinese and Caucasians at baseline.There was a significant race-by-time interaction in lateral E/eʹand lateral eʹbetween Chinese and Caucasians from pre-exercise to 30 min and 60 min after acute aerobic exercise.TheΔE/eʹwas significant correlated with baseline systolic blood pressure.Conclusion The change of LVFP was different between Chinese and Caucasians following acute aerobic exercise.The racial differences may be primarily caused by the changes of LV relaxation following exercise,baseline systolic blood pressure may also contribute to the differences. | Peng Sun Huimin Yan Sushant M.Ranadive Abbi D.Lane Rebecca MKappus Tracy Baynard Xiaodan Sun Shichang Li Bo Fernhall | 2022 | Journal of Science in Sport and Exercise2022,4,3: | 0 |
| 18 | Join us on an amazing journey towards next-generation treatments for CNS disorders:Launch of Neuroprotection,a new high-quality journal in translational neuroscience显示文摘Translational medicine in neurodegenerative and neurovascular diseases is approaching a breakthrough point.Recent years have led to dramatic progress in both experimental and clinical research.Based on a much better and continuously increasing understanding of disease mechanisms,progression and pathophysiology,new therapies with an improved translational potential to protect tissue either against acute or chronic degeneration and even approaches potentially capable of repairing damaged brain tissue are emerging. | Xunming Ji Piotr Walczak Heleen M.M.van Beusekom Ana I.Casas Andrew Clarkson Tracy Farr Jukka Jolkkonen Yajie Liang Michel M.Modo Paulo H.Rosado-de-Castro Karsten Ruscher Yan-Jiang Wang Haitao Wu Marietta Zille Shen Li Johannes Boltze | 2023 | Neuroprotection2023,1,1: | 0 |