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| 1 | Medical errors, infection-control breaches and the use of adulterated and misbranded medical devices显示文摘Several well-publicized cases of improper cleaning,disinfection or sterilization of contaminated reusable medical equipment that posed an increased risk of patientto-patient disease transmission were reported within the past few years,resulting in the notification of approximately 20 000 patients.These medical errors,the specific infection-control standards they breached,and assessments of the risk of infection associated with each are discussed.Other topics discussed include the Food and Drug Administration’s(FDA)regulation of medical devices and infection-control products;the use of adulterated,misbranded,and investigational devices;consent decrees and associated Certificates of Medical Necessity;and informed patient consent.Focus is placed on liquid chemical sterilization,its history,and the FDA’s recent censure and discontinuation of a medical device labeled with this claim,namely,the STERIS System 1 processor.Recommendations are provided for healthcare facilities,regulatory agencies,manufacturers of reusable medical devices,and professional healthcare organizations and administrations to improve public health and prevent healthcareassociated infections. | Lawrence F Muscarella | 2012 | World Journal of Clinical Infectious Diseases2012，2，2： | 3 |
| 2 | Role of interleukin 28-B in the spontaneous and treatment-related clearance of HCV infection in patients with chronic HBV/HCV dual infection显示文摘 | N. Coppola A. Marrone M. Pisaturo M. Starace G. Signoriello I. Gentile L. E. Adinolfi E. Sagnelli R. Zampino | 2014 | European Journal of Clinical Microbiology & Infectious Diseases2014，，4： | 3 |
| 3 | Acinetobacter baumannii:An emerging pathogenic threat to public health显示文摘Over the last three decades, Acinetobacter has gained importance as a leading nosocomial pathogen, partly due to its impressive genetic capabilities to acquire resistance and partly due to high selective pressure, especially in critical care units. This low-virulence organism has turned into a multidrug resistant pathogen and now alarming healthcare providers worldwide. Acinetobacter baumanni(A. baumannii) is a major species, contributing about 80% of all Acinetobacter hospital-acquired infections. It disseminates antibiotic resistance by virtue of its extraordinary ability to accept or donate resistance plasmids. The procedures for breaking the route of transmission are still proper hand washing and personal hygiene(both the patient and the healthcare professional), reducing patient's biofilm burden from skin, and judicious use of antimicrobial agents. The increasing incidence of extended-spectrum beta-lactamases and carbapenemases in A. baumannii leaves almost no cure for these 'bad bugs'.To control hospital outbreaks of multidrug resistantAcinetobacter infection, we need to contain their dissemination or require new drugs or a rational combination therapy. The optimal treatment for multidrug-resistant A. baumannii infection has not been clearly established, and empirical therapy continues to require knowledge of susceptibility patterns of isolates from one's own institution. This review mainly focused on general features and introduction to A. baumannii and its epidemiological status, potential sources of infection, risk factors, and strategies to control infection to minimize spread. | Suresh G Joshi Geetanjali M Litake | 2013 | World Journal of Clinical Infectious Diseases2013，3，3： | 3 |
| 4 | The largest outbreak of hand; foot and mouth disease in Singapore in 2008: The role of enterovirus 71 and coxsackievirus A strains显示文摘 | Yan Wu Andrea Yeo M.C. Phoon E.L. Tan C.L. Poh S.H. Quak Vincent T.K. Chow | 2010 | International Journal of Infectious Diseases2010，，： | 3 |
| 5 | Echinococcosis: a review显示文摘 | Pedro Moro Peter M. Schantz | 2008 | International Journal of Infectious Diseases2008，，2： | 3 |
| 6 | Inducible nitric oxide synthase and histopathological correlation in chronic viral hepatitis显示文摘 | Esin Atik Yusuf Onlen Lutfu Savas Figen Doran | 2007 | International Journal of Infectious Diseases2007，，1： | 2 |
| 7 | Anti-hepatitis C virus treatment may prevent the progression of liver fibrosis in non-responder human immunodeficiency virus/hepatitis C virus coinfected patients显示文摘 | Caterina Sagnelli Caterina Uberti-Foppa Laura Galli Giuseppe Pasquale Nicola Coppola Luca Albarello Carlo Doglioni Adriano Lazzarin Evangelista Sagnelli | 2013 | Brazilian Journal of Infectious Diseases2013，，： | 2 |
| 8 | An overview of a diagnostic and epidemiologic reappraisal of cutaneous leishmaniasis in Iran显示文摘 | Mahin Farahmand Hossein Nahrevanian Hasti Atashi Shirazi Sabah Naeimi Zahra Farzanehnejad | 2011 | Brazilian Journal of Infectious Diseases2011，，1： | 2 |
| 9 | Hepatitis B virus genotyping: current methods and clinical implications显示文摘 | Bassem S.S. Guirgis Radwa O. Abbas Hassan M.E. Azzazy | 2010 | International Journal of Infectious Diseases2010，，11： | 2 |
| 10 | Association between insulin resistance and sustained virologic response in hepatitis C treatment, genotypes 1 versus 2 and 3: systematic literature review and meta-analysis显示文摘 | Marcela Pezzoto Laurito Edison Roberto Parise | 2013 | Brazilian Journal of Infectious Diseases2013，，： | 2 |
| 11 | Physiological functions and clinical implications of fibrinogen-like 2:A review显示文摘Fibrinogen-like 2(FGL2) encompasses a transmembrane(m FGL2) and a soluble(s FGL2) form with differential tertiary structure and biological activities. Typically, m FGL2 functions as prothrombinase that is capable of initiating coagulation in tissue without activation of the blood clotting cascade, whereas s FGL2 largely acts as an immunosuppressor that can repress proliferation of alloreactive T lymphocytes and maturation of bone marrow dendritic cells. Protein sequences of FGL2 exhibit evolutionary conservation across wide variety of species, especially at the carboxyl terminus that contains fibrinogen related domain(FRED). The FRED of FGL2 confers specificity and complexity in the action of FGL2, including receptor recognition, calcium affiliation, and substrate binding. Constitutive expression of FGL2 during embryogenesis and in mature tissues suggests FGL2 might be physiologically important. However, excessive induction of FGL2 under certain medical conditions(e.g. pathogen invasion) could trigger complement activation, inflammatory response,cellular apoptosis, and immune dysfunctions. On the other hand, complete absence of FGL2 is also detrimental as lack of FGL2 can cause autoimmune glomerulonephritis and acute cellular rejection of xenografts. All these roles involve m FGL2, s FGL2, or their combination. Although it is not clear how m FGL2 is cleaved off its host cells and secreted into the blood, circulating s FGL2 has been found correlated with disease severity and viral loading among patients with human hepatitis B virus or hepatitis C virus infection. Further studies are warranted to understand how FGL2 expression is regulated under physiological and pathological conditions. Even more interesting is to determine whether m FGL2 can fulfill an immunoregulatory role through its FRED at carboxyl end of the molecule and, and vice versa, whether s FGL2 is procoagulant upon binding to a target cell. Knowledge in this area should shed light on development of s FGL2 as an alternative immunosuppressive agent for organ transplantation or as a biomarker for predicting disease progression, monitoring therapeutic effects, and targeting FGL2 for repression in ameliorating fulminant viral hepatitis. | Genyan Yang W Craig Hooper | 2013 | World Journal of Clinical Infectious Diseases2013，3，3： | 2 |
| 12 | Fatal Outcome of Lung Transplantation in Cystic Fibrosis Patients due to Small-Colony Variants of the Burkholderia cepacia Complex显示文摘 | S. H?u?ler C. Lehmann C. Breselge M. Rohde M. Cla?en B. Tümmler P. Vandamme I. Steinmetz | 2003 | European Journal of Clinical Microbiology & Infectious Diseases2003，，4： | 2 |
| 13 | Risk factors associated with high prevalence rates of hepatitis C infection in Egypt显示文摘 | Celeste Reker K.M. Islam | 2014 | International Journal of Infectious Diseases2014，，： | 2 |
| 14 | Antimicrobial resistance among commensal isolates of Escherichia coli and Staphylococcus aureus in the Indonesian population inside and outside hospitals显示文摘 | E. S. Lestari J. A. Severin P. M. G. Filius K. Kuntaman D. O. Duerink U. Hadi H. Wahjono H. A. Verbrugh | 2008 | European Journal of Clinical Microbiology & Infectious Diseases2008，，1： | 2 |
| 15 | Clinical manifestations and complications in 1028 cases of brucellosis: a retrospective evaluation and review of the literature显示文摘 | Turan Buzgan Mustafa Kasim Karahocagil Hasan Irmak Ali Irfan Baran Hasan Karsen Omer Evirgen Hayrettin Akdeniz | 2009 | International Journal of Infectious Diseases2009，，6： | 2 |
| 16 | Hepatitis C virus infection prevalence and liver dysfunction in a cohort of B-cell non-Hodgkin’s lymphoma patients treated with immunochemotherapy显示文摘 | L. Nosotti M. D’Andrea A. Pitidis F. Pimpinelli M. L. Dessanti F. Pisani P. Vignally M. C. Petti | 2012 | Scandinavian Journal of Infectious Diseases2012，，1： | 2 |
| 17 | Overview of neurobrucellosis: a pooled analysis of 187 cases显示文摘 | Hanefi Cem Gul Hakan Erdem Semai Bek | 2009 | International Journal of Infectious Diseases2009，，6： | 2 |
| 18 | Elevated plasma sphingomyelin (d18:1/22:0) is closely related to hepatic steatosis in patients with chronic hepatitis C virus infection显示文摘 | J.-F. Li F. Qu S.-J. Zheng H.-L. Wu M. Liu S. Liu Y. Ren F. Ren Y. Chen Z.-P. Duan J.-L. Zhang | 2014 | European Journal of Clinical Microbiology & Infectious Diseases2014，，10： | 2 |
| 19 | Trends in overall opportunistic illnesses, Pneumocystis carinii pneumonia, cerebral toxoplasmosis and Mycobacterium avium complex incidence rates over the 30 years of the HIV epidemic: a systematic review显示文摘 | Lara Coelho Valdiléa Gon?alves Veloso Beatriz Grinsztejn Paula Mendes Luz | 2013 | Brazilian Journal of Infectious Diseases2013，，： | 2 |
| 20 | Bordetella bronchiseptica pleural infection in a patient with AIDS显示文摘 | VIEJO G DE LA IGLESIA P OTERO L | 2002 | Scandinavian Journal of Infectious Diseases2002，34，8： | 1 |
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