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| 1 | CTGF antagonism with mAb FG-3019 enhances chemotherapy response without increasing drug delivery in murine ductal pancreas cancer显示文摘 | Albrecht Neesse Kristopher K. Frese Tashinga E. Bapiro Tomoaki Nakagawa Mark D. Sternlicht Todd W. Seeley Christian Pilarsky Duncan I. Jodrell Suzanne M. Spong David A. Tuveson | 2013 | Proceedings of the National Academy of Sciences2013，，30： | 3 |
| 2 | Stromal biology and therapy in pancreatic cancer显示文摘 | Neesse A Michl P Frese KK | 2011 | Gut2011，60，6： | 1 |
| 3 | The pancreas cancer microenvironment 显示文摘 | FEIG C GOPINATHAN A NEESSE A | 2012 | Clin Cancer Res2012，18，16： | 1 |
| 4 | Classification overflow显示文摘 | Schubert H Bohme S Neesse T | 1986 | Aufbereitung-Technik1986，6，： | 1 |
| 5 | ACBP-a PPAR and SREBP modulated housekeeping gene 显示文摘 | Neess D Kiilerich P Sandberg M B | 2006 | Molecular and Cellular Biochemistry2006，284，1： | 1 |
| 6 | Sperm-associated antigen 1 is expressed early in pancreatic tumorigenesis and pro- motes motility of cancer cells 显示文摘 | Neesse A Gangeswaran R Luettges J | 2007 | Oncogene2007，26，11： | 1 |
| 7 | Glutanlate receptor GRIA3 : target of CUX1 and mediator of tumor progression in pancreatic canoe 显示文摘 | Ripka S Riedel J Neesse A | 2010 | Neoplasia2010，12，8： | 1 |
| 8 | nab-Paclitaxel potentiates gemcitabine activity by reducing cytidine deaminase levels in a mouse model of pancreatic cancer显示文摘 | Frese KK Neesse A Cook N | 2012 | Cancer Discov2012，2，3： | 1 |
| 9 | Dynamic modelling of the hydrocyclone显示文摘 | Th. Neesse J. Dueck | 2006 | Minerals Engineering2006，，4： | 1 |
| 10 | Flow conditions in the air core of the hydrocyclone显示文摘 | Hararah M A Endres E Dueck J Minkov L Neesse T | | 0，，4： | 1 |
| 11 | Air core formation in the hydrocyclone 显示文摘 | Neesse T Dueck J | 2007 | Minerals Engineering2007，20，： | 1 |
| 12 | Dynamic modelling of the hydrocyclone 显示文摘 | Neesse T Dueck J | 2007 | Minerals Engineering2007，20，4： | 1 |
| 13 | Dynamic modelling of the hydrocyclone 显示文摘 | Neesse T Dueck J | 2007 | Minerals Engineering2007，20，4： | 1 |
| 14 | The pancreas cancer mi- croenvironment 显示文摘 | Feig C Gopinathan A Neesse A | 2012 | Clin Cancer Res2012，18，16： | 1 |
| 15 | Direct histological processing of EUS biopsies enables rapid molecular biomarker analysis for interventional pancreatic cancer trials显示文摘 | Rebecca J. Brais Susan E. Davies Maria O’Donovan Ben W. Simpson Natalie Cook Walter C. Darbonne Sian Chilcott Martijn P. Lolkema Albrecht Neesse Michelle Lockley Pippa G. Corrie Duncan I. Jodrell Raaj K. Praseedom Emmanuel L. Huguet Asif Jah Neville V. Ja | 2012 | Pancreatology2012，，1： | 1 |
| 16 | The pancreas cancer microenvironment 显示文摘 | Feig C Gopinathan A Neesse A | 2012 | Clinical Cancer Research2012，18，16： | 1 |
| 17 | Claudin-4 as therapeutic target in cancer 显示文摘 | NEESSE A GRIESMANN H GRESS T M | 2012 | Arch Biochem Biophys2012，524，1： | 1 |
| 18 | Metabolic programming determines the lineage-differentiation fate of murine bone marrow stromal progenitor cells显示文摘Enhanced bone marrow adipogenesis and impaired osteoblastogenesis have been observed in obesity,suggesting that the metabolic microenvironment regulates bone marrow adipocyte and osteoblast progenitor differentiation fate.To determine the molecular mechanisms,we studied two immortalized murine cell lines of adipocyte or osteoblast progenitors(BMSCs^adipo and BMSC^sosteo,respectively)under basal and adipogenic culture conditions.At baseline,BMSCs^adipo,and BMSCs^osteo exhibit a distinct metabolic program evidenced by the presence of specific global gene expression,cellular bioenergetics,and metabolomic signatures that are dependent on insulin signaling and glycolysis in BMSCs^osteo versus oxidative phosphorylation in BMSCs^adipo.To test the flexibility of the metabolic program,we treated BMSCsadipo with parathyroid hormone,S961(an inhibitor of insulin signaling)and oligomycin(an inhibitor of oxidative phosphorylation).The treatment induced significant changes in cellular bioenergetics that were associated with decreased adipocytic differentiation.Similarly,12 weeks of a high-fat diet in mice led to the expansion of adipocyte progenitors,enhanced adipocyte differentiation and insulin signaling in cultured BMSCs.Our data demonstrate that BMSC progenitors possess a distinct metabolic program and are poised to respond to exogenous metabolic cues that regulate their differentiation fate. | Michaela Tencerova Elizabeth Rendina-Ruedy Ditte Neess Nils Faergeman Florence Figeac Dalia Ali Morten Danielsen Anders Haakonsson Clifford J.Rosen Moustapha Kassem | 2019 | Bone Research2019，7，4： | 1 |
| 19 | Stromal biology and therapy in pancreatic cancer显示文摘 | Neesse A Michl P Frese KK | 2011 | Gut2011，60，6： | 1 |
| 20 | The pancreas cancer microenvironment显示文摘 | Feig C Gopinathan A Neesse A | 2012 | Clin Cancer Res2012，18，16： | 1 |
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