|
|
|
题名
|
作者
|
年代
|
出处
|
被引量
|
| 1 | High precision landing site mapping and rover localization for Chang'e-3 mission显示文摘This paper presents the comprehensive results of landing site topographic mapping and rover localization in Chang’e-3 mission.High-precision topographic products of the landing site with extremely high resolutions(up to 0.05 m)were generated from descent images and registered to CE-2 DOM.Local DEM and DOM with 0.02 m resolution were produced routinely at each waypoint along the rover traverse.The lander location was determined to be(19.51256°W,44.11884°N,-2615.451 m)using a method of DOM matching.In order to reduce error accumulation caused by wheel slippage and IMU drift in dead reckoning,cross-site visual localization and DOM matching localization methods were developed to localize the rover at waypoints;the overall traveled distance from the lander is 114.8 m from cross-site visual localization and 111.2 m from DOM matching localization.The latter is of highest accuracy and has been verified using a LRO NAC image where the rover trajeactory is directly identifiable.During CE-3 mission operations,landing site mapping and rover localization products including DEMs and DOMs,traverse maps,vertical traverse profiles were generated timely to support teleoperation tasks such as obstacle avoidance and rover path planning. | LIU ZhaoQin DI KaiChang PENG Man WAN WenHui LIU Bin LI LiChun YU TianYi WANG BaoFeng ZHOU JianLiang CHEN HongMin | 2015 | Science China(Physics,Mechanics & Astronomy)2015，58，1： | 15 |
| 2 | Recombinant CC16 protein inhibits the production of pro-inflammatory cytokines via NF-KB and p38 MAPK pathways in LPS-activated RAW264,7 macrophages显示文摘积累的证据显示克莱拉房间 protein-16 (CC16 ) 有反煽动性的功能,尽管深奥分子的小径完全没被阐明。这里,我们评估了效果肿瘤坏死因素 alpha (TNF-) 的表示上的 recombinant 老鼠 CC16 (rCC16 ) , interleukin-6 (IL-6 ) ,和在 lipopolysaccharide (LPS ) 的 IL-8 刺激了老鼠巨噬细胞(RAW264.7 房间) 并且探索了内在的分子的机制。rCC16 在两个禁止了导致 LPS 的 TNF- , IL-6,和 IL-8 表示,这被发现送信人 ribonucleicacid (mRNA ) 水平和蛋白质以一种集中依赖者方式铺平,由即时反向的 transcriptase 聚合酶链反应和连接酶的 immunosorbent 试金示威了。如此的镇压效果被 transcriptional 活动和原子因素(NF ) 的脱氧核糖核酸酸绑定活动的抑制伴随 -B 然而并非使活跃之物蛋白质(AP )-1。西方的污点分析进一步表明 rCC16 禁止了原子 NF-B 和 NF-B 禁止的蛋白质 IB,和 p38 的 cytosolic NF-B, phosphorylation 和减小的减小的增加激活 mitogen 的蛋白质 kinase (MAPK ) 依赖者由在它的 p65 子单元的 Ser276 的 phosphorylation 的 NF-B 激活。而且, rCC16 被发现没另外在c6月N终端 kinase ,c6月,或c-Jun.的原子 translocation 的 phosphorylation 上有效果,TNF-的减小, IL-6 ,并且 IL-8 被颠倒内长的uteroglobin有约束力的蛋白质的水平什么时候被 RNA 干扰在 对待rCC16 、 对待LPS 的 RAW264.7 房间减少。我们的数据建议 rCC16 由在 RAW264.7 房间使 NF-B 和 p38 MAPK 然而并非 AP-1 失去活性压制调停 LPS 的煽动性的调停人 TNF- , IL-6,和 IL-8 生产。 | Min Pang Yangyang Yuan Dong Wang Ting Li Dan Wang Xiaohong Shi Min Guo Chunfang Wang Xinri Zhang Guoping Zheng Baofeng Yu Hailong Wang | 2017 | Acta Biochimica et Biophysica Sinica2017，49，5： | 14 |
| 3 | Mechanical and Morphological Properties of Highly Dispersed Carbon Nanotubes Reinforced Cement Based Materials显示文摘Stable homogeneous suspensions of multi-walled carbon nanotubes (MWCNTs) were prepared using gum arabic (GA) as dispersant and were incorporated to Portland cement paste. The dispersion was examined by ultraviolet visible spectroscopy (UV-vis), and the concentration measurement shows that the optimum concentration of GA is 0.45 g L-1. The dispersibility of the surface-modified MWCNTs in aqueous solution and cement matrix were investigated by transmission electron microscopy (TEM) and energy dispersive spectroscopy (EDS), and the mechanical properties of the composites were investigated. The results show that the addition of the treated nanotubes can improve both the flexural strength and the compressive strength of the Portland cement composite significantly. The flexural strength of the composite increases up to 43.38% with the MWCNT concentration of 0.08% (by weight of cement). The porosity and pore size distribution of the composites were measured by mercury intrusion porosimetry (MIP), and the results indicate that the cement paste doped with MWCNTs obtained lower porosity and concentrated pore size distribution. The morphological structure was analyzed by field emission scanning electron microscopy (FESEM) and EDS. It is shown that MWCNTs act as bridges and networks across cracks and voids, which transfer the load in case of tension, and the interface bond strength between the nanotubes and matrix is very strong. | 王宝民 HAN Yu PAN Baofeng ZHANG Tingting | 2013 | Journal of Wuhan University of Technology(Materials Science)2013，28，1： | 13 |
| 4 | Construction of plasmid vector pAFP-HSVtk-IRES2-EGFP and its effect on the cytotoxicity of ganciclovir to hepatocellular carcinoma显示文摘 | Lai Zhiyong Qin Qin Yu Baofeng Xie Jun Gao Ranpeng Zhang Tiantian Li Chunfeng Niu Kai Xu Jun | 2014 | Chinese Medical Journal2014，，12： | 6 |
| 5 | Low-frequency unsteadiness of vortex wakes over slender bodies at high angle of attack显示文摘A type of flow unsteadiness with low frequencies and large amplitude was investigated experimentally for vortex wakes around an ogive-tangent cylinder. The experiments were carried out at angles of attack of 60–80 and subcritical Reynolds numbers of 0.6–1.8×105. The reduced frequencies of the unsteadiness are between 0.038 and 0.072, much less than the frequency of Karman vortex shedding. The unsteady flow induces large fluctuations of sectional side forces. The results of pressure measurements and particle image velocimetry indicate that the flow unsteadiness comes from periodic oscillation of the vortex wakes over the slender body. The time-averaged vortex patterns over the slender body are asymmetric, whose orientation is dependent on azimuthal locations of tip perturbations. Therefore, the vortex oscillation is a type of unsteady oscillation around a time-averaged asymmetric vortex structure. | Ma Baofeng Huang Yu Liu Tongxin | 2014 | Chinese Journal of Aeronautics2014，27，4： | 5 |
| 6 | HYD-PEP06 suppresses hepatocellular carcinoma metastasis,epithelial-mesenchymal transition and cancer stem cell-like properties by inhibiting PI3K/AKT and WNT/β-catenin signaling activation显示文摘HYD-PEP06,an endostatin-modified polypeptide,has been shown to produce effective anticolorectal carcinoma effects through inhibiting epithelial-mesenchymal transition(EMT).However,whether HYD-PEP06 has similar suppressive effect on hepatocellular carcinoma(HCC) remained unknown.In this study,HYD-PEP06 inhibited metastasis and EMT but not proliferation in vitro.Cignal finder pathway reporter array and Western blot analysis revealed that HYD-PEP06 suppressed HCCLM3 cell metastasis and EMT by inhibiting the PI3 K/AKT pathway.Moreover,HYD-PEP06 exerted antimetastasis effects in HepG2 cancer stem-like cells(CSCs) via suppressing the WNT/β-catenin signaling pathway.Finally,in HCCLM3 tumor-bearing BALB/c nu/nu nude mice,HYD-PEP06 substantially suppressed tumor growth,lung metastasis and HCC progress.Our results suggest that HYD-PEP06 inhibits the metastasis and EMT of HCC and CSCs as well,and thus has the potential as an agent for HCC treatment. | Wei Tian Jiatong Li Zhuo Wang Tong Zhang Ying Han Yanyan Liu Wenfeng Chu Yu Liu Baofeng Yang | 2021 | Acta Pharmaceutica Sinica B2021，11，6： | 5 |
| 7 | An extract from the earthworm Eisenia fetida non-specifically inhibits the activity of influenza and adenoviruses显示文摘OBJECTIVE:To test the in vitro antiviral activity of a crudetissueextract(CTE)fromtheearthwormEisenia fetida,determine any effective components in the CTE,andelucidatepossiblemechanismsofaction.METHODS:A CTE was made by homogenizing earthworms,followed by treatment with ammonium sulfate,then thermal denaturation.Inhibition of virus-induced cytopathic effect(CPE) was used to assess antiviral activity.Chromatographic analysis was used to identify effective components in the CTE.RESULTS:The CTE inhibited viral CPE at non-cytotoxic concentrations.Chromatography indicated that antiviral components corresponded to three active peaks indicative of proteases,nucleases and lysozymes.For adenoviruses,reduction in viral activity occurred for 100 μg/mL CTE.The reduction in adenoviral activity for four fractions was 100%,91.8%,86.9%,and 94.7%.For influenza viruses,reduction in viral activity of 100%,86.6%,69.1% and 88.3% was observed for 37 μg/mL CTE.In addition,three active fractions mixture had stronger antiviral activity(98.7% and 96.7%) than three fractions alone.Gel electrophoresis results indicated that nucleases from E.fetida could degrade the genome of influenza viruses and adenoviruses.CONCLUSION:The earthworm CTE displayed non-specific antiviral properties,possibly mediated by a combination of proteases,nucleases and lysozymes.Nucleases likely participate in the antiviral process,and degrade the genome of the virus thereby preventing further replication. | Zhizhen Liu Jianhua Wang Jianlin Zhang Baofeng Yu Bo Niu | 2012 | Journal of Traditional Chinese Medicine2012，32，4： | 4 |
| 8 | Genomic profiling and the impact of MUC19 mutation in hepatoid adenocarcinoma of the stomach显示文摘Dear Editor,Hepatoid adenocarcinoma(HAC)is a rare pathological subtype of extrahepatic tumor,featured by hepatoid differentiation andα-fetoprotein(AFP)-production[1,2].Hepatoid adenocarcinoma of the stomach(HAS),accounting for 0.3%to 1.0%of all gastric cancers(GCs),has attracted increasing attention due to its high degree of malignancy[3].Compared with classic GC,HAS showed a higher rate of vascular invasion,lymph node metastasis,and liver metastasis,with only 9.0%survival rate at 5 years[4].Currently,there is no effective treatment for HAS,and little is known about its pathogenesis.Herein,we investigated the molecular features of HAS and identified potential therapeutic targets for HAS. | Mengxuan Zhu Erbao Chen Shan Yu Chen Xu Yiyi Yu Xin Cao Wei Li Pengfei Zhang Yan Wang Baofeng Lian Shuirong Zhang Yueting Qu Lujia Huang Weiwei Shi Yuehong Cui Li Qian Tianshu Liu | 2022 | Cancer Communications2022，42，10： | 3 |
| 9 | The characteristics of shear wave splitting in the source region of the April 20,2013 Lushan earthquake显示文摘Using seismic data of the aftershocks sequence of the April 20,2013 Lushan earthquake recorded by seismic temporary and permanent stations in the source region,with the visual inspection of particle motion diagrams,this paper preliminarily contains the polarization directions of fast shear wave and the time-delays of split shear waves at every station,and analyzes the crustal anisotropic characteristics in the source region.In the study area,the polarization directions at stations BAX,TQU,L132,L133,L134,and L135 are northeast,which is consistent with the strike of Dachuan–Shuangshi fault.There are two polarization directions at MDS and L131,which are northeast and southeast.The scatter of polarization directions suggests the complex stress field around these two stations where two faults intersect.For the normalized time-delays at every station,the range is1.02–8.64 ms/km.The largest time-delay is from L134which is closest to the mainshock,and the smallest one is from L133.The variations in time-delays show the decreasing at stations BAX,L134,and L135 because of the stress–relaxation after earthquake. | Sha Liu Jiansi Yang Baofeng Tian Yu Zheng Xudong Jiang Zhiqiang Xu | 2013 | Earthquake Science2013，26，3： | 2 |
| 10 | Circulating microRNA-1 as a potential novel biomarker for acute myocardial infarction显示文摘 | Jing Ai Rong Zhang Yue Li Jielin Pu Yanjie Lu Jundong Jiao Kang Li Bo Yu Zhuqin Li Rongrong Wang Lihong Wang Qiang Li Ning Wang Hongli Shan Zhongyu Li Baofeng Yang | 2009 | Biochemical and Biophysical Research Communications2009，，： | 2 |
| 11 | Transsphenoidal microsurgical treatment of infradiaphragmatic craniopharyngioma显示文摘Objective Typically,the transcranial approach has been used for the treatment of craniopharyngiomas with suprasellar extension,whereas the transsphenoidal approach has been used mostly for infradiaphragmatic craniopharyngioma.Total resection of craniopharyngioma can reduce the recurrence rate,especially in young children,but it may lead to severe complications.Therefore,any benefit of the degree of resection must be weighed against the risk of complications by the surgeons.The purpose of this study was to explore the therapeutic outcome after transsphenoidal microsurgical treatment of infradiaphragmatic craniopharyngioma and share our experiences.Methods Between January 2003 and June 2013,30 patients with infradiaphragmatic craniopharyngioma underwent transsphenoidal microsurgical resection in our hospital.The neurological,visual,and endocrine functions,and extent of resection were analyzed retrospectively.Recurrence or growth of residual tumor tissue during follow-up was assessed using magnetic resonance imaging(MRI).Results Total resection was achieved in 25 patients(83.3%),subtotal resection was achieved in 4 patients(13.3%),and partial resection was achieved in 1 patient(3.4%).There were no perioperative deaths.Cerebrospinal fluid(CSF) leakage occurred in 6 patients,and among them,2 required surgical repair of the sella.New-onset postoperative diabetes insipidus(DI) developed in 8 patients.Vision and visual fields were improved at different levels in 13 out of 16 patients who had sight impediments before treatment.Tumor recurrence and regrowth was observed in 2 patients;1 patient underwent transsphenoidal reoperation,the condition of the other patient who had undergone several craniotomies grew worse over the 6-month follow-up period.Conclusion Transsphenoidal surgery is an ideal choice in treating infradiaphragmatic craniopharyngioma.The transsphenoidal approach,which preserves pituitary function and avoids damage to the hypothalamic structures and optic nerve,is associated with fewer complications than the transcranial approach and a low mortality rate. | Ting Lei Baofeng Wang Juan Chen Yu Xu Kai Shu Wei Sun Shaozheng Liu Xiaopeng Li | 2016 | Oncology and Translational Medicine2016，2，5： | 2 |
| 12 | Exosomes secreted from cardiomyocytes suppress the sensitivity of tumor ferroptosis in ischemic heart failure显示文摘Heart failure(HF)patients in general have a higher risk of developing cancer.Several animal studies have indicated that cardiac remodeling and HF remarkably accelerate tumor progression,highlighting a cause-and-effect relationship between these two disease entities.Targeting ferroptosis,a prevailing form of non-apoptotic cell death,has been considered a promising therapeutic strategy for human cancers.Exosomes critically contribute to proximal and distant organ-organ communications and play crucial roles in regulating diseases in a paracrine manner.However,whether exosomes control the sensitivity of cancer to ferroptosis via regulating the cardiomyocyte-tumor cell crosstalk in ischemic HF has not yet been explored.Here,we demonstrate that myocardial infarction(MI)decreased the sensitivity of cancer cells to the canonical ferroptosis activator erastin or imidazole ketone erastin in a mouse model of xenograft tumor.Post-MI plasma exosomes potently blunted the sensitivity of tumor cells to ferroptosis inducers both in vitro in mouse Lewis lung carcinoma cell line LLC and osteosarcoma cell line K7M2 and in vivo with xenograft tumorigenesis model.The expression of miR-22-3p in cardiomyocytes and plasma-exosomes was significantly upregulated in the failing hearts of mice with chronic MI and of HF patients as well.Incubation of tumor cells with the exosomes isolated from post-MI mouse plasma or overexpression of miR-22-3p alone abrogated erastin-induced ferroptotic cell death in vitro.Cardiomyocyte-enriched miR-22-3p was packaged in exosomes and transferred into tumor cells.Inhibition of cardiomyocyte-specific miR-22-3p by AAV9 sponge increased the sensitivity of cancer cells to ferroptosis.ACSL4,a pro-ferroptotic gene,was experimentally established as a target of miR-22-3p in tumor cells.Taken together,our findings uncovered for the first time that MI suppresses erastin-induced ferroptosis through releasing miR-22-3p-enriched exosomes derived from cardiomyocytes.Therefore,targeting exosome-mediated cardiomyocyte/tumor pathological communication may offer a novel approach for the ferroptosis-based antitumor therapy. | Ye Yuan Zhongting Mei Zhezhe Qu Guanghui Li Shuting Yu Yingqi Liu Kuiwu Liu Zhihua Shen Jiaying Pu Yanquan Wang Changhao Wang Zhiyong Sun Qian Liu Xiaochen Pang Ao Wang Zijing Ren Tong Wang Ying Liu Jinhuan Hong Jiajie Xie Xin Li Zhonghua Wang Weijie Du Baofeng Yang | 2023 | Signal Transduction and Targeted Therapy2023，8，4： | 2 |
| 13 | Dasatinib inhibits proliferation of liver cancer cells,but activation of Akt/mTOR compromises dasatinib as a cancer drug显示文摘Dasatinib is a multi-target protein tyrosine kinase inhibitor.Due to its potent inhibition of Src,Abl,the platelet-derived growth factor receptor(PDGFR)family kinases,and other oncogenic kinases,it has been investigated as a targeted therapy for a broad spectrum of cancer types.However,its efficacy has not been significantly extended beyond leukemia.The mechanism of resistance to dasatinib in a wide array of cancers is not clear.In the present study,we investigated the effect of dasatinib on hepatocellular carcinoma cell growth and explored the underlying mechanisms.Our results showed that dasatinib potently inhibited the proliferation of SNU-449 cells,but not that of other cell lines,such as SK-Hep-1,even though it inhibited the phosphorylation of Src on both negative and positive regulation sites in all these cells.Dasatinib activated the phosphoinositide-dependent protein kinase1(PDK1)/protein kinase B(Akt)/mammalian target of rapamycin(mTOR)signaling pathway in SK-Hep-1 cells,but not in SNU-449 cells.Blocking the Akt/mTOR signaling pathway strongly promoted the efficacy of dasatinib in SK-Hep-1 cells.In SNU-449 cells,dasatinib promoted apoptosis and the cleavage of caspase-3 and caspase-7,induced cell cycle arrest in the G1 phase,and inhibited the expression of Cyclin-dependent kinase(CDK4)/6/CyclinD1 complex.These findings demonstrate that dasatinib exerts its anti-proliferative effect on hepatocellular cell proliferation by blocking the Src family kinases;however,it causes Akt activation,which compromises dasatinib as an anti-cancer drug. | Chang Liu Xiaoxia Zhu Yuqi Jia Fenqing Chi Keru Qin Jinhong Pei Chan Zhang Xiuli Mu Hongwei Zhang Xiushan Dong Jun Xu Baofeng Yu | 2021 | Acta Biochimica et Biophysica Sinica2021，53，7： | 2 |
| 14 | Scale-dependent effects of habitat fragmentation on the genetic diversity of Actinidia chinensis populations in China显示文摘Spatial scale partly explains the differentiated effects of habitat fragmentation on plant biodiversity,but the mechanisms remain unclear.To investigate the effects of habitat fragmentation on genetic diversity at different scales,we sampled Actinidia chinensis Planch.at broad and fine scales,China.The broad-scale sampling included five mountain populations and one oceanic island population(Zhoushan Archipelago),and the fine-scale sampling covered 11 lake islands and three neighboring land populations in Thousand-Island Lake(TIL).These populations were genotyped at 30 microsatellite loci,and genetic diversity,gene flow,and genetic differentiation were evaluated.Genetic differentiation was positively related to geographical distance at the broad scale,indicating an isolation-bydistance effect of habitat fragmentation on genetic diversity.The oceanic population differed from the mainland populations and experienced recent bottleneck events,but it showed high gene flow with low genetic differentiation from a mountain population connected by the Yangtze River.At the fine scale,no negative genetic effects of habitat fragmentation were found because seed dispersal with water facilitates gene flow between islands.The population size of A.chinensis was positively correlated with the area of TIL islands,supporting island biogeography theory,but no correlation was found between genetic diversity and island area.Our results highlight the scale-dependent effects of habitat fragmentation on genetic diversity and the importance of connectivity between island-like isolated habitats at both the broad and fine scales. | Wenhao Yu Baofeng Wu Xinyu Wang Zhi Yao Yonghua Li Yongbo Liu | 2020 | Horticulture Research2020，7，1： | 1 |
| 15 | A novel reciprocal loop between microRNA-21 and TGFβRIII is involved in cardiac fibrosis显示文摘 | Haihai Liang Chun Zhang Tao Ban Yu Liu Liu Mei Xianmei Piao Dan Zhao Yanjie Lu Wenfeng Chu Baofeng Yang | 2012 | International Journal of Biochemistry and Cell Biology2012，，12： | 1 |
| 16 | Energy-efficiency resource allocation of very large multi-user MIMO systems显示文摘 | Ying Hu Baofeng Ji Yongming Huang Fei Yu Luxi Yang | 2014 | Wireless Networks2014，，： | 1 |
| 17 | Relational governance and opportunism in logistics outsourcing relationships: empirical evidence from China显示文摘 | Fujun Lai Yu Tian Baofeng Huo | 2012 | International Journal of Production Research2012，，9： | 1 |
| 18 | The functions of CD4 T-helper lymphocytes in chronic obstructive pulmonary disease显示文摘Chronic obstructive pulmonary disease(COPD)has been increasingly accounted for global morbidity and mortality worldwide.Although it is partially reversible,the obstructive ventilatory schema of COPD often causes chronic inflammation that primarily affects peripheral airways,pulmonary parenchyma,and the development of lung lymphoid follicles.Among various T-helper(Th)cell types associated with COPD,Th1,Th2 and Th17 cell numbers are increased in COPD patients,whereas Treg cell number is reduced.Here,we reviewed recent advance in understanding the roles of Th1/Th2 and Th17/Treg in the pathogenesis of COPD and discussed the potential underlying mechanism. | Keru Qin Baixue Xu Min Pang Hailong Wang Baofeng Yu | 2022 | Acta Biochimica et Biophysica Sinica2022，54，2： | 1 |
| 19 | Mechanical and morphological properties of highly dispersed carbon nanotubes reinforced cement based materials显示文摘 | Baomin Wang Yu Han Baofeng Pan Tingting Zhang | 2013 | Journal of Wuhan University of Technology-Mater Sci Ed2013，，1： | 1 |
| 20 | MoS_(2)/MoO_(2) nanosheets anchored on carbon cloth for high-performance magnesium-and sodium-ion storage显示文摘Developing new types of rechargeable metal-ion batteries beyond lithium-ions,including alkaline ion(such as Na+,K+)and multivalent ion(such as Mg 2+,Zn 2+,Ca 2+and Al 3+)batteries,is progressing quickly towards large-scale energy storage systems.However,the major obstacle to their large-scale applications has been a lack of appropriate electrode materials with reversible metal ions insertion/extraction be-havior,resulting in inferior electrochemical performance.Here we develop a well-designed MoS_(2)/MoO_(2) hybrid nanosheets anchored on carbon cloth(MoS_(2)/MoO_(2)/CC)as electrode materials.This rational de-sign can effectively shorten ion diffusion distance,increase electric conductivity of the electrode,and buffer volume change.Benefiting from the synergistic effect of structural and compositional features,the MoS_(2)/MoO_(2)/CC electrode exhibits high initial reversible capacities(326 mA h g^(−1) at 0.1 A g^(−1) in magnesium-ion storage;1270 mA h g^(−1) at 0.1 A g^(−1) in sodium-ion storage),excellent rate capacities(57 mA h g^(−1) at 10 A g^(−1) in magnesium-ion storage;335 mA h g^(−1) at 5 A g^(−1) in sodium-ion storage)and long-term cycling stability(105 mA h g^(−1) after 600 cycle at 1 A g^(−1) in magnesium-ion storage;208 mA h g^(−1) after 600 cycles at 5 A g^(−1) in sodium-ion storage).We expect that the multi-engineering strategy will provide some valuable insights for the development of other advanced electrode materials for high-performance metal-ion batteries. | Hui Wu Zhennan Xiong Yiyang Mao Huihui Zhang Yingying Hu Jun Shen Baofeng Wang Xuebin Yu | 2023 | Journal of Materials Science & Technology2023，，12： | 0 |
您所在的机构暂未开通此部分的功能，请联系管理员