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| 1 | 卒中或短暂性脑缺血发作患者的卒中预防指南 美国心脏协会/美国卒中协会为医疗卫生专业人员制定的指南显示文摘本指南旨在为缺血性卒中或短暂性脑缺血发作存活患者的卒中预防提供全面和及时的循证推荐,包括危险因素的控制、动脉粥样硬化性疾病的于预、心源性栓塞的抗栓治疗以及非心源性栓塞性卒中的抗血小板治疗。另外,还对其他许多特殊情况下的复发性卒中预防提供了推荐意见,包括动脉夹层分离、卯圆孔未闭、高同型半胱氨酸血症、高凝状态、镰状细胞病、脑静脉窦血栓形成、女性卒中(尤其是与妊娠和绝经后雌激素替代治疗相关性卒中)、脑出血后抗凝药的使用等,以及实施本指南及其在高危人群中应用的特定方法。 | Karen L. Furie Scott E. Kasner Robert J. Adams Gregory W. Albers Ruth L. Bush Susan C. Fagan Jonathan L. Halperin S. Claiborne Johnston Irene Katzan Walter N. Kernan Pamela H. Mitchell Bruce Ovbiagele Yuko Y. Palesch Ralph L. Sacco Lee H. Schwamm Sylvia Wassertheil-Smoller Tanya N. Turan Deidre Wentworth 李海峰(译) 刘涛(译) 杨潘(译) 王鹏(译) | 2011 | 国际脑血管病杂志2011，19，1： | 391 |
| 2 | 从113例不同疾病患者的活动障碍分析看作业治疗的必要性显示文摘损伤或患病后是否需要接受作业治疗在我国是一个认识模糊的问题。本文就此对ll3例不同疾病的患者采用加拿大作业活动测量表进行了采访调查。统计学分析表明,所有患者均有继发于损伤或疾病而亟待解决的日常作业活动方面的问题,他们都需要在不同的侧面接受作业治疗;COPM是目前以患者为中心的作业治疗模式的一个很有用的测量工具,值得在临床工作中推荐。 | 恽晓平 Tanya L.Packer 欧阳多利 黄丽萍 陈琪 | 1995 | 中国康复理论与实践1995，1，1： | 12 |
| 3 | Stool DNA methylation assays in colorectal cancer screening显示文摘Colorectal cancer(CRC) is fourth most common cancer in men and third in women worldwide. Developing a diagnostic panel of sensitive and specific biomarkers for the early detection of CRC is recognised as to be crucial for early initial diagnosis, which in turn leads to better long term survival. Most of the research on novel potential CRC biomarkers in the last 2 decades has been focussed on stool DNA analysis. In this paper, we describe the recent advances in non-invasive CRC screening and more specifically in molecular assays for aberrantly methylated BMP3 and NDRG4 promoter regions. In several research papers these markers showed superior rates for sensitivity and specificity in comparison to previously described assays. These tests detected the majority of adenomas ≥ 1 cm in size and the detection rates progressively increased with larger adenomas. The methylation status of the BMP3 and NDRG4 promoters demonstrated effective detection of neoplasms at all sites throughout the colon and was not affected by common clinical variables. Recently, a multitarget stool DNA test consisting of molecular assays for aberrantly methylated BMP3 and NDRG4 promoter regions, mutant KRAS and immunochemical assay for human haemoglobin has been made commercially available and is currently reimbursed in the United States. Although this is the most sensitive noninvasive CRC screening test, there is the need for further research in several areas- establishment of the best timeframe for repeated DNA stool testing; validation of the results in populations outside of North America; usefulness for surveillance and prognosis of patients; cost-effectiveness of DNA stool testing in real-life populations. | Tanya Kadiyska Alexander Nossikoff | 2015 | World Journal of Gastroenterology2015，21，35： | 9 |
| 4 | HOS3, an ELO-Like Gene, Inhibits Effects of ABA and Implicates a S-1-P/Ceramide Control System for Abiotic Stress Responses in Arabidopsis thaliana显示文摘Arabidopsis thaliana 的 hyper-osmotically 敏感的异种,指明的 hos3-1 (渗透地应答的基因的高表示) ,基于它 RD29A 的超被识别: 在有 NaCl 和骆驼毛的织物的处理之上的 LUC 倡导者熔化植物。这些回答含有破坏基因作为 RD29A 压力应答的小径的一个直接或间接的否定管理者。由定序 T-DNA 边阶的 flanking 区域,破坏基因在地点 At4g36830,这被决定,作为与高相同编码通常认为的蛋白质到 CIG30 (ELO2/FEN1 ) 注解了。CIG30 在合成被含有很长锁住丰满的酸(VLCFA ) ,它是为 sphingolipids 和 ceramides 的必要先锋。改变了骆驼毛的织物超敏性特征的压力回答,包括减少的根生长抑制并且与骆驼毛的织物处理减少了萌芽并且从叶子减少了水损失,被突变而产生之遗传的 hos3-1 和 hos3-2 异种展出。hos3-2 异种部分在它的抄本丰富被压制并且作为一个后退的特点被继承。进一步,在 35SCaMV 倡导者的控制下面的 HOS3 ORF 象 RD29A 一样恢复了野类型的 NaCl 根和骆驼毛的织物根生长敏感: 在变异的植物的 LUC 光。我们也这里证明 HOS3 野类型的基因机能上地补充 elo2 和 elo3 酵母异种的敏感到 monensin。而且, hos3-1 和 hos3-2 等位基因分享了增加的敏感到除草剂 Metolachlor,它在 VLCFA 的合成禁止酰链延伸,并且 HOS3 机能上地补充了 elo2 和 elo3 并且恢复了 VLCFA 的层次。一起,这些数据证实 HOS3 禁止调停骆驼毛的织物的压力回答并且含有控制为不能生活的应力发信号和回答的几个方面指的 VLCFA 小径和产品。结果也在有气孔的行为的控制为 ceramide 的一个角色提供支持。 | Tanya M. Quist Irina Sokolchik Huazhong Shi Robert J. Joly Ray A. Bressan Albino Maggio Meena Narsimhan Xia Li | 2009 | Molecular Plant2009，2，1： | 7 |
| 5 | 21世纪的卒中新定义:美国心脏协会/美国卒中协会对医疗专业人员的声明显示文摘尽管脑血管病的影响广泛且对其病理生理学的理解已取得一些进展,但“卒中”一词在临床实践、临床研究和公共卫生评价中的定义并不统一。经典的卒中定义主要是临床定义,不能反映科学技术的进步。因此,美国心脏协会卒中委员会/美国卒中协会召集了一个写作组,旨在对21世纪的卒中新定义制定一份专家共识文件。中枢神经系统(centralnervoussystem,CNS)梗死被定义为缺血导致的脑、脊髓或视网膜细胞死亡,并且具有永久性损伤的神经病理学、神经影像学和(或)临床证据。CNS梗死具有一系列临床表现:缺血性卒中特指伴有明显症状的CNS梗死,而无症状梗死则顾名思义不引起明显症状。广义的卒中还包括脑出血和蛛网膜下腔出血。卒中的新定义结合了临床和组织学标准,因此能被融入临床实践、研究和公共卫生评价。 | Ralph L. Sacco Scott E. Kasner Joseph P. Broderick Louis R. Caplan J.J. (Buddy) Connors Antonio Culebras Mitchell S.V. Elkind Mary G. George Allen D. Hamdan Randall T. Higashida Brian L. Hoh L. Scott Janis Carlos S. Kase Dawn O. Kleindorfer Jin-Moo Lee Michael E. Moseley Eric D. Peterson Tanya N. Turan Amy L. Valderrama Harry V. Vinters 王胜男(译) 吴齐恒(译) 邓镇(译) 周晓(译) 袁超(译) 潘速跃(译) | 2014 | 国际脑血管病杂志2014，22，1： | 7 |
| 6 | Curcumin reverses T cell-mediated adaptive immune dysfunctions in tumor-bearing hosts显示文摘Immune dysfunction is well documented during tumor progression and likely contributes to tumor immune evasion.CD81 cytotoxic T lymphocytes(CTLs)are involved in antigen-specific tumor destruction and CD41 T cells are essential for helping this CD81 T cell-dependent tumor eradication.Tumors often target and inhibit T-cell function to escape from immune surveillance.This dysfunction includes loss of effector and memory T cells,bias towards type 2 cytokines and expansion of T regulatory(Treg)cells.Curcumin has previously been shown to have antitumor activity and some research has addressed the immunoprotective potential of this plant-derived polyphenol in tumor-bearing hosts.Here we examined the role of curcumin in the prevention of tumor-induced dysfunction of T cell-based immune responses.We observed severe loss of both effector and memory T-cell populations,downregulation of type 1 and upregulation of type 2 immune responses and decreased proliferation of effector T cells in the presence of tumors.Curcumin,in turn,prevented this loss of T cells,expanded central memory T cell(TCM)/effector memory T cell(TEM)populations,reversed the type 2 immune bias and attenuated the tumor-induced inhibition of T-cell proliferation in tumor-bearing hosts.Further investigation revealed that tumor burden upregulated Treg cell populations and stimulated the production of the immunosuppressive cytokines transforming growth factor(TGF)-b and IL-10 in these cells.Curcumin,however,inhibited the suppressive activity of Treg cells by downregulating the production of TGF-b and IL-10 in these cells.More importantly,curcumin treatment enhanced the ability of effector T cells to kill cancer cells.Overall,our observations suggest that the unique properties of curcumin may be exploited for successful attenuation of tumor-induced suppression of cell-mediated immune responses. | Sankar Bhattacharyya Dewan Md Sakib Hossain Suchismita Mohanty Gouri Sankar Sen Sreya Chattopadhyay Shuvomoy Banerjee Juni Chakraborty Kaushik Das Diptendra Sarkar Tanya Das Gaurisankar Sa | 2010 | Cellular & Molecular Immunology2010，7，4： | 7 |
| 7 | 诺西那生治疗脊髓性肌萎缩显示文摘目的观察诺西那生(Nusinersen)治疗脊髓性肌萎缩(SMA)患者的疗效和不良反应。方法回顾性分析2017年9月至2018年3月以色列施耐德儿童医学中心神经内科就诊的12例SMA患儿的临床资料。所有患儿均经基因检测证实为运动神经元存活基因(SMN)1纯合子7号外显子缺失,无SMN1杂合子突变。应用诺西那生12mg腰椎穿刺鞘内注射治疗,前4次鞘内给药为负荷剂量12mg,分别于1、15、30、60d给药,以后每4个月给予1次维持剂量12mg。于181d运用费城儿童医院神经肌肉障碍婴儿测试量表(CHOP-INTEND)和Hammersmith运动功能评分量表(HFMSE)量表评价患儿运动功能改善情况。结果治疗181d评价,早发型(SMA-Ⅰ)4例中2例CHOP-INTEND评分提高3分和5分,另2例肌张力有明显改善;晚发型(SMA-Ⅱ/SMA-Ⅲ/SMA-Ⅳ)8例HFMSE评分均有不同程度改善,最低提高3.1分,最高提高5.2分,平均提高4.1分。不良反应少且轻微。结论诺西那生治疗SMA的疗效肯定,安全性较好。 | 陈峥 Yoram Nevo Tanya Moss | 2019 | 中国临床研究2019，32，6： | 7 |
| 8 | 单片系统在星载计算机上的应用显示文摘小卫星的设计目标之一是低成本、轻质量以及研制周期短 ,使用商用现货的元器件和开发工具可以更好的支持这一思想。可编程单片系统是其中的一项关键技术。这种单片系统方案可以降低小卫星电子系统 (比如数管 ,通信和控制系统 )的体积、复杂度和成本。该文阐述了单片系统在航天器上的应用 ,并以一星载计算机的单片系统作为设计举证 ,包括一个微处理器软核、其他的外围接口设备软核及所采用的目标芯片 ,并对以微处理器软核为核心的片上系统如何实现进行描述 。 | 郑岱珣 Tanya Vladimirova | 2004 | 中国空间科学技术2004，24，1： | 6 |
| 9 | Ammonia-oxidizing archaea and bacteria are structured by geography in biological soil crusts across North American arid lands显示文摘Introduction:Biological soil crusts(BSCs)can dominate surface cover in dry lands worldwide,playing an integral role in arid land biogeochemistry,particularly in N fertilization through fixation and cycling.Nitrification is a characteristic and universal N transformation in BSCs that becomes important for the export of N beyond the microscopic bounds of the crust itself.The contribution of ammonia-oxidizing bacteria(AOB)in BSCs has been shown,but the role and extent of the recently discovered ammonia-oxidizing archaea(AOA)have not.Methods:We sampled various types of crusts in four desert regions across the western United States and characterized the composition and size of ammonia-oxidizing communities using clone libraries and quantitative PCR targeting the amoA gene,which codes for the ammonia monooxygenase enzyme,universally present in ammonia-oxidizing microbes.Results:All archaeal amoA sequences retrieved from BSCs belonged to the Thaumarchaeota(Nitrososphaera associated Group I.1b).Sequences from the Sonoran Desert,Colorado Plateau,and Great Basin were indistinguishable from each other but distinct from those of the Chihuahuan Desert.Based on amoA gene abundances,archaeal and bacterial ammonia oxidizers were ubiquitous in our survey,but the ratios of archaeal to bacterial ammonia oxidizers shifted from bacterially dominated in northern,cooler deserts to archaeally dominated in southern,warmer deserts.Conclusions:Archaea are shown to be potentially important biogeochemical agents of biological soil crust N cycling.Conditions associated with different types of BSCs and biogeographical factors reveal a niche differentiation between AOA and AOB,possibly driven by temperature. | Yevgeniy Marusenko Scott T Bates Ian Anderson Shannon L Johnson Tanya Soule Ferran Garcia-Pichel | 2013 | Ecological Processes2013，2，1： | 4 |
| 10 | A Dominant Allele of Arabidopsis Pectin-Binding Wall-Associated Kinase Induces a Stress Response Suppressed by MPK6 but Not MPK3 Mutations显示文摘植物房间墙由纤维素纤维,灵活果胶聚合物,和一连串的分类糖类和蛋白质的一个矩阵组成。Arabidopsis 的像受体的联系墙的 Kinases (WAK ) 在墙中绑果胶,并且在开发期间并且为对病原体的回答为房间扩大两个都是必要的并且伤害。Mitogen 激活蛋白质 Kinases (MPK ) 在优核质形成在房间表面受体和 transcriptional 和酶规定之间的一个主要发信号连接,并且 Arabidopsis MPK6 和 MPK3 确实在到应力和病原体的开发和反应有重要角色。WAK2 的主导的等位基因要求 kinase 活动并且激活包括增加的 ROS 累积和涉及病原体抵抗的众多的基因的起来规定的压力回答,伤害,并且房间墙生物的续生说。这主导的等位基因要求功能的果胶绑定和 kinase 领域,显示它从事表明小径的 WAK。主要血浆膜生产 ROS 酶建筑群的空异种, rbohd/f 不压制导致 WAK2cTAP 的显型。mpk6,然而并非 mpk3,空等位基因能压制这个主导的 WAK2 变化,因此区分的 MPK3 和 MPK6 的效果,其活动以前被认为冗余。基因表示的果胶激活被消除在一 wak2 空,但是由导致的 WAK 主导的等位基因的 tempered 被提高基础压力相关的抄本层次。结果建议细胞墙的变化能在细胞的回答和帮助在导致一个大变化解释病原体和伤害的罐头怎么在生长上有一般效果的机制。 | Bruce D. Kohorn Susan L. Kohorn Tanya Todorova Gillian Baptiste Kevin Stansky Meghan McCullough | 2012 | Molecular Plant2012，5，4： | 4 |
| 11 | The diagnosis, treatment, and follow-up of cesarean scar pregnancy显示文摘 | Ilan E. Timor-Tritsch Ana Monteagudo Rosalba Santos Tanya Tsymbal Grace Pineda Alan A. Arslan | 2012 | American Journal of Obstetrics and Gynecology2012，，1： | 3 |
| 12 | 口服抗栓药预防非瓣膜性心房颤动患者卒中:美国心脏协会/美国卒中协会对医疗卫生专业人员的科学建议显示文摘成年心房颤动(atrial fibrillation,AF)患者的卒中发生率存在很大差异,其范围为每年1%~20%(平均4.5%/年),取决于合并症和既往脑血管事件病史。卒中风险分层十分重要,因为用于降低AF相关性卒中发生率的抗栓药的主要风险是出血。对于华法林而言,这涉及到对出血风险(每年1%~12%)与缺血性事件风险的权衡,它通常被用于血栓栓塞风险最高的那些患者。 | Karen L. Furie LarryB. Goldstein Gregory W. Albers Pooja Khatri Ron Neyens Mintu P. Turakhia Tanya N. Turan Kathryn A. Wood 严晓铭(译) 何晟(译) 丁妹(译) 柯开富(译) | 2012 | 国际脑血管病杂志2012，20，12： | 3 |
| 13 | High Prevalence of Sessile Serrated Adenomas With BRAF Mutations: A Prospective Study of Patients Undergoing Colonoscopy显示文摘 | Kevin J. Spring Zhen Zhen Zhao Rozemary Karamatic Michael D. Walsh Vicki L.J. Whitehall Tanya Pike Lisa A. Simms Joanne Young Michael James Grant W. Montgomery Mark Appleyard David Hewett Kazutomo Togashi Jeremy R. Jass Barbara A. Leggett | 2006 | Gastroenterology2006，，5： | 3 |
| 14 | Budd-Chiari syndrome:A single-center experience显示文摘AIM:To investigate challenges,risk factors,prognostic indicators,and treatment outcomes associated with Budd-Chiari syndrome(BCS)at a tertiary care center.METHODS:A retrospective cohort study was conducted at the University of Pennsylvania in patients with a diagnosis of BCS or hepatic vein thrombosis.All patients receiving care at the University of Pennsylvania,and who had at least 2 clinical encounters in the University of Pennsylvania Health system from January1,2008 to September 10,2013 were eligible for study inclusion.Data were extracted from the electronic medical record of each patient,and recorded in a secure Research Electronic Data Capture database.Logistic regression analyses were applied to identify predictors of outcome of liver transplant(LT)or death.RESULTS:Between January 1,2008 and September10,2013,forty-seven patients were identified.Median age was 42.4 years.Thirty-one(66.0%)were women.A majority were Caucasian(68.1%).At diagnosis,43(91.5%)patients had ascites,27(57.4%)patients had a hematologic disorder associated with a hypercoagulable state and 26(55.3%)had cirrhosis.Forty(85.1%)patients were on anticoagulation(AC),30(63.8%)of whom were maintained on warfarin.Two patients(4.3%)underwent thrombolytic therapy.A transjugular intrahepatic portosystemic shunt(TIPS)was placed in21(44.7%)patients,19(90.5%)of whom were also on AC.Twenty-one(44.7%)received AC alone.Over a median of 974 d,8(17.0%)patients received LT,and10(21.3%)died.The median time from listing to death was 26 mo[interquartile range(IQR)=16,65)].TIPS with AC was utilized more frequently in younger patients(P=0.02).Age,cirrhosis and chronic kidney disease(CKD)were significant predictors of LT or death.CONCLUSION:AC alone was employed as frequently as TIPS with AC,though the latter was used more frequently in younger patients with polycythemia vera.There were no significant differences in treatment outcome regardless of the therapeutic intervention employed.Significant predictors of poor prognosis included age,cirrhosis and CKD. | Tanya M Pavri Alan Herbst Rajender Reddy Kimberly A Forde | 2014 | World Journal of Gastroenterology2014，20，43： | 2 |
| 15 | Permafrost-associated natural gas hydrate occurrences on the Alaska North Slope显示文摘 | Timothy S. Collett Myung W. Lee Warren F. Agena John J. Miller Kristen A. Lewis Margarita V. Zyrianova Ray Boswell Tanya L. Inks | 2009 | Marine and Petroleum Geology2009，，2： | 2 |
| 16 | White matter hyperintensity progression is associated with incident probable dementia or mild cognitive impairment显示文摘Background White matter hyperintensity(WMH)on brain MRI is associated with developing dementia or mild cognitive impairment(MCI),but WMH progression over time has not been fully investigated as an independent risk factor.Methods We performed a post hoc analysis of the Systolic Blood Pressure Intervention Trial-Memory and Cognition in Decreased Hypertension(SPRINT MIND)trial.The primary outcome was incident probable dementia or MCI(dementia/MCI)before the follow-up MRI at 48 months from enrolment.The primary predictor was WMH progression,defined as the Z score difference between the follow-up and baseline WMH volumes.The secondary predictor was a binary WMH progression threshold(≥1.4 mL vs<1.4 mL).Results Among the 433 included patients,33(7.6%)developed dementia/MCI.There were 156(36.0%)patients who met the WMH progression threshold of≥1.4 mL,in whom the rate of dementia/MCI was 12.8%(20/156)vs 4.7%(13/277)of patients with<1.4 mL WMH progression(p=0.002).In multivariable logistic regression,the Z score of WMH progression was associated with dementia/MCI(OR 1.51,95%CI 1.12 to 2.04,p=0.007)as was the WMH progression threshold of≥1.4 mL(OR 2.89,95%CI 1.23 to 6.81,p=0.015).Conclusions In this post hoc analysis of SPRINT MIND,WMH progression over 48 months was associated with the development of probable dementia or MCI. | Adam de Havenon Kevin N Sheth Sharon D Yeatts Tanya N Turan Shyam Prabhakaran | 2022 | Stroke & Vascular Neurology2022，7，4： | 2 |
| 17 | Emissive displays with transfer-printed assemblies of 8 μm × 15 μm inorganic light-emitting diodes显示文摘Displays using direct light emission from microscale inorganic light-emitting diodes(μILEDs)have the potential to be very bright and also very power efficient.High-throughput technologies that accurately and cost-effectively assemble microscale devices on display substrates with high yield are key enablers forμILED displays.Elastomer stamp transfer printing is such a candidate assembly technology.A variety ofμILED displays have been designed and fabricated by transfer printing,including passive-matrix and active-matrix displays on glass and plastic substrates. | CHRISTOPHER A.BOWER MATTHEW A.MEITL BROOK RAYMOND ERICH RADAUSCHER RONALD COK SALVATORE BONAFEDE DAVID GOMEZ TANYA MOORE CARL PREVATTE BRENT FISHER ROBERT ROTZOLL GEORGE A.MELNIK ALIN FECIORU ANTóNIO JOSé TRINDADE | 2017 | Photonics Research2017，5，2： | 2 |
| 18 | A case-control study of maternal recreational physical activity and risk of gestational diabetes mellitus显示文摘 | Jennifer C. Dempsey Carole L. Butler Tanya K. Sorensen I-Min Lee Mary Lou Thompson Raymond S. Miller Ihunnaya O. Frederick Michelle A. Williams | 2004 | Diabetes Research and Clinical Practice2004，，2： | 2 |
| 19 | Role of TNFSF15 in the intestinal inflammatory response显示文摘Gastrointestinal diseases, specifically Crohn's disease, ulcerative colitis, diverticular disease, and primary biliary cirrhosis are all characterized by complicated inflammation of the digestive tract. Their pathology is multifactorial, and risk factors encompass both genetic and environmental factors. Recent advances in the genetic component of inflammatory bowel diseases(IBDs) have revealed that the tumor necrosis factor superfamily member 15(TNFSF15) contains a number of risk alleles associated not only with IBD but also with other diseases such as diverticular disease and primary biliary cirrhosis. These risk alleles in TNFSF15 and the altered expression of its gene product can serve as the common ground between these disorders by explaining at least some of the underlying processes that lead to a dysregulated immune response and subsequent chronic inflammation. Here, we aim to outline how the TNFSF15 gene is involved in the proliferation and cell fate of different populations of T cells and subsequently in the control of both pro-and anti-inflammatory cytokines. Furthermore, we summarize what is currently known of TNFSF15 control region variants, how they are associated with each mentioned disease, and how these variants can explain the autoimmune pathology of said diseases through altered TNFSF15 expression. | Tanya Kadiyska Ivan Tourtourikov Ana-Maria Popmihaylova Hilda Kadian Ani Chavoushian | 2018 | World Journal of Gastrointestinal Pathophysiology2018，9，4： | 2 |
| 20 | Virus-Induced Gene Silencing in the Culinary Ginger (Zingiber officinale): An Effective Mechanism for Down-Regulating Gene Expression in Tropical Monocots显示文摘导致病毒的基因 silencing (VIGS ) 被显示了有效为短暂在植物到的基因表示击倒在开发和压力相关的回答分析特定的基因的效果。VIGS 很好在 Solanaceae, Brassicaceae, Leguminaceae,和 Poaceae 以内为模型系统和庄稼的研究被建立,但是仅仅最近被用于住在这些家庭外面的植物。这里,我们证明了那个大麦条纹马赛克病毒(BSMV ) 能在 Zingiberaceae 以内感染二种,并且那 BSMVVIGS 能被用于明确地在厨房的生姜姜 officinale 的下面调整 phytoene desaturase。这些结果建议到除谷物以外的 monocots 的 BSMVVIGS 的扩展为许多重要适度、热带的庄稼种类的指导基因分析有潜力。 | Tanya Renner Jennifer Bragg Heather E. Driscoll Juliana Cho Andrew O. Jackson Chelsea D. Specht | 2009 | Molecular Plant2009，2，5： | 2 |
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