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| 1 | Diagnosis and Treatment of Keloids and Hypertrophic Scars—Japan Scar Workshop Consensus Document 2018显示文摘There has been a long-standing need for guidelines on the diagnosis and treatment of keloids and hypertrophic scars that are based on an understanding of the pathomechanisms that underlie these skin fibrotic diseases.This is particularly true for clinicians who deal with Asian and African patients because these ethnicities are highly prone to these diseases.By contrast,Caucasians are less likely to develop keloids and hypertrophic scars,and if they do,the scars tend not to be severe.This ethnic disparity also means that countries vary in terms of their differential diagnostic algorithms.The lack of clear treatment guidelines also means that primary care physicians are currently applying a hotchpotch of treatments,with uneven outcomes.To overcome these issues,the Japan Scar Workshop(JSW)has created a tool that allows clinicians to objectively diagnose and distinguish between keloids,hypertrophic scars,and mature scars.This tool is called the JSW Scar Scale(JSS)and it involves scoring the risk factors of the individual patients and the affected areas.The tool is simple and easy to use.As a result,even physicians who are not accustomed to keloids and hypertrophic scars can easily diagnose them and judge their severity.The JSW has also established a committee that,in cooperation with outside experts in various fields,has prepared a Consensus Document on keloid and hypertrophic scar treatment guidelines.These guidelines are simple and will allow even inexperienced clinicians to choose the most appropriate treatment strategy.The Consensus Document is provided in this article.It describes(1)the diagnostic algorithm for pathological scars and how to differentiate them from clinically similar benign and malignant tumors,(2)the general treatment algorithms for keloids and hypertrophic scars at different medical facilities,(3)the rationale behind each treatment for keloids and hypertrophic scars,and(4)the body site-specific treatment protocols for these scars.We believe that this Consensus Document will be helpful for physicians from all over the world who treat keloids and hypertrophic scars. | Rei Ogawa Sadanori Akita Satoshi Akaishi Noriko Aramaki-Hattori Teruyuki Dohi Toshihiko Hayashi Kazuo Kishi Taro Kono Hajime Matsumura Gan Muneuchi Naoki Murao Munetomo Nagao Keisuke Okabe Fumiaki Shimizu Mamiko Tosa Yasuyoshi Tosa Satoko Yamawaki Shinichi Ansai Norihisa Inazu Toshiko Kamo Reiko Kazki Shigehiko Kuribayashi | 2019 | Burns & Trauma2019，7，1： | 31 |
| 2 | Sentinel Node Mapping for Gastric Cancer: A Prospective Multicenter Trial in Japan显示文摘 | Yuko Kitagawa Hiroya Takeuchi Yu Takagi Shoji Natsugoe Masanori Terashima Nozomu Murakami Takashi Fujimura Hironori Tsujimoto Hideki Hayashi Nobunari Yoshimizu Akinori Takagane Yasuhiko Mohri Kazuhito Nabeshima Yoshikazu Uenosono Shinichi Kinami Junichi S | 2013 | Journal of Clinical Oncology2013，，29： | 11 |
| 3 | Adiponectin deficiency exacerbates lipopolysaccharide/ D-galactosamine-induced liver injury in mice显示文摘瞄准:在 Kupffer 房间的功能上检验 adiponectin 的效果,脂肪的多糖(LPS ) 的关键调节的人导致了肝损伤。方法:D-galactosamine (GalN ) 和 LPS intraperitoneally 被注入 adiponectin-/- 老鼠和野类型老鼠。Kupffer 房间,从 Sprague-Dawley 孤立老鼠,是 preincubated 与或没有 adiponectin,然后与 LPS 对待。结果:在猛烈老鼠, GalN/LPS 注射显著地降低了幸存率,显著地提起了丙氨酸转氨酶和肿瘤坏死 factor-alpha (TNF-alpha ) 的血浆层次并且显著地减少了与野类型老鼠相比的 IL-10 层次。在肝的 TNF-alpha 基因表示是哪个更高并且 IL-10 的那些比在野类型老鼠在猛烈老鼠是更低的。在有教养的 adiponectin-pre-treated Kupffer 房间, LPS 显著地降低了 TNF-alpha 层次并且在培养基和他们的各自的基因表示层次提起了 IL-10 层次,与没有 adiponectin-pre-treatment 的 Kupffer 房间相比。结论:Adiponectin supresses TNF-alpha 生产并且响应 LPS 刺激由 Kupffer 房间导致 IL-10 生产,并且 adiponectin 的缺乏提高导致 LPS 的肝损伤。 | Hitoshi Matsumoto Shinji Tamura Yoshihiro Kamada Shinichi Kiso Juichi Fukushima Akira Wada Norikazu Maeda Shinji Kihara Tohru Funahashi Yuji Matsuzawa Iichiro Shimomura Norio Hayashi | 2006 | World Journal of Gastroenterology2006，12，21： | 5 |
| 4 | A case report of anaplastic carcinoma of the pancreas with remarkable intraductal tumor growth into the main pancreatic duct显示文摘We herein report a case of anaplastic carcinoma of the pancreas with remarkable intraductal tumor growth into the main pancreatic duct.A 76-year-old male was referred to our hospital for treatment of a pancreatic tumor.Preoperative examinations revealed a poorly defined tumor in the main pancreatic duct in the body of the pancreas,accompanied with severe dilatation of the main pancreatic duct,which was diagnosed as an intraductal papillary-mucinous neoplasm.We performed distal pancreatectomy and splenectomy.The pathological examination revealed that the tumor consisted of a mixture of anaplastic carcinoma(giant cell type)and adenocarcinoma in the pancreas.There was a papillary projecting tumor composed of anaplastic carcinoma in the dilated main pancreatic duct.The patient is now receiving chemotherapy because liver metastasis was detected 12 mo after surgery.In this case,we could observe a remarkable intraductal tumor growth into the main pancreatic duct.We also discuss the pathogenesis and characteristics of this rare tumor with specific tumor growth. | Mitsuyoshi Okazaki Isamu Makino Hirohisa Kitagawa Shinichi Nakanuma Hironori Hayashi Hisatoshi Nakagawara Tomoharu Miyashita Hidehiro Tajima Hiroyuki Takamura Tetsuo Ohta | 2014 | World Journal of Gastroenterology2014，20，3： | 2 |
| 5 | Long-term use of entecavir in nucleoside-na?ve Japanese patients with chronic hepatitis B infection显示文摘 | Osamu Yokosuka Koichi Takaguchi Shinichi Fujioka Michiko Shindo Kazuaki Chayama Haruhiko Kobashi Norio Hayashi Chifumi Sato Kendo Kiyosawa Kyuichi Tanikawa Hiroki Ishikawa Nobuyuki Masaki Taku Seriu Masao Omata | 2010 | Journal of Hepatology2010，，6： | 2 |
| 6 | Hypoadiponectinemia accelerates hepatic tumor formation in a nonalcoholic steatohepatitis mouse model显示文摘 | Yoshihiro Kamada Hitoshi Matsumoto Shinji Tamura Juichi Fukushima Shinichi Kiso KojiFukui Takumi Igura Norikazu Maeda $hinji Kihara Tohru Funahashi Yuji Matsuzawa Iichiro Shimomura Norio Hayashi | 2007 | Journal of Hepatology2007，，4： | 1 |
| 7 | Aggravation by Selective COX-1 and COX-2 Inhibitors of Dextran Sulfate Sodium (DSS)-Induced Colon Lesions in Rats显示文摘 | Mitsuaki Okayama Shusaku Hayashi Yoko Aoi Hikaru Nishio Shinichi Kato Koji Takeuchi | 2007 | Digestive Diseases and Sciences2007，，9： | 1 |
| 8 | RTRA differentially regulates interleukin-1β and interleukin-1 receptor antagonist production by human alveolar macrophages 显示文摘 | Shu Hashimoto Shinichi Hayashi Sachiko Yoshida | 1998 | Leukemia Research1998，22，： | 1 |
| 9 | Laparoscopic esophagojejunostomy using the EndoStitch and a circular stapler under a direct view created by the ENDOCAMELEON显示文摘 | Takaki Yoshikawa Tsutomu Hayashi Toru Aoyama Haruhiko Cho Hirohito Fujikawa Junya Shirai Shinichi Hasegawa Takanobu Yamada Takashi Oshima Norio Yukawa Yasushi Rino Munetaka Masuda Takashi Ogata Akira Tsuburaya | 2013 | Gastric Cancer2013，，4： | 1 |
| 10 | Low?dose paclitaxel inhibits the induction of epidermal?mesenchymaltransition in the human cholangiocarcinoma CCKS?1 cell line显示文摘 | Atsushi Hirose Hidehiro Tajima Tetsuo Ohta Tomoya Tsukada Koichi Okamoto Shinichi Nakanuma Seisho Sakai Jun Kinoshita Isamu Makino Hiroyuki Furukawa Hironori Hayashi Keishi Nakamura Katsunobu Oyama Masafumi Inokuchi Hisatoshi Nakagawara Tomoharu Miyashita | 2013 | Oncology Letters2013，，4： | 1 |
| 11 | Aggravation by Selective COX-1 and COX-2 Inhibitors of Dextran Sulfate Sodium (DSS)-Induced Colon Lesions in Rats显示文摘 | Mitsuaki Okayama Shusaku Hayashi Yoko Aoi Hikaru Nishio Shinichi Kato Koji Takeuchi | 2007 | Digestive Diseases and Sciences2007，，9： | 1 |
| 12 | Axonal protection by brain-derived neurotrophic factor associated with CREB phosphorylation in tumor necrosis factor-α-induced optic nerve degeneration显示文摘 | Hiromi Fujino Yasushi Kitaoka Yasuhiro Hayashi Yasunari Munemasa Hiroyuki Takeda Toshio Kumai Shinichi Kobayashi Satoki Ueno | 2009 | Acta Neuropathologica2009，，1： | 1 |
| 13 | Have Surgical Outcomes of Pathologic T4 Esophageal Squamous Cell Carcinoma Really Improved? Analysis of 268 Cases During 45 Years of Experience显示文摘 | Hideaki Shimada Tooru Shiratori Shinichi Okazumi Hisahiro Matsubara Yoshihiro Nabeya Kiyohiko Shuto Yasunori Akutsu Hideki Hayashi Kaichi Isono Takenori Ochiai | 2008 | Journal of the American College of Surgeons2008，，1： | 1 |
| 14 | Mutations of theBtk gene in 12 unrelated families with X-linked agammaglobulinemia in Japan显示文摘 | Shigetoshi Kobayashi Tsutomu Iwata Makiko Saito Ryu Iwasaki Hiroshi Matsumoto Shinichi Naritaka Yoichi Kono Yasuhide Hayashi | 1996 | Human Genetics1996，，4： | 1 |
| 15 | Long-term outcome of transoral organ-preserving pharyngeal endoscopic resection for superficial pharyngeal cancer显示文摘 | Manabu Muto Hironaga Satake Tomonori Yano Keiko Minashi Ryuichi Hayashi Satoshi Fujii Atsushi Ochiai Atsushi Ohtsu Shuko Morita Takahiro Horimatsu Yasumasa Ezoe Shinichi Miyamoto Ryo Asato Ichiro Tateya Akihiko Yoshizawa Tsutomu Chiba | 2011 | Gastrointestinal Endoscopy2011，，3： | 1 |
| 16 | Hepatocellular carcinoma complicated by gastrointestinal hemorrhage caused by direct tumor invasion of stomach显示文摘 | Akira Maruyama Koji Murabayashi Masanobu Hayashi Hideaki Nakano Shuji Isaji Shinichi Uehara Tsukasa Kusuda Shigeki Miyahara Akinobu Kondo Hiroshi Nakano Tadashi Yabana | 1999 | Journal of Hepato - Biliary - Pancreatic Surgery1999，，1： | 1 |
| 17 | Have Surgical Outcomes of Pathologic T4 Esophageal Squamous Cell Carcinoma Really Improved? Analysis of 268 Cases During 45 Years of Experience显示文摘 | Hideaki Shimada Tooru Shiratori Shinichi Okazumi Hisahiro Matsubara Yoshihiro Nabeya Kiyohiko Shuto Yasunori Akutsu Hideki Hayashi Kaichi Isono Takenori Ochiai | 2008 | Journal of the American College of Surgeons2008，，1： | 1 |
| 18 | Malignant transformation of hyperplastic gastric polyps: An immunohistochemicaland pathological study of the changes of neoplastic phenotype显示文摘 | Johji Imura Shinichi Hayashi Kazuhito Ichikawa Shigeharu Miwa Takahiko Nakajima Kazuhiro Nomoto Koichi Tsuneyama Tatsuya Nogami Hitoaki Saitoh Takahiro Fujimori | 2014 | Oncology Letters2014，，5： | 1 |
| 19 | A factor analysis-multiple regression model for source apportionment of suspended particulate matter显示文摘 | Shinichi Okamoto Masayuki Hayashi Masaomi Nakaji- ma etal | 1990 | Atmospheric Environment1990，24，8： | 1 |
| 20 | Clutch Cutter knife efficacy in endoscopic submucosal dissection for early gastric neoplasms显示文摘AIM To compare the outcomes of endoscopic submucosal dissection(ESD) for gastric neoplasms using Clutch Cutter(ESD-C) or other knives(ESD-O).METHODS This was a single-center retrospective study. Gastric neoplasms treated by ESD between April 2016 and October 2017 at Kitakyushu Municipal Medical Center were reviewed. Multivariate analyses and propensity score matching were used to reduce biases. Covariates included factors that might affect outcomes of ESD, including age, sex, underlying disease, anti-thrombotic drugs use, tumor location, tumor position, tumor size, tumor depth, tumor morphology, tumor histology, ulcer(scar), and operator skill. The treatment outcomes were compared among two groups. The primary outcome was ESD procedure time. Secondary outcomes were en bloc, complete, and curative resection rates, and adverse events rates including perforation and delayed bleeding.RESULTS A total of 155 patients were included in this study; 44 pairs were created by propensity score matching. Background characteristics were quite similar among two groups after matching. Procedure time was significantly shorter for ESD-C(median; 49 min) than for ESD-O(median; 88.5 min)(P < 0.01). However, there was no significant difference in treatment outcomes between ESD-C and ESD-O including en bloc resection rate(100% in both groups), complete resection rate(100% in both groups), curative resection rate(86.4% vs 88.6%, P = 0.730), delayed bleeding(2.3% vs 6.8%, P = 0.62) and perforation(0% in both groups).CONCLUSION ESD-C achieved shorter procedure time without an increase in complication risk. Therefore, ESD-C could become an effective ESD option for gastric neoplasms. | Yasuyo Hayashi Mitsuru Esaki Sho Suzuki Eikichi Ihara Azusa Yokoyama Seiichiro Sakisaka Taizo Hosokawa Yoshimasa Tanaka Takahiro Mizutani Shinichi Tsuruta Aya Iwao Shun Yamakawa Akira Irie Yosuke Minoda Yoshitaka Hata Haruei Ogino Hirotada Akiho Yoshihiro Ogawa | 2018 | World Journal of Gastrointestinal Oncology2018，10，12： | 1 |
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